Lars Wärngård

965 total citations
35 papers, 790 citations indexed

About

Lars Wärngård is a scholar working on Health, Toxicology and Mutagenesis, Cancer Research and Molecular Biology. According to data from OpenAlex, Lars Wärngård has authored 35 papers receiving a total of 790 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Health, Toxicology and Mutagenesis, 20 papers in Cancer Research and 19 papers in Molecular Biology. Recurrent topics in Lars Wärngård's work include Carcinogens and Genotoxicity Assessment (18 papers), Toxic Organic Pollutants Impact (18 papers) and Effects and risks of endocrine disrupting chemicals (8 papers). Lars Wärngård is often cited by papers focused on Carcinogens and Genotoxicity Assessment (18 papers), Toxic Organic Pollutants Impact (18 papers) and Effects and risks of endocrine disrupting chemicals (8 papers). Lars Wärngård collaborates with scholars based in Sweden, Japan and United States. Lars Wärngård's co-authors include Sten Flodström, H. G. Hemming, Ronny Fransson-Steen, Ulf G. Ahlborg, Kerstin Kenne, Ian A. Cotgreave, Yoshihisa Kato, Tony Kronevi, Heike Hellmold and Tim Hofer and has published in prestigious journals such as Free Radical Biology and Medicine, Chemosphere and International Journal of Cancer.

In The Last Decade

Lars Wärngård

35 papers receiving 743 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lars Wärngård Sweden 19 387 336 312 71 70 35 790
Michael Schwarz Germany 16 385 1.0× 365 1.1× 301 1.0× 40 0.6× 74 1.1× 23 928
Pavel Krčmář Czechia 19 300 0.8× 310 0.9× 223 0.7× 47 0.7× 39 0.6× 29 744
Donna R. Galeazzi United States 13 373 1.0× 403 1.2× 296 0.9× 59 0.8× 144 2.1× 15 874
Sten Flodström Sweden 15 339 0.9× 161 0.5× 269 0.9× 58 0.8× 48 0.7× 20 516
J HASEMAN United States 11 499 1.3× 156 0.5× 372 1.2× 57 0.8× 50 0.7× 14 932
Giocondo Lorenzon France 13 236 0.6× 373 1.1× 285 0.9× 133 1.9× 70 1.0× 23 980
V. Morrison United States 13 224 0.6× 169 0.5× 209 0.7× 32 0.5× 84 1.2× 20 520
Willy A. Solis United States 10 500 1.3× 437 1.3× 281 0.9× 40 0.6× 139 2.0× 14 1.1k
Gregory L. Erexson United States 18 221 0.6× 345 1.0× 552 1.8× 144 2.0× 42 0.6× 36 806
Franz Kiefer Germany 12 212 0.5× 337 1.0× 231 0.7× 37 0.5× 182 2.6× 22 683

Countries citing papers authored by Lars Wärngård

Since Specialization
Citations

This map shows the geographic impact of Lars Wärngård's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lars Wärngård with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lars Wärngård more than expected).

Fields of papers citing papers by Lars Wärngård

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lars Wärngård. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lars Wärngård. The network helps show where Lars Wärngård may publish in the future.

Co-authorship network of co-authors of Lars Wärngård

This figure shows the co-authorship network connecting the top 25 collaborators of Lars Wärngård. A scholar is included among the top collaborators of Lars Wärngård based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lars Wärngård. Lars Wärngård is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Johansson, Inger, et al.. (2004). Differentiation of human hepatoma cells during confluence as revealed by gene expression profiling. Biochemical Pharmacology. 67(7). 1249–1258. 21 indexed citations
2.
Bajak, Edyta, et al.. (2002). Protein S-glutathionylation correlates to selective stress gene expression and cytoprotection. Archives of Biochemistry and Biophysics. 406(2). 241–252. 22 indexed citations
3.
Vondracek, Martin, David A. Weaver, Jesper J. Hedberg, et al.. (2002). Transcript profiling of enzymes involved in detoxification of xenobiotics and reactive oxygen in human normal and simian virus 40 T antigen‐immortalized oral keratinocytes. International Journal of Cancer. 99(6). 776–782. 33 indexed citations
4.
Hellmold, Heike, Charlotte B. Nilsson, Ina Schuppe‐Koistinen, Kerstin Kenne, & Lars Wärngård. (2002). Identification of end points relevant to detection of potentially adverse drug reactions. Toxicology Letters. 127(1-3). 239–243. 8 indexed citations
5.
Hedberg, Jesper J., et al.. (2001). Micro-array chip analysis of carbonyl-metabolising enzymes in normal, immortalised and malignant human oral keratinocytes. Cellular and Molecular Life Sciences. 58(11). 1719–1725. 18 indexed citations
6.
Wärngård, Lars, et al.. (1999). Induction of Altered Hepatic Foci by a Mixture of Dioxin-like Compounds with and without 2,2′,4,4′,5,5′-Hexachlorobiphenyl in Female Sprague–Dawley Rats. Toxicology and Applied Pharmacology. 156(1). 30–39. 13 indexed citations
7.
Johansson, Niklas, et al.. (1998). Interactive Effects of Three Structurally Different Polychlorinated Biphenyls in a Rat Liver Tumor Promotion Bioassay. Toxicology and Applied Pharmacology. 152(1). 153–165. 28 indexed citations
8.
Kato, Yoshihisa, Kerstin Kenne, Koichi Haraguchi, et al.. (1998). Inhibition of cell-cell communication by methylsulfonyl metabolites of polychlorinated biphenyl congeners in rat liver epithelial IAR 20 cells. Archives of Toxicology. 72(3). 178–182. 15 indexed citations
9.
Kato, Yoshihisa, et al.. (1997). Promotion of Enzyme Altered Foci in Female Rat Livers by 2,3,3′,4,4′,5-Hexachlorobiphenyl. Toxicology and Applied Pharmacology. 147(1). 46–55. 9 indexed citations
10.
Hemming, H. G., et al.. (1995). Liver tumour promoting activity of 3,4,5,3′,4′-pentachlorobiphenyl and its interaction with 2,3,7,8-tetrachlorodibenzo-p-dioxin. European Journal of Pharmacology Environmental Toxicology and Pharmacology. 292(3-4). 241–249. 22 indexed citations
11.
Hemming, H. G., et al.. (1995). Interaction of 3,4,5,3′,4′‐Pentachlorobiphenyl and 2,4,5,2′,4′,5′‐Hexachlorobiphenyl in Promotion of Altered Hepatic Foci in Rats. Pharmacology & Toxicology. 77(2). 149–154. 16 indexed citations
12.
13.
14.
Fransson-Steen, Ronny, Sten Flodström, & Lars Wärngård. (1992). The insecticide endosulfan and its two stereoisomers promote the growth of altered hepatic foci in rats. Carcinogenesis. 13(12). 2299–2303. 15 indexed citations
15.
Fransson-Steen, Ronny & Lars Wärngård. (1992). Inhibitory effects of endosulfan on gap junctional intercellular communication in WB-F344 rat liver cells and primary rat hepatocytes. Carcinogenesis. 13(4). 657–662. 8 indexed citations
16.
Hemming, H. G., Lars Wärngård, & Ulf G. Ahlborg. (1991). Inhibition of Dye Transfer in Rat Liver WB Cell Culture by Polychlorinated Biphenyls. Pharmacology & Toxicology. 69(6). 416–420. 24 indexed citations
17.
Ruch, Randall J., et al.. (1990). Inhibition of hepatocyte gap junctional intercellular communication by endosulfan, chlordane and heptachlor. Carcinogenesis. 11(7). 1097–1101. 37 indexed citations
18.
Wärngård, Lars, et al.. (1989). Mechanistic studies on the DDT-induced inhibition of intercellular communication. Carcinogenesis. 10(3). 471–476. 21 indexed citations
19.
Flodström, Sten, Lars Wärngård, H. G. Hemming, & Ulf G. Ahlborg. (1988). Chlorobenzilate-induced effects on enzyme-altered foci in rat liver and intercellular communication in rat liver WB-F344 epithelial cells. Cancer Letters. 43(3). 161–166. 5 indexed citations
20.
Wärngård, Lars, et al.. (1988). Calmodulin involvement in TPA and DDT induced inhibition of intercellular communication. Chemico-Biological Interactions. 65(1). 41–49. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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