Louis D. Van de Kar

7.5k total citations · 1 hit paper
136 papers, 6.3k citations indexed

About

Louis D. Van de Kar is a scholar working on Cellular and Molecular Neuroscience, Social Psychology and Molecular Biology. According to data from OpenAlex, Louis D. Van de Kar has authored 136 papers receiving a total of 6.3k indexed citations (citations by other indexed papers that have themselves been cited), including 76 papers in Cellular and Molecular Neuroscience, 74 papers in Social Psychology and 42 papers in Molecular Biology. Recurrent topics in Louis D. Van de Kar's work include Neuroendocrine regulation and behavior (74 papers), Neurotransmitter Receptor Influence on Behavior (66 papers) and Receptor Mechanisms and Signaling (37 papers). Louis D. Van de Kar is often cited by papers focused on Neuroendocrine regulation and behavior (74 papers), Neurotransmitter Receptor Influence on Behavior (66 papers) and Receptor Mechanisms and Signaling (37 papers). Louis D. Van de Kar collaborates with scholars based in United States, Israel and Hungary. Louis D. Van de Kar's co-authors include Gonzalo A. Carrasco, Stanley A. Lorens, Nancy A. Muma, Peter A. Rittenhouse, Cynthia L. Bethea, Francisca García, George Battaglia, Thackery S. Gray, Giuseppe Battaglia and Mark S. Brownfield and has published in prestigious journals such as Journal of Neuroscience, Biological Psychiatry and Brain Research.

In The Last Decade

Louis D. Van de Kar

135 papers receiving 6.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Neuroendocrine pharmacology of stress

2003 712 citations Gonzalo A. Carrasco, Louis D. Van de Kar European Journal of Pharmacology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Louis D. Van de Kar United States	45	2.7k	2.4k	2.2k	1.5k	905	136	6.3k
Gyula Telegdy Hungary	38	2.7k 1.0×	1.9k 0.8×	1.6k 0.7×	1.6k 1.1×	1.2k 1.4×	328	5.8k
Garth Bissette United States	48	3.3k 1.2×	1.7k 0.7×	3.2k 1.5×	2.0k 1.4×	827 0.9×	135	7.6k
Maarten van den Buuse Australia	48	3.4k 1.2×	1.4k 0.6×	1.8k 0.8×	2.0k 1.4×	476 0.5×	254	7.7k
Jeffrey G. Tasker United States	43	2.3k 0.8×	2.9k 1.2×	2.6k 1.2×	903 0.6×	2.3k 2.6×	98	7.0k
György Bagdy Hungary	50	2.6k 1.0×	1.2k 0.5×	1.6k 0.7×	1.3k 0.9×	1.0k 1.1×	260	7.4k
William E. Cullinan United States	35	2.6k 0.9×	3.6k 1.5×	4.9k 2.3×	880 0.6×	1.8k 1.9×	47	8.5k
Errol B. De Souza United States	46	2.3k 0.8×	1.2k 0.5×	3.0k 1.4×	1.4k 0.9×	566 0.6×	112	6.2k
Emilio Merlo Pich Italy	48	3.8k 1.4×	1.3k 0.5×	1.7k 0.8×	3.7k 2.5×	782 0.9×	145	8.9k
Randall R. Sakai United States	47	1.2k 0.5×	2.1k 0.9×	2.8k 1.3×	1.2k 0.8×	1.4k 1.5×	91	6.9k
Stephen J. Lolait United States	43	4.1k 1.5×	2.7k 1.1×	1.4k 0.6×	2.1k 1.5×	1.9k 2.1×	89	10.4k

Countries citing papers authored by Louis D. Van de Kar

Since Specialization

Citations

This map shows the geographic impact of Louis D. Van de Kar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Louis D. Van de Kar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Louis D. Van de Kar more than expected).

Fields of papers citing papers by Louis D. Van de Kar

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Louis D. Van de Kar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Louis D. Van de Kar. The network helps show where Louis D. Van de Kar may publish in the future.

Co-authorship network of co-authors of Louis D. Van de Kar

This figure shows the co-authorship network connecting the top 25 collaborators of Louis D. Van de Kar. A scholar is included among the top collaborators of Louis D. Van de Kar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Louis D. Van de Kar. Louis D. Van de Kar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Crane, James W., Keiko Shimizu, Gonzalo A. Carrasco, et al.. (2007). 5-HT1A receptors mediate (+)8-OH-DPAT-stimulation of extracellular signal-regulated kinase (MAP kinase) in vivo in rat hypothalamus: Time dependence and regional differences. Brain Research. 1183. 51–59. 23 indexed citations

D’Souza, Deborah N., Yahong Zhang, Katerina J. Damjanoska, et al.. (2004). Estrogen Reduces Serotonin-1A Receptor-Mediated Oxytocin Release and Gαi/o/z Proteins in the Hypothalamus of Ovariectomized Rats. Neuroendocrinology. 80(1). 31–41. 20 indexed citations

Damjanoska, Katerina J., Louis D. Van de Kar, Gisela Kindel, et al.. (2003). Chronic Fluoxetine Differentially Affects 5-Hydroxytryptamine2A Receptor Signaling in Frontal Cortex, Oxytocin- and Corticotropin-Releasing Factor-Containing Neurons in Rat Paraventricular Nucleus. Journal of Pharmacology and Experimental Therapeutics. 306(2). 563–571. 37 indexed citations

Raap, Danı́ K., Lydia L. DonCarlos, Francisca García, et al.. (2000). Estrogen desensitizes 5-HT1A receptors and reduces levels of Gz, Gi1 and Gi3 proteins in the hypothalamus. Neuropharmacology. 39(10). 1823–1832. 92 indexed citations

Muma, Nancy A., et al.. (2000). Coadministration of 5-Hydroxytryptamine1AAntagonist WAY-100635 Prevents Fluoxetine-Induced Desensitization of Postsynaptic 5-Hydroxytryptamine1A Receptors in Hypothalamus. Journal of Pharmacology and Experimental Therapeutics. 294(1). 296–301. 24 indexed citations

Battaglia, George, Theresa M. Cabrera-Vera, & Louis D. Van de Kar. (2000). Prenatal cocaine exposure potentiates 5-HT2a receptor function in male and female rat offspring. Synapse. 35(3). 163–172. 17 indexed citations

Kar, Louis D. Van de, et al.. (1998). Neuroendocrinology of stress: Behavioral and neurobiological considerations. Data Archiving and Networked Services (DANS). 3 indexed citations

Vicentic, Aleksandra, Qian Li, George Battaglia, & Louis D. Van de Kar. (1998). WAY-100635 inhibits 8-OH-DPAT-stimulated oxytocin, ACTH and corticosterone, but not prolactin secretion. European Journal of Pharmacology. 346(2-3). 261–266. 84 indexed citations

Muma, Nancy A., et al.. (1997). A Desensitization of Hypothalamic 5-HT1A Receptors by Repeated Injections of Paroxetine: Reduction in the Levels of Gi and Go Proteins and Neuroendocrine Responses, but Not in the Density of 5-HT1AReceptors. Journal of Pharmacology and Experimental Therapeutics. 282(3). 1581–1590. 135 indexed citations

10.

Kar, Louis D. Van de, et al.. (1997). The enigma of renin and 5-HT1A agonists. The FASEB Journal. 11(3). 1 indexed citations

11.

Muma, Nancy A., et al.. (1997). Fluoxetine gradually increases [ 125 I]DOI-labelled 5-HT 2A/2C receptors in the hypothalamus without changing the levels of G q - and G 11 -proteins. Brain Research. 775(1-2). 225–228. 37 indexed citations

12.

Li, Qian, Andrew D. Levy, Theresa M. Cabrera, et al.. (1993). Long-term fluoxetine, but not desipramine, inhibits the ACTH and oxytocin responses to the 5-HT1A agonist, 8-OH-DPAT, in male rats. Brain Research. 630(1-2). 148–156. 123 indexed citations

13.

Rittenhouse, Peter A., Erica A. Bakkum, Patricia A. O’Connor, et al.. (1992). Comparison of neuroendocrine and behavioral effects of ipsapirone, a 5-HT1A agonist, in three stress paradigms: immobilization, forced swim and conditioned fear. Brain Research. 580(1-2). 205–214. 57 indexed citations

14.

Kar, Louis D. Van de, et al.. (1992). Cocaine-induced suppression of renin secretion is mediated in the brain: Investigation of cardiovascular and local anesthetic mechanisms. Brain Research Bulletin. 28(5). 837–842. 5 indexed citations

15.

Kar, Louis D. Van de. (1991). Neuroendocrine Pharmacology of Serotonergic (5-HT) Neurons. The Annual Review of Pharmacology and Toxicology. 31(1). 289–320. 218 indexed citations

16.

Lorens, Stanley A. & Louis D. Van de Kar. (1987). Differential Effects of Serotonin (5-HT1A and 5-HT2) Agonists and Antagonists on Renin and Corticosterone Secretion. Neuroendocrinology. 45(4). 305–310. 109 indexed citations

17.

Kar, Louis D. Van de, et al.. (1987). Partial characterization of a renin-releasing factor from plasma and hypothalamus.. Hypertension. 9(6). 598–606. 16 indexed citations

18.

Kar, Louis D. Van de, et al.. (1985). Pharmacological Studies on the Serotoninergic and Nonserotonin-Mediated Stimulation of Prolactin and Corticosterone Secretion by Fenfluramine. Neuroendocrinology. 41(4). 283–288. 74 indexed citations

19.

Urban, Janice H., Cynthia L. Bethea, & Louis D. Van de Kar. (1984). Serotonin mediated effect of fenfluramine on prolactin but not corticosterone secretion. Federation Proceedings. 43(3). 1 indexed citations

20.

Kar, Louis D. Van de, et al.. (1982). Evidence that the Mediobasal Hypothalamus is Involved in Serotonergic Stimulation of Renin Secretion. Neuroendocrinology. 34(5). 323–326. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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