Lijing Tang

927 total citations · 1 hit paper
21 papers, 672 citations indexed

About

Lijing Tang is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Pathology and Forensic Medicine. According to data from OpenAlex, Lijing Tang has authored 21 papers receiving a total of 672 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Pulmonary and Respiratory Medicine and 4 papers in Pathology and Forensic Medicine. Recurrent topics in Lijing Tang's work include Ferroptosis and cancer prognosis (4 papers), Mitochondrial Function and Pathology (3 papers) and Genetic and phenotypic traits in livestock (3 papers). Lijing Tang is often cited by papers focused on Ferroptosis and cancer prognosis (4 papers), Mitochondrial Function and Pathology (3 papers) and Genetic and phenotypic traits in livestock (3 papers). Lijing Tang collaborates with scholars based in China, Belgium and United Kingdom. Lijing Tang's co-authors include Xiu‐Ju Luo, Jun Peng, Nian‐Sheng Li, Xiao-Ming Xiong, Jie-Jie Zhang, Hua Tu, Heng Chen, Jingxin Liu, Xiaojie Zhang and Jun Peng and has published in prestigious journals such as Nature Communications, Cancer Research and Scientific Reports.

In The Last Decade

Lijing Tang

21 papers receiving 668 citations

Hit Papers

Ubiquitin-specific protease 7 promotes ferroptosis via ac... 2020 2026 2022 2024 2020 50 100 150 200

Peers

Lijing Tang
Lijing Tang
Citations per year, relative to Lijing Tang Lijing Tang (= 1×) peers Huiqiong He

Countries citing papers authored by Lijing Tang

Since Specialization
Citations

This map shows the geographic impact of Lijing Tang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lijing Tang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lijing Tang more than expected).

Fields of papers citing papers by Lijing Tang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lijing Tang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lijing Tang. The network helps show where Lijing Tang may publish in the future.

Co-authorship network of co-authors of Lijing Tang

This figure shows the co-authorship network connecting the top 25 collaborators of Lijing Tang. A scholar is included among the top collaborators of Lijing Tang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lijing Tang. Lijing Tang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhou, Yuguang, Meijing Liu, Na Lü, et al.. (2025). Advances in the design and application of glycolysis-related nanodelivery systems for anti-tumor. Colloids and Surfaces B Biointerfaces. 254. 114844–114844. 2 indexed citations
2.
Xiao, Shuang, et al.. (2025). Sorafenib-induced cardiovascular toxicity: A cause for concern. Chemico-Biological Interactions. 410. 111388–111388. 2 indexed citations
3.
Tang, Lijing, Benjamin Swedlund, Sébastien Dupont, et al.. (2024). GWAS reveals determinants of mobilization rate and dynamics of an active endogenous retrovirus of cattle. Nature Communications. 15(1). 2154–2154. 7 indexed citations
4.
Mao, Jingxin, Guowei Wang, Lihong Tan, et al.. (2023). Combined Network Pharmacology and Molecular Docking to Verify the Treatment of Type 2 Diabetes with Pueraria Lobata Radix and Salviae Miltiorrhizae Radix. Computational and Mathematical Methods in Medicine. 2023(1). 9150324–9150324. 8 indexed citations
5.
Tang, Lijing, Thomas Lopdell, В. А. Петров, et al.. (2023). An organism-wide ATAC-seq peak catalog for the bovine and its use to identify regulatory variants. Genome Research. 33(10). 1848–1864. 10 indexed citations
6.
Zhang, Yiyue, et al.. (2023). Inhibition of MALT1 reduces ferroptosis in rat hearts following ischemia/reperfusion via enhancing the Nrf2/SLC7A11 pathway. European Journal of Pharmacology. 950. 175774–175774. 19 indexed citations
7.
Mao, Jingxin, et al.. (2023). Systematic pharmacology-based strategy to explore the mechanism of Semen Strychni for treatment of papillary thyroid carcinoma. Scientific Reports. 13(1). 18492–18492. 7 indexed citations
9.
Tang, Lijing, et al.. (2022). The SPATA2/CYLD pathway contributes to doxorubicin-induced cardiomyocyte ferroptosis via enhancing ferritinophagy. Chemico-Biological Interactions. 368. 110205–110205. 27 indexed citations
10.
Wathes, D.C., Zhangrui Cheng, Mazdak Salavati, et al.. (2021). Relationships between metabolic profiles and gene expression in liver and leukocytes of dairy cows in early lactation. Journal of Dairy Science. 104(3). 3596–3616. 28 indexed citations
11.
Wathes, D.C., Frank Becker, Laura Buggiotti, et al.. (2021). Associations between Circulating IGF-1 Concentrations, Disease Status and the Leukocyte Transcriptome in Early Lactation Dairy Cows. Ghent University Academic Bibliography (Ghent University). 1(2). 147–177. 19 indexed citations
12.
Tu, Hua, Lijing Tang, Xiao-Ming Xiong, et al.. (2021). Combination of ponatinib with deferoxamine synergistically mitigates ischemic heart injury via simultaneous prevention of necroptosis and ferroptosis. European Journal of Pharmacology. 898. 173999–173999. 40 indexed citations
13.
Zhou, Jianqiu, et al.. (2021). miR-125a-5p-targeted regulation of TRA2β expression inhibits proliferation and metastasis of hepatocellular carcinoma cells.. PubMed. 13(12). 14074–14080. 3 indexed citations
14.
Tang, Lijing, Xiu‐Ju Luo, Hua Tu, et al.. (2020). Ferroptosis occurs in phase of reperfusion but not ischemia in rat heart following ischemia or ischemia/reperfusion. Naunyn-Schmiedeberg s Archives of Pharmacology. 394(2). 401–410. 123 indexed citations
15.
Tang, Lijing, Xiao-Ming Xiong, Nian‐Sheng Li, et al.. (2020). Ubiquitin-specific protease 7 promotes ferroptosis via activation of the p53/TfR1 pathway in the rat hearts after ischemia/reperfusion. Free Radical Biology and Medicine. 162. 339–352. 241 indexed citations breakdown →
16.
Chen, Heng, Lijing Tang, Hua Tu, et al.. (2020). Arctiin protects rat heart against ischemia/reperfusion injury via a mechanism involving reduction of necroptosis. European Journal of Pharmacology. 875. 173053–173053. 39 indexed citations
17.
Chen, Heng, Lijing Tang, Hua Tu, et al.. (2019). Upregulation of mitochondrial E3 ubiquitin ligase 1 in rat heart contributes to ischemia/reperfusion injury. Canadian Journal of Physiology and Pharmacology. 98(5). 259–266. 10 indexed citations
18.
Zhang, Yi‐Shuai, Lijing Tang, Hua Tu, et al.. (2018). Fasudil ameliorates the ischemia/reperfusion oxidative injury in rat hearts through suppression of myosin regulatory light chain/NADPH oxidase 2 pathway. European Journal of Pharmacology. 822. 1–12. 16 indexed citations
19.
Liu, Jingxin, et al.. (2007). Treatment of lycorine on SCID mice model with human APL cells. Biomedicine & Pharmacotherapy. 61(4). 229–234. 64 indexed citations
20.
Tao, Jun, Wei‐Xin Hu, Lijing Tang, et al.. (2005). Investigation of serum protein systems in Chinese pigs. Animal Genetics. 36(3). 223–226. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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