Ling Sun

469 total citations
19 papers, 338 citations indexed

About

Ling Sun is a scholar working on Molecular Biology, Cancer Research and Hematology. According to data from OpenAlex, Ling Sun has authored 19 papers receiving a total of 338 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 6 papers in Cancer Research and 5 papers in Hematology. Recurrent topics in Ling Sun's work include Acute Myeloid Leukemia Research (5 papers), MicroRNA in disease regulation (4 papers) and Cancer-related molecular mechanisms research (3 papers). Ling Sun is often cited by papers focused on Acute Myeloid Leukemia Research (5 papers), MicroRNA in disease regulation (4 papers) and Cancer-related molecular mechanisms research (3 papers). Ling Sun collaborates with scholars based in China, United States and Norway. Ling Sun's co-authors include Zhongxing Jiang, Ping Tang, Yumin Huang, Yanfang Liu, Haixia Cao, Hui Sun, Ran Zhang, Lina Sang, Weijie Cao and Chong Wang and has published in prestigious journals such as Scientific Reports, Biochemical and Biophysical Research Communications and FEBS Letters.

In The Last Decade

Ling Sun

18 papers receiving 336 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ling Sun China 11 201 118 81 68 61 19 338
Jing-Yuan Chooi Singapore 9 277 1.4× 87 0.7× 85 1.0× 80 1.2× 41 0.7× 10 328
Anna Wojtuszkiewicz Netherlands 11 432 2.1× 166 1.4× 79 1.0× 65 1.0× 48 0.8× 17 557
Claudia Merz Germany 8 258 1.3× 122 1.0× 30 0.4× 80 1.2× 45 0.7× 13 340
Leutz Buon United States 10 241 1.2× 44 0.4× 113 1.4× 133 2.0× 56 0.9× 28 368
Juliana M. Benito United States 5 125 0.6× 103 0.9× 70 0.9× 50 0.7× 24 0.4× 10 230
M.D. Minden Canada 7 152 0.8× 87 0.7× 86 1.1× 52 0.8× 41 0.7× 16 289
Kaya Zhu United States 7 276 1.4× 93 0.8× 40 0.5× 94 1.4× 52 0.9× 8 381
Shiho Fujiwara Japan 8 195 1.0× 51 0.4× 125 1.5× 130 1.9× 42 0.7× 16 323
Gopichand Pendurti United States 6 133 0.7× 48 0.4× 52 0.6× 98 1.4× 76 1.2× 11 287

Countries citing papers authored by Ling Sun

Since Specialization
Citations

This map shows the geographic impact of Ling Sun's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ling Sun with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ling Sun more than expected).

Fields of papers citing papers by Ling Sun

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ling Sun. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ling Sun. The network helps show where Ling Sun may publish in the future.

Co-authorship network of co-authors of Ling Sun

This figure shows the co-authorship network connecting the top 25 collaborators of Ling Sun. A scholar is included among the top collaborators of Ling Sun based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ling Sun. Ling Sun is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Chai, Mengnan, Ting‐Yu Liu, Xiaoyuan Xu, et al.. (2025). Overexpression of the Transcription Factor GmbZIP60 Increases Salt and Drought Tolerance in Soybean (Glycine max). International Journal of Molecular Sciences. 26(7). 3455–3455.
2.
Hao, Qianqian, Yu Liu, Yajun Liu, et al.. (2024). Cysteine- and glycine-rich protein 1 predicts prognosis and therapy response in patients with acute myeloid leukemia. Clinical and Experimental Medicine. 24(1). 57–57. 2 indexed citations
3.
Tjønnfjord, Geir E., Sandip M. Kanse, Anders Erik Astrup Dahm, et al.. (2021). Tissue factor pathway inhibitor upregulates CXCR7 expression and enhances CXCL12-mediated migration in chronic lymphocytic leukemia. Scientific Reports. 11(1). 5127–5127. 14 indexed citations
4.
Sun, Ling, et al.. (2021). Meta-analysis of the benefit of hypomethylating agents before allogeneic hematopoietic stem cell transplantation in myelodysplastic syndromes. Clinical and Experimental Medicine. 21(4). 537–543. 7 indexed citations
5.
Cao, Haixia, Lina Sang, Yumin Huang, et al.. (2020). Circ_0009910 promotes imatinib resistance through ULK1-induced autophagy by sponging miR-34a-5p in chronic myeloid leukemia. Life Sciences. 243. 117255–117255. 60 indexed citations
6.
Yu, Jifeng, Yingmei Li, Yafei Li, et al.. (2020). Gene mutational analysis by NGS and its clinical significance in patients with myelodysplastic syndrome and acute myeloid leukemia. Experimental Hematology and Oncology. 9(1). 2–2. 53 indexed citations
7.
Ma, Ping, Lijie Han, Feifei Wu, et al.. (2020). High PD‑L1 expression drives glycolysis via an Akt/mTOR/HIF‑1α axis in acute myeloid leukemia. Oncology Reports. 43(3). 999–1009. 25 indexed citations
8.
Chen, Huili, Ping Ma, Yanli Chen, et al.. (2019). [Effect of Metformin on Proliferation Capacity, Apoptosis and Glycolysis in K562 Cells].. PubMed. 27(5). 1387–1394. 5 indexed citations
9.
Zhang, Ronghui, Ping Tang, Fang Wang, et al.. (2018). Tumor suppressor miR‐139‐5p targets Tspan3 and regulates the progression of acute myeloid leukemia through the PI3K/Akt pathway. Journal of Cellular Biochemistry. 120(3). 4423–4432. 21 indexed citations
10.
Wang, Chong, et al.. (2018). [Metformin Induces Apoptosis of Human Multiple Myeloma Cell U266 through the Mitochondrial Apoptotic Pathway].. PubMed. 26(2). 489–492. 3 indexed citations
11.
Wang, Yan, et al.. (2017). Overexpression of microRNA-125b inhibits human acute myeloid leukemia cells invasion, proliferation and promotes cells apoptosis by targeting NF-κB signaling pathway. Biochemical and Biophysical Research Communications. 488(1). 60–66. 15 indexed citations
12.
Guo, Rong, Fang Wang, Zhong‐Xing Jiang, et al.. (2017). Retraction: IRF9 inhibits human acute myeloid leukemia through the SIRT1‐p53 signaling pathway. FEBS Letters. 591(18). 2951–2951. 1 indexed citations
13.
Gao, Fengcai, et al.. (2017). [Proliferation and Apoptosis of Leukemia Cell Line K562 Treated with HSP90 Inhibitor 17-DMAG].. PubMed. 25(4). 998–1002. 4 indexed citations
14.
Liu, Jia, et al.. (2017). Targeting the mTOR pathway in breast cancer. Tumor Biology. 39(6). 3726132682–3726132682. 23 indexed citations
16.
Jiang, Zhongxing, Fang Wang, Rong Guo, et al.. (2016). IRF3 is involved in human acute myeloid leukemia through regulating the expression of miR-155. Biochemical and Biophysical Research Communications. 478(3). 1130–1135. 15 indexed citations
17.
Wang, Chong, Jing Chen, Weijie Cao, et al.. (2015). Aurora-B and HDAC synergistically regulate survival and proliferation of lymphoma cell via AKT, mTOR and Notch pathways. European Journal of Pharmacology. 779. 1–7. 38 indexed citations
18.
He, Fei, Xue Fu, Juntang Lin, et al.. (2014). High expression of heat shock protein 90 alpha and its significance in human acute leukemia cells. Gene. 542(2). 122–128. 34 indexed citations
19.
Sang, Lina, Ling Sun, Yingmei Li, et al.. (2014). [The clinical observation of hemorrhagic cystitis following hematopoietic stem cell transplantation].. PubMed. 35(8). 747–9. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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