Ludmila Solomin

2.3k total citations · 1 hit paper
13 papers, 1.9k citations indexed

About

Ludmila Solomin is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Immunology. According to data from OpenAlex, Ludmila Solomin has authored 13 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 5 papers in Immunology. Recurrent topics in Ludmila Solomin's work include Nuclear Receptors and Signaling (6 papers), T-cell and Retrovirus Studies (4 papers) and Retinoids in leukemia and cellular processes (4 papers). Ludmila Solomin is often cited by papers focused on Nuclear Receptors and Signaling (6 papers), T-cell and Retrovirus Studies (4 papers) and Retinoids in leukemia and cellular processes (4 papers). Ludmila Solomin collaborates with scholars based in Sweden, United States and Belgium. Ludmila Solomin's co-authors include Rolf Zetterström, Thomas Perlmann, Barry J. Hoffer, Barbara K. Felber, George N. Pavlakis, Thimios A. Mitsiadis, Thomas Perlmann, Alexander Mata de Urquiza, Åsa Wallén and Yoshiyuki Shimizu and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Ludmila Solomin

13 papers receiving 1.9k citations

Hit Papers

Dopamine Neuron Agenesis in Nurr1-Deficient Mice 1997 2026 2006 2016 1997 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Dopamine Neuron Agenesis in Nurr1-Deficient Mice
    1997 903 citations Rolf Zetterström, Ludmila Solomin et al. Science profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ludmila Solomin Sweden 12 1.3k 1.1k 396 247 208 13 1.9k
Joanna C. Bakowska United States 20 567 0.4× 835 0.7× 84 0.2× 224 0.9× 316 1.5× 26 1.8k
Giovanna Lalli United Kingdom 22 759 0.6× 934 0.8× 148 0.4× 124 0.5× 203 1.0× 31 1.9k
Kathryn Bobb United States 9 819 0.6× 917 0.8× 139 0.4× 225 0.9× 275 1.3× 13 1.7k
M. E. Greenberg United States 9 624 0.5× 1.7k 1.5× 169 0.4× 272 1.1× 119 0.6× 9 2.2k
Susana Montenegro Gouveia United States 15 510 0.4× 1.6k 1.5× 64 0.2× 229 0.9× 139 0.7× 15 2.6k
Baptiste N. Jaeger United States 16 274 0.2× 943 0.8× 895 2.3× 163 0.7× 310 1.5× 21 2.3k
Amy E. Sheehan United States 17 725 0.6× 1.1k 1.0× 390 1.0× 148 0.6× 176 0.8× 21 2.0k
Changan Jiang China 14 759 0.6× 1.1k 1.0× 251 0.6× 195 0.8× 85 0.4× 21 1.7k
S. Ymer Australia 12 2.1k 1.7× 2.0k 1.8× 392 1.0× 194 0.8× 132 0.6× 15 3.2k
Brigitte Grima France 15 1.1k 0.9× 736 0.7× 175 0.4× 273 1.1× 129 0.6× 19 2.3k

Countries citing papers authored by Ludmila Solomin

Since Specialization
Citations

This map shows the geographic impact of Ludmila Solomin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ludmila Solomin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ludmila Solomin more than expected).

Fields of papers citing papers by Ludmila Solomin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ludmila Solomin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ludmila Solomin. The network helps show where Ludmila Solomin may publish in the future.

Co-authorship network of co-authors of Ludmila Solomin

This figure shows the co-authorship network connecting the top 25 collaborators of Ludmila Solomin. A scholar is included among the top collaborators of Ludmila Solomin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ludmila Solomin. Ludmila Solomin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Misner, Dinah, Yoshiyuki Shimizu, Alexander Mata de Urquiza, et al.. (2001). Vitamin A deprivation results in reversible loss of hippocampal long-term synaptic plasticity. Proceedings of the National Academy of Sciences. 98(20). 11714–11719. 210 indexed citations
2.
Urquiza, Alexander Mata de, Ludmila Solomin, & Thomas Perlmann. (1999). Feedback-inducible nuclear-receptor-driven reporter gene expression in transgenic mice. Proceedings of the National Academy of Sciences. 96(23). 13270–13275. 26 indexed citations
3.
Wallén, Åsa, et al.. (1999). Fate of Mesencephalic AHD2-Expressing Dopamine Progenitor Cells in Nurr1 Mutant Mice. Experimental Cell Research. 253(2). 737–746. 193 indexed citations
4.
Solomin, Ludmila, Clas B. Johansson, Rolf Zetterström, et al.. (1998). Retinoid-X receptor signalling in the developing spinal cord. Nature. 395(6700). 398–402. 117 indexed citations
5.
Cheng, Henrich, Rolf Zetterström, Ludmila Solomin, et al.. (1998). On CNS repair and protection strategies: novel approaches with implications for spinal cord injury and Parkinson's disease1Published on the World Wide Web on 13 January 1998.1. Brain Research Reviews. 26(2-3). 302–305. 11 indexed citations
6.
Zetterström, Rolf, et al.. (1997). Dopamine Neuron Agenesis in Nurr1-Deficient Mice. Science. 276(5310). 248–250. 903 indexed citations breakdown →
7.
Zetterström, Rolf, et al.. (1996). Retinoid X receptor heterodimerization and developmental expression distinguish the orphan nuclear receptors NGFI-B, Nurr1, and Nor1.. Molecular Endocrinology. 10(12). 1656–1666. 200 indexed citations
8.
Wahren‐Herlenius, Marie, Ludmila Solomin, I. Pettersson, & David Isenberg. (1996). Autoantibody Repertoire to Ro/SSA and La/SSB Antigens in Patients with Primary and Secondary Sjögren's syndrome. Journal of Autoimmunity. 9(4). 537–544. 24 indexed citations
9.
Nasioulas, George, Andrei S. Zolotukhin, Carmen Tabernero, et al.. (1994). Elements distinct from human immunodeficiency virus type 1 splice sites are responsible for the Rev dependence of env mRNA. Journal of Virology. 68(5). 2986–2993. 114 indexed citations
10.
Pavlakis, George N., et al.. (1992). Structure, regulation and oncogenic mechanisms of HTLV-I and HTLV-II.. PubMed. 6 Suppl 3. 176S–180S. 3 indexed citations
11.
Unge, Torsten, et al.. (1991). The Rex regulatory protein of human T-cell lymphotropic virus type I binds specifically to its target site within the viral RNA.. Proceedings of the National Academy of Sciences. 88(16). 7145–7149. 43 indexed citations
12.
Solomin, Ludmila, Barbara K. Felber, & George N. Pavlakis. (1990). Different sites of interaction for Rev, Tev, and Rex proteins within the Rev-responsive element of human immunodeficiency virus type 1. Journal of Virology. 64(12). 6010–6017. 58 indexed citations
13.
Robinson, Robin A., et al.. (1990). Binding of trans-dominant mutant Rev protein of human immunodeficiency virus type 1 to the cis-acting Rev-responsive element does not affect the fate of viral mRNA.. PubMed. 2(12). 1111–22. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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