Louis Fries
- Epidemiology top 1%
- Infectious Diseases top 1%
- Immunology top 2%
- Molecular Biology
- Public Health, Environmental and Occupational Health top 5%
- Co-authors
- Michael M. FrankGregory M. GlennGale SmithM L ClementsMartin FrankCarl H. HammerEloi KpameganHanxin Lu
- Topics
- Respiratory viral infections research (31 papers)Influenza Virus Research Studies (29 papers)Monoclonal and Polyclonal Antibodies Research (13 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Clinical InvestigationThe Journal of Experimental Medicine
- Partner nations
- United StatesCanadaBelgium
In The Last Decade
Louis Fries
76 papers receiving 3.4k citations
Peers
Comparison fields: 5 of 118
- Epidemiology 1.8k
- Infectious Diseases 1.1k
- Immunology 1.1k
- Molecular Biology 568
- Public Health, Environmental and Occupational Health 500
Countries citing papers authored by Louis Fries
This map shows the geographic impact of Louis Fries's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Louis Fries with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Louis Fries more than expected).
Fields of papers citing papers by Louis Fries
This network shows the impact of papers produced by Louis Fries. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Louis Fries. The network helps show where Louis Fries may publish in the future.
Co-authorship network of co-authors of Louis Fries
This figure shows the co-authorship network connecting the top 25 collaborators of Louis Fries. A scholar is included among the top collaborators of Louis Fries based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Louis Fries. Louis Fries is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 4 | |
| 3 | 11 | |
| 4 | 59 | |
| 5 | 9 | |
| 6 | 6 | |
| 7 | 20 | |
| 8 | 21 | |
| 9 | 56 | |
| 10 | 108 | |
| 11 | 32 | |
| 12 | 3 | |
| 13 | 65 | |
| 14 | 74 | |
| 15 | 53 | |
| 16 | 372 | |
| 17 | 49 | |
| 18 | 49 | |
| 19 | 13 | |
| 20 | 45 |
About Louis Fries
Louis Fries is a scholar working on Infectious Diseases, Epidemiology and Immunology, having authored 77 papers that have together received 3.5k indexed citations. Recurring topics across this work include Respiratory viral infections research (31 papers), Influenza Virus Research Studies (29 papers) and Monoclonal and Polyclonal Antibodies Research (13 papers). The work is most often cited by research in Infectious Diseases (1.1k citations), Epidemiology (1.8k citations) and Immunology (1.1k citations). Louis Fries has collaborated with scholars based in United States, Canada and Belgium. Frequent co-authors include Michael M. Frank, Gregory M. Glenn, Gale Smith, M L Clements, Martin Frank, Carl H. Hammer, Eloi Kpamegan, Hanxin Lu, Brian R. Murphy and Somia P. Hickman. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and The Journal of Experimental Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.