Leonard E. Parra

481 total citations
8 papers, 375 citations indexed

About

Leonard E. Parra is a scholar working on Pulmonary and Respiratory Medicine, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Leonard E. Parra has authored 8 papers receiving a total of 375 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Pulmonary and Respiratory Medicine, 4 papers in Cellular and Molecular Neuroscience and 3 papers in Molecular Biology. Recurrent topics in Leonard E. Parra's work include Respiratory and Cough-Related Research (5 papers), Neuropeptides and Animal Physiology (4 papers) and Receptor Mechanisms and Signaling (3 papers). Leonard E. Parra is often cited by papers focused on Respiratory and Cough-Related Research (5 papers), Neuropeptides and Animal Physiology (4 papers) and Receptor Mechanisms and Signaling (3 papers). Leonard E. Parra collaborates with scholars based in United States, United Kingdom and Bulgaria. Leonard E. Parra's co-authors include Robbie L. McLeod, John A. Hey, Robert W. Egan, Yanlin Jia, Xin Wang, Deen Tulshian, April Smith‐Torhan, Ahmad Fawzi, Galen Carey and Francis M. Cuss and has published in prestigious journals such as Journal of Pharmacology and Experimental Therapeutics, British Journal of Pharmacology and Pharmacology.

In The Last Decade

Leonard E. Parra

8 papers receiving 344 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Leonard E. Parra United States 8 157 131 130 129 86 8 375
J. Crist United States 9 248 1.6× 203 1.5× 17 0.1× 115 0.9× 77 0.9× 12 550
Yasuo Shimo Japan 16 297 1.9× 270 2.1× 84 0.6× 266 2.1× 38 0.4× 57 612
N. Mañé Spain 13 78 0.5× 111 0.8× 22 0.2× 95 0.7× 91 1.1× 18 400
M. Raab Germany 13 140 0.9× 94 0.7× 33 0.3× 76 0.6× 31 0.4× 26 410
I. A. Vladimirova Ukraine 8 123 0.8× 165 1.3× 12 0.1× 97 0.8× 58 0.7× 28 328
S D Collins United Kingdom 7 160 1.0× 148 1.1× 7 0.1× 262 2.0× 56 0.7× 14 452
Ingrid Liebmann Austria 9 153 1.0× 54 0.4× 22 0.2× 196 1.5× 11 0.1× 23 356
Nikita Mikhailov Finland 9 41 0.3× 91 0.7× 34 0.3× 121 0.9× 13 0.2× 11 259
Kevin Ellsworth United States 12 222 1.4× 198 1.5× 35 0.3× 41 0.3× 10 0.1× 17 415
D.J. Trezise United Kingdom 11 147 0.9× 194 1.5× 10 0.1× 133 1.0× 217 2.5× 15 436

Countries citing papers authored by Leonard E. Parra

Since Specialization
Citations

This map shows the geographic impact of Leonard E. Parra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leonard E. Parra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leonard E. Parra more than expected).

Fields of papers citing papers by Leonard E. Parra

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Leonard E. Parra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leonard E. Parra. The network helps show where Leonard E. Parra may publish in the future.

Co-authorship network of co-authors of Leonard E. Parra

This figure shows the co-authorship network connecting the top 25 collaborators of Leonard E. Parra. A scholar is included among the top collaborators of Leonard E. Parra based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Leonard E. Parra. Leonard E. Parra is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
McLeod, Robbie L., Deen Tulshian, Ginny D. Ho, et al.. (2009). Effect of a Novel NOP Receptor Agonist (SCH 225288) on Guinea Pig Irritant-Evoked, Feline Mechanically Induced and Canine Infectious Tracheobronchitis Cough. Pharmacology. 84(3). 153–161. 11 indexed citations
3.
McLeod, Robbie L., Yanlin Jia, Nansie A. McHugh, et al.. (2006). Sulfur-dioxide exposure increases TRPV1-mediated responses in nodose ganglia cells and augments cough in guinea pigs. Pulmonary Pharmacology & Therapeutics. 20(6). 750–757. 17 indexed citations
4.
McLeod, Robbie L., Yanlin Jia, Xiomara Fernandez, et al.. (2004). Antitussive Profile of the NOP Agonist Ro-64-6198 in the Guinea Pig. Pharmacology. 71(3). 143–149. 24 indexed citations
5.
McLeod, Robbie L., et al.. (2002). Characterization of nasal obstruction in the allergic guinea pig using the forced oscillation method. Journal of Pharmacological and Toxicological Methods. 48(3). 153–159. 9 indexed citations
6.
Jia, Yanlin, Robbie L. McLeod, Xin Wang, et al.. (2002). Anandamide induces cough in conscious guinea‐pigs through VR1 receptors. British Journal of Pharmacology. 137(6). 831–836. 65 indexed citations
7.
McLeod, Robbie L., Donald C. Bolser, Yanlin Jia, et al.. (2002). Antitussive Effect of Nociceptin/Orphanin FQ in Experimental Cough Models. Pulmonary Pharmacology & Therapeutics. 15(3). 213–216. 37 indexed citations
8.
McLeod, Robbie L., Leonard E. Parra, Galen Carey, et al.. (2001). Nociceptin inhibits cough in the guinea‐pig by activation of ORL1 receptors. British Journal of Pharmacology. 132(6). 1175–1178. 84 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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