Leonard Wu

3.4k total citations · 1 hit paper
22 papers, 2.8k citations indexed

About

Leonard Wu is a scholar working on Molecular Biology, Cancer Research and Cell Biology. According to data from OpenAlex, Leonard Wu has authored 22 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 9 papers in Cancer Research and 3 papers in Cell Biology. Recurrent topics in Leonard Wu's work include DNA Repair Mechanisms (22 papers), Carcinogens and Genotoxicity Assessment (9 papers) and Cancer therapeutics and mechanisms (5 papers). Leonard Wu is often cited by papers focused on DNA Repair Mechanisms (22 papers), Carcinogens and Genotoxicity Assessment (9 papers) and Cancer therapeutics and mechanisms (5 papers). Leonard Wu collaborates with scholars based in United Kingdom, United States and Switzerland. Leonard Wu's co-authors include Ian D. Hickson, Christine Ralf, Sally L. Davies, N C Levitt, Julia Karow, Csanád Z. Bachrati, Kok‐Lung Chan, Kevin C. Gatter, Helen Turley and Jean‐François Riou and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Leonard Wu

22 papers receiving 2.8k citations

Hit Papers

The Bloom's syndrome helicase suppresses crossing over du... 2003 2026 2010 2018 2003 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Leonard Wu United Kingdom 20 2.7k 793 597 406 313 22 2.8k
Joshua A. Sommers United States 33 2.6k 1.0× 796 1.0× 436 0.7× 362 0.9× 127 0.4× 57 2.8k
Julia Karow United Kingdom 9 2.0k 0.7× 579 0.7× 418 0.7× 257 0.6× 181 0.6× 12 2.1k
Stephen Van Komen United States 22 2.7k 1.0× 575 0.7× 327 0.5× 535 1.3× 219 0.7× 25 2.8k
Giordano Liberi Italy 27 4.2k 1.5× 892 1.1× 491 0.8× 805 2.0× 879 2.8× 38 4.4k
Neal Sugawara United States 26 3.3k 1.2× 499 0.6× 617 1.0× 375 0.9× 302 1.0× 32 3.5k
Eun Yong Shim United States 19 2.4k 0.9× 347 0.4× 299 0.5× 464 1.1× 240 0.8× 26 2.6k
Matthew J. Neale United Kingdom 21 3.0k 1.1× 386 0.5× 581 1.0× 516 1.3× 398 1.3× 33 3.2k
Kevin Hiom United Kingdom 21 2.6k 0.9× 414 0.5× 275 0.5× 583 1.4× 167 0.5× 36 3.0k
Risa Kitagawa United States 18 1.8k 0.7× 342 0.4× 259 0.4× 455 1.1× 455 1.5× 34 2.1k
James Hejna United States 16 1.8k 0.7× 515 0.6× 213 0.4× 294 0.7× 256 0.8× 23 1.9k

Countries citing papers authored by Leonard Wu

Since Specialization
Citations

This map shows the geographic impact of Leonard Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leonard Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leonard Wu more than expected).

Fields of papers citing papers by Leonard Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Leonard Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leonard Wu. The network helps show where Leonard Wu may publish in the future.

Co-authorship network of co-authors of Leonard Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Leonard Wu. A scholar is included among the top collaborators of Leonard Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Leonard Wu. Leonard Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wu, Leonard, et al.. (2011). Human HEL308 Localizes to Damaged Replication Forks and Unwinds Lagging Strand Structures. Journal of Biological Chemistry. 286(18). 15832–15840. 30 indexed citations
2.
Sidebotham, Julie M., et al.. (2010). Mph1 requires mismatch repair-independent and -dependent functions of MutSα to regulate crossover formation during homologous recombination repair. Nucleic Acids Research. 38(6). 1889–1901. 21 indexed citations
3.
Wu, Leonard, et al.. (2010). Overlapping Roles for Yen1 and Mus81 in Cellular Holliday Junction Processing. Journal of Biological Chemistry. 285(15). 11427–11432. 56 indexed citations
4.
Wu, Leonard. (2008). Wrestling off RAD51: a novel role for RecQ helicases. BioEssays. 30(4). 291–295. 17 indexed citations
5.
Wu, Leonard, Csanád Z. Bachrati, Jiongwen Ou, et al.. (2006). BLAP75/RMI1 promotes the BLM-dependent dissolution of homologous recombination intermediates. Proceedings of the National Academy of Sciences. 103(11). 4068–4073. 222 indexed citations
6.
Wu, Leonard & Ian D. Hickson. (2006). DNA Helicases Required for Homologous Recombination and Repair of Damaged Replication Forks. Annual Review of Genetics. 40(1). 279–306. 138 indexed citations
7.
Ralf, Christine, Ian D. Hickson, & Leonard Wu. (2006). The Bloom's Syndrome Helicase Can Promote the Regression of a Model Replication Fork. Journal of Biological Chemistry. 281(32). 22839–22846. 181 indexed citations
8.
Wu, Leonard, Kok‐Lung Chan, Christine Ralf, et al.. (2005). The HRDC domain of BLM is required for the dissolution of double Holliday junctions. The EMBO Journal. 24(14). 2679–2687. 143 indexed citations
9.
Sharma, Sudha, Joshua A. Sommers, Leonard Wu, et al.. (2004). Stimulation of Flap Endonuclease-1 by the Bloom's Syndrome Protein. Journal of Biological Chemistry. 279(11). 9847–9856. 88 indexed citations
10.
Wu, Leonard & Ian D. Hickson. (2003). The Bloom's syndrome helicase suppresses crossing over during homologous recombination. Nature. 426(6968). 870–874. 857 indexed citations breakdown →
11.
Braybrooke, Jeremy, et al.. (2003). Functional Interaction between the Bloom's Syndrome Helicase and the RAD51 Paralog, RAD51L3 (RAD51D). Journal of Biological Chemistry. 278(48). 48357–48366. 73 indexed citations
12.
Wu, Leonard & Ian D. Hickson. (2002). RecQ helicases and cellular responses to DNA damage. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 509(1-2). 35–47. 34 indexed citations
13.
Turley, Helen, Leonard Wu, Marta Cañamero, Kevin C. Gatter, & Ian D. Hickson. (2001). The distribution and expression of the Bloom’s syndrome gene product in normal and neoplastic human cells. British Journal of Cancer. 85(2). 261–265. 29 indexed citations
14.
Wu, Leonard, Sally L. Davies, N C Levitt, & Ian D. Hickson. (2001). Potential Role for the BLM Helicase in Recombinational Repair via a Conserved Interaction with RAD51. Journal of Biological Chemistry. 276(22). 19375–19381. 243 indexed citations
15.
Wu, Leonard & Ian D. Hickson. (2001). RecQ helicases and topoisomerases: components of a conserved complex for the regulation of genetic recombination. Cellular and Molecular Life Sciences. 58(7). 894–901. 37 indexed citations
16.
Wu, Leonard & Ian D. Hickson. (2001). DNA Ends RecQ-uire Attention. Science. 292(5515). 229–230. 31 indexed citations
17.
Wu, Leonard, S L Davies, & Ian D. Hickson. (2000). Roles of RecQ Family Helicases in the Maintenance of Genome Stability. Cold Spring Harbor Symposia on Quantitative Biology. 65(0). 573–582. 15 indexed citations
18.
Karow, Julia, Leonard Wu, & Ian D. Hickson. (2000). RecQ family helicases: roles in cancer and aging. Current Opinion in Genetics & Development. 10(1). 32–38. 149 indexed citations
19.
Wu, Leonard, Sally L. Davies, Phillip North, et al.. (2000). The Bloom's Syndrome Gene Product Interacts with Topoisomerase III. Journal of Biological Chemistry. 275(13). 9636–9644. 268 indexed citations
20.
Wu, Leonard, Julia Karow, & Ian D. Hickson. (1999). Genetic recombination: Helicases and topoisomerases link up. Current Biology. 9(14). R518–R520. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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