Lihe Lu

1.8k citations
30 papers · 1.4k indexed · 2 hit papers · h-index 19

Lihe Lu

30 papers receiving 1.4k citations

Hit Papers

Repression of the antiporter SLC7A11/glutathione/glutathi...238202020262022202450100150200

Peers

Lihe Lu
Comparison fields: 5 of 112
  • Nephrology 205
  • Cancer Research 257
  • Geriatrics and Gerontology 48
  • Physiology 52
  • Cardiology and Cardiovascular Medicine 250
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Citations per field
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Citations per year

Countries citing papers authored by Lihe Lu

Since Specialization
Citations

This map shows the geographic impact of Lihe Lu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lihe Lu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lihe Lu more than expected).

Fields of papers citing papers by Lihe Lu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lihe Lu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lihe Lu. The network helps show where Lihe Lu may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Lihe Lu, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Lihe Lu Line = papers co-authored together Lihe Lu links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 202314
2 202318
3 202217
4
Repression of the antiporter SLC7A11/glutathione/glutathione peroxidase 4 axis drives ferroptosis of vascular smooth muscle cells to facilitate vascular calcificationbreakdown →
2022238
5 202144
6 202132
7
Fuzi Polysaccharide Inhibits VascularSmooth Muscle Cell Calcification via Ceramide Signaling
20202
8 202025
9 201942
10 201828
11 201626
12 201678
13 201634
14 201522
15
[High glucose promotes vascular smooth muscle cell calcification by activating WNT signaling pathway].
20159
16 201360
17 20131
18 2008148
19 200898
20 200410

About Lihe Lu

Lihe Lu is a scholar working on Nephrology, Geriatrics and Gerontology, Physiology, Cell Biology and Cancer Research, having authored 30 papers that have together received 1.4k indexed citations. Recurring topics across this work include Parathyroid Disorders and Treatments (6 papers), Caveolin-1 and cellular processes (4 papers), Bone Metabolism and Diseases (4 papers), Biomarkers in Disease Mechanisms (3 papers), Endoplasmic Reticulum Stress and Disease (3 papers), Dermatological and Skeletal Disorders (3 papers), Autophagy in Disease and Therapy (3 papers) and Metabolism, Diabetes, and Cancer (3 papers). The work is most often cited by research in Nephrology (205 citations), Cancer Research (257 citations), Geriatrics and Gerontology (48 citations), Physiology (52 citations) and Cardiology and Cardiovascular Medicine (250 citations). Lihe Lu has collaborated with scholars based in China, Japan and Macao. Frequent co-authors include Jianyun Yan, Yining Li, Xiaoyu Liu, Huimin Yu, Qingchun Liang, Jing‐Song Ou, An Chen, WU Wei-kang, Qianqian Dong and Mingwei Fu. Their work appears in journals such as European Journal of Pharmacology, The Journal of Pathology, Journal of Molecular and Cellular Cardiology, International Journal of Cardiology and Toxicology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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