Lorna Pearn

781 total citations
12 papers, 628 citations indexed

About

Lorna Pearn is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Lorna Pearn has authored 12 papers receiving a total of 628 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Hematology and 3 papers in Genetics. Recurrent topics in Lorna Pearn's work include Acute Myeloid Leukemia Research (6 papers), Protein Kinase Regulation and GTPase Signaling (2 papers) and Chronic Lymphocytic Leukemia Research (2 papers). Lorna Pearn is often cited by papers focused on Acute Myeloid Leukemia Research (6 papers), Protein Kinase Regulation and GTPase Signaling (2 papers) and Chronic Lymphocytic Leukemia Research (2 papers). Lorna Pearn collaborates with scholars based in United Kingdom, Czechia and United States. Lorna Pearn's co-authors include Richard L. Darley, Alex Tonks, Alan K. Burnett, Alan K. Burnett, Paul S. Hole, Amanda J. Tonks, Robert K. Hills, Joanna Zabkiewicz, Janet Fisher and P. Bruce White and has published in prestigious journals such as Blood, Molecular and Cellular Biology and Leukemia.

In The Last Decade

Lorna Pearn

12 papers receiving 624 citations

Peers

Lorna Pearn
Anagha Inguva Sheth United States
Joanna Zabkiewicz United Kingdom
Chu Myong Seong South Korea
Nina Kurrle Germany
Sijana H. Dzinic United States
Anagha Inguva Sheth United States
Lorna Pearn
Citations per year, relative to Lorna Pearn Lorna Pearn (= 1×) peers Anagha Inguva Sheth

Countries citing papers authored by Lorna Pearn

Since Specialization
Citations

This map shows the geographic impact of Lorna Pearn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lorna Pearn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lorna Pearn more than expected).

Fields of papers citing papers by Lorna Pearn

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lorna Pearn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lorna Pearn. The network helps show where Lorna Pearn may publish in the future.

Co-authorship network of co-authors of Lorna Pearn

This figure shows the co-authorship network connecting the top 25 collaborators of Lorna Pearn. A scholar is included among the top collaborators of Lorna Pearn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lorna Pearn. Lorna Pearn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Zabkiewicz, Joanna, Lorna Pearn, Robert K. Hills, et al.. (2013). The PDK1 master kinase is over-expressed in acute myeloid leukemia and promotes PKC-mediated survival of leukemic blasts. Haematologica. 99(5). 858–864. 45 indexed citations
2.
Hole, Paul S., Joanna Zabkiewicz, Chinmay Munje, et al.. (2013). Overproduction of NOX-derived ROS in AML promotes proliferation and is associated with defective oxidative stress signaling. Blood. 122(19). 3322–3330. 184 indexed citations
3.
Morgan, Gareth, Lorna Pearn, Kate Liddiard, et al.. (2012). γ-Catenin is overexpressed in acute myeloid leukemia and promotes the stabilization and nuclear localization of β-catenin. Leukemia. 27(2). 336–343. 45 indexed citations
4.
Morgan, Gareth, Lorna Pearn, Kate Liddiard, et al.. (2010). Distinct Regulation of β- and γ-Catenin throughout Hematopoietic Development Contrasts with Their Cooperative Roles In Acute Myeloid Leukemia.. Blood. 116(21). 1573–1573. 1 indexed citations
5.
Hole, Paul S., Lorna Pearn, Amanda J. Tonks, et al.. (2009). Ras-induced reactive oxygen species promote growth factor–independent proliferation in human CD34+ hematopoietic progenitor cells. Blood. 115(6). 1238–1246. 107 indexed citations
6.
Tonks, Alex, Lorna Pearn, Amanda Gilkes, et al.. (2007). Transcriptional dysregulation mediated by RUNX1-RUNX1T1 in normal human progenitor cells and in acute myeloid leukaemia. Leukemia. 21(12). 2495–2505. 68 indexed citations
7.
Pearn, Lorna, Janet Fisher, Alan K. Burnett, & Richard L. Darley. (2007). The role of PKC and PDK1 in monocyte lineage specification by Ras. Blood. 109(10). 4461–4469. 42 indexed citations
8.
Omidvar, Nader, Lorna Pearn, Alan K. Burnett, & Richard L. Darley. (2006). Ral Is both Necessary and Sufficient for the Inhibition of Myeloid Differentiation Mediated by Ras. Molecular and Cellular Biology. 26(10). 3966–3975. 14 indexed citations
9.
Tonks, Alex, et al.. (2005). Optimized Retroviral Transduction Protocol Which Preserves the Primitive Subpopulation of Human Hematopoietic Cells. Biotechnology Progress. 21(3). 953–958. 15 indexed citations
10.
Tonks, Amanda J., Alex Tonks, Lorna Pearn, et al.. (2004). Expression of AML1-ETO in human myelomonocytic cells selectively inhibits granulocytic differentiation and promotes their self-renewal. Leukemia. 18(7). 1238–1245. 32 indexed citations
11.
Darley, Richard L., Lorna Pearn, Nader Omidvar, et al.. (2002). Protein kinase C mediates mutant N-Ras–induced developmental abnormalities in normal human erythroid cells. Blood. 100(12). 4185–4192. 26 indexed citations
12.
Tonks, Alex, Lorna Pearn, Amanda J. Tonks, et al.. (2002). The AML1-ETO fusion gene promotes extensive self-renewal of human primary erythroid cells. Blood. 101(2). 624–632. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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