Linda A. Leis

528 total citations
18 papers, 427 citations indexed

About

Linda A. Leis is a scholar working on Hematology, Cardiology and Cardiovascular Medicine and Molecular Biology. According to data from OpenAlex, Linda A. Leis has authored 18 papers receiving a total of 427 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Hematology, 6 papers in Cardiology and Cardiovascular Medicine and 5 papers in Molecular Biology. Recurrent topics in Linda A. Leis's work include Platelet Disorders and Treatments (7 papers), Fibromyalgia and Chronic Fatigue Syndrome Research (3 papers) and Antiplatelet Therapy and Cardiovascular Diseases (3 papers). Linda A. Leis is often cited by papers focused on Platelet Disorders and Treatments (7 papers), Fibromyalgia and Chronic Fatigue Syndrome Research (3 papers) and Antiplatelet Therapy and Cardiovascular Diseases (3 papers). Linda A. Leis collaborates with scholars based in United States and France. Linda A. Leis's co-authors include Gerhard J. Johnson, R Bach, P C Dunlop, James G. White, G. J. Johnson, G. H. Rao, G J Johnson, Thomas S. Rector, Gary S. Francis and Marcy Krumwiede and has published in prestigious journals such as Circulation, Journal of Clinical Investigation and PLoS ONE.

In The Last Decade

Linda A. Leis

18 papers receiving 408 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Linda A. Leis United States 10 140 133 110 74 74 18 427
Andrew J. Johnson United Kingdom 12 69 0.5× 210 1.6× 73 0.7× 115 1.6× 15 0.2× 24 464
J. Bohner Germany 13 92 0.7× 187 1.4× 90 0.8× 43 0.6× 9 0.1× 35 594
Enrico Russolillo Italy 7 45 0.3× 284 2.1× 76 0.7× 22 0.3× 35 0.5× 7 506
L. Ek Sweden 11 27 0.2× 151 1.1× 137 1.2× 25 0.3× 7 0.1× 21 520
R.G. Kladetzky Germany 6 28 0.2× 100 0.8× 110 1.0× 14 0.2× 36 0.5× 10 479
Elke Schüler Germany 11 20 0.1× 175 1.3× 115 1.0× 24 0.3× 7 0.1× 13 473
Elias Rawish Germany 9 31 0.2× 132 1.0× 55 0.5× 18 0.2× 16 0.2× 16 347
Joris van Rossum Netherlands 13 49 0.3× 69 0.5× 104 0.9× 17 0.2× 8 0.1× 20 497
G. Singbartl Germany 10 50 0.4× 36 0.3× 101 0.9× 21 0.3× 7 0.1× 42 386
Isola A.M. Brown United States 6 29 0.2× 59 0.4× 131 1.2× 12 0.2× 17 0.2× 7 401

Countries citing papers authored by Linda A. Leis

Since Specialization
Citations

This map shows the geographic impact of Linda A. Leis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Linda A. Leis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Linda A. Leis more than expected).

Fields of papers citing papers by Linda A. Leis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Linda A. Leis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Linda A. Leis. The network helps show where Linda A. Leis may publish in the future.

Co-authorship network of co-authors of Linda A. Leis

This figure shows the co-authorship network connecting the top 25 collaborators of Linda A. Leis. A scholar is included among the top collaborators of Linda A. Leis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Linda A. Leis. Linda A. Leis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Johnson, Gerhard J., et al.. (2016). Blood Biomarkers of Chronic Inflammation in Gulf War Illness. PLoS ONE. 11(6). e0157855–e0157855. 83 indexed citations
2.
Johnson, Gerhard J., et al.. (2014). Elevated plasma biomarkers of chronic inflammation in Gulf War illness (591.3). The FASEB Journal. 28(S1). 3 indexed citations
3.
Johnson, Gerhard J., et al.. (2013). Elevated platelet count, C-reactive protein and thromboxane analog-induced platelet aggregation in patients with Gulf War veterans’ illnesses. Blood Coagulation & Fibrinolysis. 24(7). 736–741. 15 indexed citations
4.
Leis, Linda A., R Bach, & Gerhard J. Johnson. (2009). Tissue factor activity of blood mononuclear cells is increased after total knee arthroplasty. Thrombosis and Haemostasis. 102(10). 728–734. 34 indexed citations
5.
Leis, Linda A., et al.. (2007). The critical role of myosin IIA in platelet internal contraction. Journal of Thrombosis and Haemostasis. 5(7). 1516–1529. 36 indexed citations
6.
Johnson, G J, Linda A. Leis, P C Dunlop, & Ε. Kenneth Weir. (2003). The effect of the anorectic agent, d-fenfluramine, and its primary metabolite, d-norfenfluramine, on intact human platelet serotonin uptake and efflux. Journal of Thrombosis and Haemostasis. 1(12). 2663–2668. 8 indexed citations
7.
Dunlop, P C, Linda A. Leis, & Gerhard J. Johnson. (2000). Epinephrine correction of impaired platelet thromboxane receptor signaling. American Journal of Physiology-Cell Physiology. 279(6). C1760–C1771. 3 indexed citations
8.
Johnson, Gerhard J., Linda A. Leis, & P C Dunlop. (1996). Specificity of Gαq and Gα11 gene expression in platelets and erythrocytes. Expressions of cellular differentiation and species differences. Biochemical Journal. 318(3). 1023–1031. 31 indexed citations
9.
Johnson, G. J., Linda A. Leis, & P C Dunlop. (1993). Thromboxane-insensitive dog platelets have impaired activation of phospholipase C due to receptor-linked G protein dysfunction.. Journal of Clinical Investigation. 92(5). 2469–2479. 29 indexed citations
10.
Johnson, Gerhard J., Linda A. Leis, & Richard A. King. (1991). Thromboxane Responsiveness of Dog Platelets Is Inherited as an Autosomal Recessive Trait. Thrombosis and Haemostasis. 65(5). 578–580. 17 indexed citations
11.
Johnson, G J, Linda A. Leis, & Gary S. Francis. (1990). Adenosine potentiates the inhibitory effects of calcium channel antagonists on human platelet aggregation induced by thromboxane A2 or U46619. Thrombosis Research. 59(1). 139–148. 5 indexed citations
12.
Johnson, G. J., P C Dunlop, Linda A. Leis, & Arthur H. L. From. (1988). Dihydropyridine agonist Bay K 8644 inhibits platelet activation by competitive antagonism of thromboxane A2-prostaglandin H2 receptor.. Circulation Research. 62(3). 494–505. 29 indexed citations
13.
14.
Johnson, Gerhard J., et al.. (1981). Effect of inhibition of platelet function with carbenicillin or aspirin on experimental canine sudden death.. PubMed. 98(5). 660–72. 6 indexed citations
15.
Leis, Linda A., Terry K. Rosborough, Gary S. Johnson, & G. J. Johnson. (1980). Ristocetin-induced aggregation of canine platelets. Thrombosis Research. 19(3). 309–316. 7 indexed citations
16.
Johnson, Gerhard J., Bradford S. Schwartz, & Linda A. Leis. (1979). In vivo platelet retention in human bleeding-time wounds. I. Normal subjects and patients with platelet dysfunction.. PubMed. 94(4). 563–73. 4 indexed citations
17.
Johnson, Gerhard J., Linda A. Leis, G. H. Rao, & James G. White. (1979). Arachidonate-induced platelet aggregation in the dog. Thrombosis Research. 14(1). 147–154. 72 indexed citations
18.
Johnson, Gerhard J., Linda A. Leis, G. H. Rao, & James G. White. (1978). Prostaglandin synthesis remains intact in non-aggregating carbenicillin treated platelets. Circulation. 58. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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