Leihua Weng

572 total citations
11 papers, 445 citations indexed

About

Leihua Weng is a scholar working on Immunology, Neurology and Genetics. According to data from OpenAlex, Leihua Weng has authored 11 papers receiving a total of 445 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 4 papers in Neurology and 2 papers in Genetics. Recurrent topics in Leihua Weng's work include Neuroinflammation and Neurodegeneration Mechanisms (4 papers), Immune cells in cancer (2 papers) and Adenosine and Purinergic Signaling (1 paper). Leihua Weng is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (4 papers), Immune cells in cancer (2 papers) and Adenosine and Purinergic Signaling (1 paper). Leihua Weng collaborates with scholars based in China, United States and Hong Kong. Leihua Weng's co-authors include Yun Xu, Xiang Cao, Hailan Meng, Xiaoying Wang, Haoran Zhao, Lai Qian, Min‐Sheng Zhu, Yi Liu, He Zhang and Junwei Hao and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Scientific Reports.

In The Last Decade

Leihua Weng

10 papers receiving 442 citations

Peers

Leihua Weng

NEURO

MB

IMMUN

PHYSI

NEURO

Brittany K. Matlock United States

Cun‐Jin Zhang China

Dong Gil Lee South Korea

Shota Tanaka Japan

Sun‐Ryung Lee South Korea

Janet E. Holley United Kingdom

Yong-Ping Wu China

Erika Daniela González-Renovato Mexico

Mitsumasa Watanabe Japan

Brittany K. Matlock United States

Leihua Weng

81 ×0.5

NEURO

300 ×2.3

MB

95 ×0.8

IMMUN

74 ×1.3

PHYSI

12 ×0.2

NEURO

Citations per year, relative to Leihua Weng Leihua Weng (= 1×) peers Brittany K. Matlock

Countries citing papers authored by Leihua Weng

Since Specialization

Citations

This map shows the geographic impact of Leihua Weng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leihua Weng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leihua Weng more than expected).

Fields of papers citing papers by Leihua Weng

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Leihua Weng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leihua Weng. The network helps show where Leihua Weng may publish in the future.

Co-authorship network of co-authors of Leihua Weng

This figure shows the co-authorship network connecting the top 25 collaborators of Leihua Weng. A scholar is included among the top collaborators of Leihua Weng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Leihua Weng. Leihua Weng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Sun, Guangyi, Hongxing Liu, Leihua Weng, et al.. (2025). A brain-computer interface roadmap for diagnosing and treating neurological disorders. 1(1). 100016–100016.

2.

Zhang, J, et al.. (2020). [Evaluation of human milk feeding in hospitalized very low and extremely low birth weight infants].. PubMed. 58(5). 387–391. 4 indexed citations

3.

Meng, Hailan, Haoran Zhao, Xiang Cao, et al.. (2019). Double-negative T cells remarkably promote neuroinflammation after ischemic stroke. Proceedings of the National Academy of Sciences. 116(12). 5558–5563. 138 indexed citations

4.

Weng, Leihua, Xiang Cao, Li‐Juan Han, et al.. (2017). Association of increased Treg and Th17 with pathogenesis of moyamoya disease. Scientific Reports. 7(1). 44 indexed citations

5.

Wu, Jun, et al.. (2016). Valproic acid protects against MPP+-mediated neurotoxicity in SH-SY5Y Cells through autophagy. Neuroscience Letters. 638. 60–68. 21 indexed citations

6.

Weng, Leihua, He Zhang, Xiaoxi Li, et al.. (2016). Ampelopsin attenuates lipopolysaccharide-induced inflammatory response through the inhibition of the NF-κB and JAK2/STAT3 signaling pathways in microglia. International Immunopharmacology. 44. 1–8. 69 indexed citations

7.

Weng, Leihua, Zhenhua Wu, Weihong Zheng, et al.. (2016). Malibatol A enhances alternative activation of microglia by inhibiting phosphorylation of Mammalian Ste20-like kinase1 in OGD-BV-2 cells. Neurological Research. 38(4). 342–348. 17 indexed citations

8.

Yan, Jun, Yun Xu, Li Zhang, et al.. (2016). Increased Expressions of Plasma Galectin-3 in Patients with Amyotrophic Lateral Sclerosis. Chinese Medical Journal. 129(23). 2797–2803. 21 indexed citations

9.

Zhang, Zhuo, Shenyang Zhang, Hui Yang, et al.. (2014). Human umbilical cord mesenchymal stem cells protect against ischemic brain injury in mouse by regulating peripheral immunoinflammation. Brain Research. 1594. 293–304. 53 indexed citations

10.

Nie, Yunzhong, Yongbin Mou, Leihua Weng, et al.. (2013). Low Frequency Magnetic Fields Enhance Antitumor Immune Response against Mouse H22 Hepatocellular Carcinoma. PLoS ONE. 8(11). e72411–e72411. 26 indexed citations

11.

Nie, Yunzhong, Leilei Du, Yongbin Mou, et al.. (2013). Effect of low frequency magnetic fields on melanoma: tumor inhibition and immune modulation. BMC Cancer. 13(1). 582–582. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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