Li‐Hong Ben

416 total citations
12 papers, 249 citations indexed

About

Li‐Hong Ben is a scholar working on Immunology, Surgery and Immunology and Allergy. According to data from OpenAlex, Li‐Hong Ben has authored 12 papers receiving a total of 249 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 6 papers in Surgery and 3 papers in Immunology and Allergy. Recurrent topics in Li‐Hong Ben's work include Eosinophilic Esophagitis (5 papers), IL-33, ST2, and ILC Pathways (5 papers) and Allergic Rhinitis and Sensitization (3 papers). Li‐Hong Ben is often cited by papers focused on Eosinophilic Esophagitis (5 papers), IL-33, ST2, and ILC Pathways (5 papers) and Allergic Rhinitis and Sensitization (3 papers). Li‐Hong Ben collaborates with scholars based in United States and China. Li‐Hong Ben's co-authors include Andrew Murphy, Jamie Orengo, Matthew A. Sleeman, Seblewongel Asrat, André Limnander, Xia Liu, George D. Yancopoulos, Zheng Lian, Michael K. Racke and Yuhong Yang and has published in prestigious journals such as The Journal of Immunology, Annals of Neurology and Journal of Allergy and Clinical Immunology.

In The Last Decade

Li‐Hong Ben

10 papers receiving 246 citations

Peers

Li‐Hong Ben

IMMUN

PHYSI

CMN

Holm Bußler Germany

Ferdinand Jagot France

Debora Pehl Germany

Marian James United Kingdom

Sandra Martín-Salamanca Spain

Ana Amorim Switzerland

David Miguel Ferreira Francisco Switzerland

Alice Bernard Germany

Miko Valori Finland

Xiumao Li China

Holm Bußler Germany

Li‐Hong Ben

92 ×1.0

IMMUN

27 ×0.3

PHYSI

21 ×0.3

78 ×1.7

95 ×2.4

CMN

Citations per year, relative to Li‐Hong Ben Li‐Hong Ben (= 1×) peers Holm Bußler

Countries citing papers authored by Li‐Hong Ben

Since Specialization

Citations

This map shows the geographic impact of Li‐Hong Ben's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Li‐Hong Ben with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Li‐Hong Ben more than expected).

Fields of papers citing papers by Li‐Hong Ben

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Li‐Hong Ben. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Li‐Hong Ben. The network helps show where Li‐Hong Ben may publish in the future.

Co-authorship network of co-authors of Li‐Hong Ben

This figure shows the co-authorship network connecting the top 25 collaborators of Li‐Hong Ben. A scholar is included among the top collaborators of Li‐Hong Ben based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Li‐Hong Ben. Li‐Hong Ben is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

Srivatsan, Subhashini, Seblewongel Asrat, Kirsten Nagashima, et al.. (2023). Interleukin-33 (IL-33) Drives Type 1 and Type 2 Inflammation and Instructs Airway Remodeling. A4473–A4473.

Asrat, Seblewongel, Ye Zhou, Vishal Kamat, et al.. (2023). Itepekimab Binds to IL-33 With High Affinity, Prevents the Formation of IL-33/ST2/IL-1RAcP Signaling Complex and Blocks Mediators of Airway Inflammation. A4478–A4478.

Scott, George, Seblewongel Asrat, Jeanne Allinne, et al.. (2022). IL-4 and IL-13, not eosinophils, drive type 2 airway inflammation, remodeling and lung function decline. Cytokine. 162. 156091–156091. 54 indexed citations

Atanasio, Amanda, Matthew C. Franklin, Vishal Kamat, et al.. (2021). Targeting immunodominant Bet v 1 epitopes with monoclonal antibodies prevents the birch allergic response. Journal of Allergy and Clinical Immunology. 149(1). 200–211. 32 indexed citations

Asrat, Seblewongel, Xia Liu, Li‐Hong Ben, et al.. (2020). Chronic allergen exposure drives accumulation of long-lived IgE plasma cells in the bone marrow, giving rise to serological memory. Science Immunology. 5(43). 60 indexed citations

Atanasio, Amanda, Matthew C. Franklin, Li‐Hong Ben, et al.. (2020). Targeting birch allergy with monoclonal IgG antibodies that bind allergen and prevent IgE effector cell activation. Journal of Allergy and Clinical Immunology. 145(2). AB62–AB62. 1 indexed citations

Allinne, Jeanne, Wei Keat Lim, George Scott, et al.. (2019). IL-33 blockade prevents exacerbations in a model of chronic airway inflammation by blunting persistent inflammation and remodeling. PA3876–PA3876. 1 indexed citations

Allinne, Jeanne, George Scott, Dylan Birchard, et al.. (2018). IL-33 blockade impacts mediators of persistence and exacerbation of chronic airway inflammation.. OA1944–OA1944. 2 indexed citations

Yang, Yuhong, Yue Liu, Ping Wei, et al.. (2009). Silencing Nogo‐A promotes functional recovery in demyelinating disease. Annals of Neurology. 67(4). 498–507. 63 indexed citations

10.

Yang, Yuhong, Robert B. Ratts, Rehana Z. Hussain, et al.. (2007). CD28:B7 interaction is necessary for the protective effect of T cell vaccination in EAE. European Journal of Immunology. 37(7). 2032–2042. 6 indexed citations

11.

Lian, Zheng, Li‐Hong Ben, Jordan S. Pober, & Alfred L.M. Bothwell. (2002). Porcine Endothelial Cells, Unlike Human Endothelial Cells, Can Be Killed by Human CTL Via Fas Ligand and Cannot Be Protected by Bcl-2. The Journal of Immunology. 169(12). 6850–6855. 23 indexed citations

12.

Ben, Li‐Hong, Jian Zhao, Shunmei Xin, Shen-qiu Luo, & Gang Pei. (1999). Attenuation of δ opioid receptor-mediated signaling by kainic acid in neural cells: involvement of protein kinase C and intracellular Ca2+. Neuropharmacology. 38(7). 991–998. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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