Lora W. Forman

911 total citations
19 papers, 743 citations indexed

About

Lora W. Forman is a scholar working on Molecular Biology, Oncology and Epidemiology. According to data from OpenAlex, Lora W. Forman has authored 19 papers receiving a total of 743 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 9 papers in Oncology and 3 papers in Epidemiology. Recurrent topics in Lora W. Forman's work include Protein Kinase Regulation and GTPase Signaling (5 papers), Cell death mechanisms and regulation (5 papers) and Cancer-related Molecular Pathways (5 papers). Lora W. Forman is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (5 papers), Cell death mechanisms and regulation (5 papers) and Cancer-related Molecular Pathways (5 papers). Lora W. Forman collaborates with scholars based in United States. Lora W. Forman's co-authors include Douglas V. Faller, Susan P. Perrine, Yan Dai, Changyan Chen, Duyen Ngo, Sibaji Sarkar, Michael W. Phelan, Shu‐Hua Xia, Ana Lúcia Abujamra and Robert M. Williams and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Oncology and Molecular and Cellular Biology.

In The Last Decade

Lora W. Forman

19 papers receiving 739 citations

Peers

Lora W. Forman

ONCOL

EPIDE

Cynthia S.W. Ho Canada

Ray-Whay Chiu Yen United States

Fulvio Chiacchiera Italy

Robert A. H. van de Ven United States

Patrizia Damonte Italy

Xiaofang Ying China

Can Tan United States

Jennifer Campbell Canada

Krishna Seshu Tummala Spain

Ryoichi Asaka Japan

Cynthia S.W. Ho Canada

Lora W. Forman

675 ×1.3

271 ×1.7

ONCOL

89 ×0.8

191 ×1.7

137 ×1.3

EPIDE

Citations per year, relative to Lora W. Forman Lora W. Forman (= 1×) peers Cynthia S.W. Ho

Countries citing papers authored by Lora W. Forman

Since Specialization

Citations

This map shows the geographic impact of Lora W. Forman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lora W. Forman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lora W. Forman more than expected).

Fields of papers citing papers by Lora W. Forman

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lora W. Forman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lora W. Forman. The network helps show where Lora W. Forman may publish in the future.

Co-authorship network of co-authors of Lora W. Forman

This figure shows the co-authorship network connecting the top 25 collaborators of Lora W. Forman. A scholar is included among the top collaborators of Lora W. Forman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lora W. Forman. Lora W. Forman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

19 of 19 papers shown

Chen, Zhihong, Lora W. Forman, Robert M. Williams, & Douglas V. Faller. (2014). Protein kinase C-delta inactivation inhibits the proliferation and survival of cancer stem cells in culture and in vivo. BMC Cancer. 14(1). 90–90. 47 indexed citations

Rankin, A. M., Lora W. Forman, Sibaji Sarkar, & Douglas V. Faller. (2013). Enhanced Cytotoxicity from Deoxyguanosine-Enriched T-oligo in Prostate Cancer Cells. Nucleic Acid Therapeutics. 23(5). 311–321. 12 indexed citations

Sorescu, George P., Lora W. Forman, & Douglas V. Faller. (2012). Effect of inhibition of protein kinase C delta (PKCδ) on pancreatic cancer cells.. Journal of Clinical Oncology. 30(15_suppl). e14591–e14591. 3 indexed citations

Chen, Zhihong, et al.. (2011). Protein kinase Cδ inactivation inhibits cellular proliferation and decreases survival in human neuroendocrine tumors. Endocrine Related Cancer. 18(6). 759–771. 16 indexed citations

Sarkar, Sibaji, et al.. (2011). Histone deacetylase inhibitors reverse CpG methylation by regulating DNMT1 through ERK signaling.. PubMed. 31(9). 2723–32. 120 indexed citations

Byles, Vanessa, et al.. (2010). Aberrant Cytoplasm Localization and Protein Stability of SIRT1 is Regulated by PI3K/IGF-1R Signaling in Human Cancer Cells. International Journal of Biological Sciences. 6(6). 599–612. 92 indexed citations

Forman, Lora W., et al.. (2009). Identification of LTR‐specific small non‐coding RNA in FeLV infected cells. FEBS Letters. 583(8). 1386–1390. 13 indexed citations

Dai, Yan, et al.. (2008). Prohibitin and the SWI/SNF ATPase subunit BRG1 are required for effective androgen antagonist-mediated transcriptional repression of androgen receptor-regulated genes. Carcinogenesis. 29(9). 1725–1733. 53 indexed citations

Xia, Shu‐Hua, Zhihong Chen, Lora W. Forman, & Douglas V. Faller. (2008). PKCδ survival signaling in cells containing an activated p21Ras protein requires PDK1. Cellular Signalling. 21(4). 502–508. 29 indexed citations

10.

Xia, Shu‐Hua, Lora W. Forman, & Douglas V. Faller. (2007). Protein Kinase Cδ Is Required for Survival of Cells Expressing Activated p21. Journal of Biological Chemistry. 282(18). 13199–13210. 41 indexed citations

11.

Dai, Yan, et al.. (2007). Sirtuin 1 Is Required for Antagonist-Induced Transcriptional Repression of Androgen-Responsive Genes by the Androgen Receptor. Molecular Endocrinology. 21(8). 1807–1821. 81 indexed citations

12.

Ghosh, Sajal K., Lora W. Forman, Idowu Akinsheye, Susan P. Perrine, & Douglas V. Faller. (2006). Short, discontinuous exposure to butyrate effectively sensitizes latently EBV-infected lymphoma cells to nucleoside analogue antiviral agents. Blood Cells Molecules and Diseases. 38(1). 57–65. 14 indexed citations

13.

Forman, Lora W., et al.. (2002). Desensitization of the PDGFβ Receptor by Modulation of the Cytoskeleton: The Role of p21Ras and Rho Family GTPases. Experimental Cell Research. 275(1). 17–30. 8 indexed citations

14.

Chen, Changyan, et al.. (1998). Differential Regulation of Discrete Apoptotic Pathways by Ras. Journal of Biological Chemistry. 273(27). 16700–16709. 52 indexed citations

15.

Phelan, Michael W., Lora W. Forman, Susan P. Perrine, & Douglas V. Faller. (1998). Hypoxia increases thrombospondin-1 transcript and protein in cultured endothelial cells. Journal of Laboratory and Clinical Medicine. 132(6). 519–529. 89 indexed citations

16.

Chen, Changyan, et al.. (1998). Correlation of genetic instability and apoptosis in the presence of oncogenic Ki-Ras. Cell Death and Differentiation. 5(11). 984–995. 16 indexed citations

17.

Chen, Changyan, Lora W. Forman, & Douglas V. Faller. (1996). Calcium-Dependent Immediate-Early Gene Induction in Lymphocytes Is Negatively Regulated by p21 ^Ha- ^ras . Molecular and Cellular Biology. 16(11). 6582–6592. 26 indexed citations

18.

Faller, Douglas V., et al.. (1994). v-mos suppresses platelet-derived growth factor (PDGF) type-beta receptor autophosphorylation and inhibits PDGF-BB-mediated signal transduction.. Journal of Biological Chemistry. 269(7). 5022–5029. 8 indexed citations

19.

Forman, Lora W., et al.. (1994). Platelet-derived growth factor (PDGF) induction of egr-1 is independent of PDGF receptor autophosphorylation on tyrosine.. Journal of Biological Chemistry. 269(23). 16137–16142. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact