Lucas T. Gray

3.3k total citations
10 papers, 924 citations indexed

About

Lucas T. Gray is a scholar working on Molecular Biology, Plant Science and Oncology. According to data from OpenAlex, Lucas T. Gray has authored 10 papers receiving a total of 924 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 2 papers in Plant Science and 1 paper in Oncology. Recurrent topics in Lucas T. Gray's work include RNA and protein synthesis mechanisms (5 papers), DNA and Nucleic Acid Chemistry (4 papers) and RNA Interference and Gene Delivery (4 papers). Lucas T. Gray is often cited by papers focused on RNA and protein synthesis mechanisms (5 papers), DNA and Nucleic Acid Chemistry (4 papers) and RNA Interference and Gene Delivery (4 papers). Lucas T. Gray collaborates with scholars based in United States, Japan and France. Lucas T. Gray's co-authors include Nancy Maizels, Aarthy C. Vallur, Alan M. Weiner, Thomas Pavelitz, Giang Huong Nguyen, Raymond J. Monnat, Ana I. Robles, Curtis C. Harris, Weiliang Tang and Richard P. Beyer and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Human Molecular Genetics and Nature Chemical Biology.

In The Last Decade

Lucas T. Gray

10 papers receiving 921 citations

Peers

Lucas T. Gray
Petko Fiziev United States
Anna V. Kotrys Poland
Yuewan Luo China
Vipin T. Sreedharan Germany
Etienne Raimondeau United Kingdom
Nicholas J. McGlincy United Kingdom
Pablo Armas Argentina
Devesh Bhimsaria United States
Lori A. Pile United States
Maksim Erokhin Russia
Petko Fiziev United States
Lucas T. Gray
Citations per year, relative to Lucas T. Gray Lucas T. Gray (= 1×) peers Petko Fiziev

Countries citing papers authored by Lucas T. Gray

Since Specialization
Citations

This map shows the geographic impact of Lucas T. Gray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lucas T. Gray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lucas T. Gray more than expected).

Fields of papers citing papers by Lucas T. Gray

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lucas T. Gray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lucas T. Gray. The network helps show where Lucas T. Gray may publish in the future.

Co-authorship network of co-authors of Lucas T. Gray

This figure shows the co-authorship network connecting the top 25 collaborators of Lucas T. Gray. A scholar is included among the top collaborators of Lucas T. Gray based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lucas T. Gray. Lucas T. Gray is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Gray, Lucas T., Emilia Puig Lombardi, Daniela Verga, et al.. (2019). G-quadruplexes Sequester Free Heme in Living Cells. Cell chemical biology. 26(12). 1681–1691.e5. 53 indexed citations
2.
Gray, Lucas T., Zizhen Yao, Thuc Nghi Nguyen, et al.. (2017). Layer-specific chromatin accessibility landscapes reveal regulatory networks in adult mouse visual cortex. eLife. 6. 43 indexed citations
3.
Tang, Weiliang, Ana I. Robles, Richard P. Beyer, et al.. (2016). The Werner syndrome RECQ helicase targets G4 DNA in human cells to modulate transcription. Human Molecular Genetics. 25(10). 2060–2069. 73 indexed citations
4.
Gray, Lucas T., et al.. (2014). G quadruplexes are genomewide targets of transcriptional helicases XPB and XPD. Nature Chemical Biology. 10(4). 313–318. 169 indexed citations
5.
Nguyen, Giang Huong, Weiliang Tang, Ana I. Robles, et al.. (2014). Regulation of gene expression by the BLM helicase correlates with the presence of G-quadruplex DNA motifs. Proceedings of the National Academy of Sciences. 111(27). 9905–9910. 96 indexed citations
6.
Maizels, Nancy & Lucas T. Gray. (2013). The G4 Genome. PLoS Genetics. 9(4). e1003468–e1003468. 394 indexed citations
7.
Weiner, Alan M. & Lucas T. Gray. (2013). What role (if any) does the highly conserved CSB-PGBD3 fusion protein play in Cockayne syndrome?. Mechanisms of Ageing and Development. 134(5-6). 225–233. 11 indexed citations
8.
Pavelitz, Thomas, Lucas T. Gray, Stéphanie L. Padilla, Arnold D. Bailey, & Alan M. Weiner. (2013). PGBD5: a neural-specific intron-containing piggyBac transposase domesticated over 500 million years ago and conserved from cephalochordates to humans. Mobile DNA. 4(1). 23–23. 22 indexed citations
9.
Gray, Lucas T., Kimberly K. Fong, Thomas Pavelitz, & Alan M. Weiner. (2012). Tethering of the Conserved piggyBac Transposase Fusion Protein CSB-PGBD3 to Chromosomal AP-1 Proteins Regulates Expression of Nearby Genes in Humans. PLoS Genetics. 8(9). e1002972–e1002972. 29 indexed citations
10.
Bailey, Arnold D., Lucas T. Gray, Thomas Pavelitz, et al.. (2012). The conserved Cockayne syndrome B-piggyBac fusion protein (CSB-PGBD3) affects DNA repair and induces both interferon-like and innate antiviral responses in CSB-null cells. DNA repair. 11(5). 488–501. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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