Léa P. Wilhelm

682 total citations
10 papers, 457 citations indexed

About

Léa P. Wilhelm is a scholar working on Molecular Biology, Epidemiology and Cell Biology. According to data from OpenAlex, Léa P. Wilhelm has authored 10 papers receiving a total of 457 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Epidemiology and 5 papers in Cell Biology. Recurrent topics in Léa P. Wilhelm's work include Autophagy in Disease and Therapy (5 papers), Cellular transport and secretion (4 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Léa P. Wilhelm is often cited by papers focused on Autophagy in Disease and Therapy (5 papers), Cellular transport and secretion (4 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Léa P. Wilhelm collaborates with scholars based in United Kingdom, France and United States. Léa P. Wilhelm's co-authors include Fabien Alpy, Catherine Tomasetto, Toshihide Kobayashi, Corinne Wendling, Guillaume Drin, Benoı̂t Vedie, Marie‐Pierre Chenard, Ian G. Ganley, Yves Nominé and Danièle Spehner and has published in prestigious journals such as SHILAP Revista de lepidopterología, The EMBO Journal and Journal of Medicinal Chemistry.

In The Last Decade

Léa P. Wilhelm

10 papers receiving 456 citations

Peers

Léa P. Wilhelm

MB

CB

EPIDE

PHYSI

SURGE

Marielle Boonen Belgium

Risa Mukai Japan

Birol Cabukusta Netherlands

Wonyul Jang Germany

Adriano Vissa Canada

Mary Grace Lin United States

Saskia Heybrock Germany

Selma Y. Dejgaard Canada

Daxiao Sun China

Cheryl L. Meyerkord United States

Marielle Boonen Belgium

Léa P. Wilhelm

282 ×1.0

MB

225 ×1.3

CB

68 ×0.7

EPIDE

127 ×1.7

PHYSI

32 ×0.6

SURGE

Citations per year, relative to Léa P. Wilhelm Léa P. Wilhelm (= 1×) peers Marielle Boonen

Countries citing papers authored by Léa P. Wilhelm

Since Specialization

Citations

This map shows the geographic impact of Léa P. Wilhelm's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Léa P. Wilhelm with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Léa P. Wilhelm more than expected).

Fields of papers citing papers by Léa P. Wilhelm

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Léa P. Wilhelm. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Léa P. Wilhelm. The network helps show where Léa P. Wilhelm may publish in the future.

Co-authorship network of co-authors of Léa P. Wilhelm

This figure shows the co-authorship network connecting the top 25 collaborators of Léa P. Wilhelm. A scholar is included among the top collaborators of Léa P. Wilhelm based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Léa P. Wilhelm. Léa P. Wilhelm is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Singh, Pawan, Shalini Agarwal, Léa P. Wilhelm, et al.. (2025). Kinome screening identifies integrated stress response kinase EIF2AK1/HRI as a negative regulator of PINK1 mitophagy signaling. Science Advances. 11(19). eadn2528–eadn2528. 1 indexed citations

2.

Singh, François, Léa P. Wilhelm, Alan R. Prescott, et al.. (2024). PINK1 regulated mitophagy is evident in skeletal muscles. SHILAP Revista de lepidopterología. 3(1). 2326402–2326402. 11 indexed citations

3.

Bell, Shane, Léa P. Wilhelm, Oliver M. Russell, et al.. (2023). PINK1-Dependent Mitophagy Inhibits Elevated Ubiquitin Phosphorylation Caused by Mitochondrial Damage. Journal of Medicinal Chemistry. 66(11). 7645–7656. 13 indexed citations

4.

Wilhelm, Léa P., Juan Zapata‐Muñoz, Beatriz Villarejo‐Zori, et al.. (2022). BNIP3L / NIX regulates both mitophagy and pexophagy. The EMBO Journal. 41(24). e111115–e111115. 59 indexed citations

5.

Wilhelm, Léa P. & Ian G. Ganley. (2022). Mitochondria and peroxisomes: partners in autophagy. Autophagy. 19(7). 2162–2163. 8 indexed citations

6.

Wilhelm, Léa P., et al.. (2019). Intracellular and Plasma Membrane Cholesterol Labeling and Quantification Using Filipin and GFP-D4. Methods in molecular biology. 1949. 137–152. 45 indexed citations

7.

Mattia, Thomas Di, Léa P. Wilhelm, Souade Ikhlef, et al.. (2018). Identification of MOSPD2, a novel scaffold for endoplasmic reticulum membrane contact sites. EMBO Reports. 19(7). 92 indexed citations

8.

Wilhelm, Léa P., Corinne Wendling, Benoı̂t Vedie, et al.. (2017). STARD 3 mediates endoplasmic reticulum‐to‐endosome cholesterol transport at membrane contact sites. The EMBO Journal. 36(10). 1412–1433. 202 indexed citations

9.

Wilhelm, Léa P., Catherine Tomasetto, & Fabien Alpy. (2016). Touché! STARD3 and STARD3NL tether the ER to endosomes. Biochemical Society Transactions. 44(2). 493–498. 18 indexed citations

10.

Rousseau, Adrien, Léa P. Wilhelm, Catherine Tomasetto, & Fabien Alpy. (2014). The phosphoinositide-binding protein TRAF4 modulates tight junction stability and migration of cancer cells. Tissue Barriers. 2(4). e975597–e975597. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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