Kun Ling

1.7k total citations
52 papers, 1.3k citations indexed

About

Kun Ling is a scholar working on Nephrology, Surgery and Molecular Biology. According to data from OpenAlex, Kun Ling has authored 52 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Nephrology, 15 papers in Surgery and 15 papers in Molecular Biology. Recurrent topics in Kun Ling's work include Renal Diseases and Glomerulopathies (10 papers), Extracellular vesicles in disease (8 papers) and Chronic Kidney Disease and Diabetes (7 papers). Kun Ling is often cited by papers focused on Renal Diseases and Glomerulopathies (10 papers), Extracellular vesicles in disease (8 papers) and Chronic Kidney Disease and Diabetes (7 papers). Kun Ling collaborates with scholars based in China, United Kingdom and Thailand. Kun Ling's co-authors include Bi Cheng Liu, Xiong Z. Ruan, Ze Bo Hu, Chen Lu, Jaffar Nourooz‐Zadeh, Yang Zhang, Simon P. Wolff, Pei Pei Chen, Jian Lü and Gui Hua Wang and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Biochemical Journal.

In The Last Decade

Kun Ling

49 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kun Ling China 20 433 236 232 198 186 52 1.3k
Hassan Argani Iran 25 336 0.8× 289 1.2× 283 1.2× 271 1.4× 164 0.9× 113 1.8k
Tso-Hsiao Chen Taiwan 20 592 1.4× 207 0.9× 328 1.4× 224 1.1× 136 0.7× 56 1.6k
Wenshan Lv China 14 410 0.9× 149 0.6× 288 1.2× 143 0.7× 192 1.0× 32 1.5k
Juan Chen China 25 506 1.2× 182 0.8× 118 0.5× 171 0.9× 240 1.3× 95 1.5k
Romina di Giuseppe Germany 27 324 0.7× 163 0.7× 217 0.9× 120 0.6× 351 1.9× 71 1.9k
Nadereh Rashtchizadeh Iran 23 391 0.9× 185 0.8× 111 0.5× 162 0.8× 78 0.4× 91 1.5k
Manuela Ciocoiu Romania 17 549 1.3× 172 0.7× 104 0.4× 358 1.8× 477 2.6× 98 1.7k
Qinghua Wu China 22 436 1.0× 223 0.9× 90 0.4× 109 0.6× 188 1.0× 108 1.5k

Countries citing papers authored by Kun Ling

Since Specialization
Citations

This map shows the geographic impact of Kun Ling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kun Ling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kun Ling more than expected).

Fields of papers citing papers by Kun Ling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kun Ling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kun Ling. The network helps show where Kun Ling may publish in the future.

Co-authorship network of co-authors of Kun Ling

This figure shows the co-authorship network connecting the top 25 collaborators of Kun Ling. A scholar is included among the top collaborators of Kun Ling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kun Ling. Kun Ling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Jin-Liang, et al.. (2025). Lag formation control for nonlinear second-order multiagent systems without and with external disturbances. Neurocomputing. 627. 129547–129547.
2.
Tang, Hong, et al.. (2025). Assessment of the efficacy of early versus delayed mobility exercise after arthroscopic rotator cuff repair. International Orthopaedics. 49(6). 1411–1420.
3.
Ling, Kun, et al.. (2024). Formation control and lag formation control for human–machine interaction second-order multiagent systems. Neurocomputing. 610. 128603–128603. 4 indexed citations
4.
Wang, Tian Xiang, et al.. (2024). ITGAM-mediated macrophages contribute to basement membrane damage in diabetic nephropathy and atherosclerosis. BMC Nephrology. 25(1). 72–72. 4 indexed citations
5.
Chen, Pei Pei, et al.. (2024). Deficiency of thiosulfate sulfurtransferase mediates the dysfunction of renal tubular mitochondrial fatty acid oxidation in diabetic kidney disease. Cell Death and Differentiation. 31(12). 1636–1649. 9 indexed citations
6.
Chen, Pei Pei, Liang Liu, Gui Hua Wang, et al.. (2023). Activation of acetyl-CoA synthetase 2 mediates kidney injury in diabetic nephropathy. JCI Insight. 8(20). 13 indexed citations
7.
Zhao, Qing, et al.. (2023). Application of cloud server-based machine learning for assisting pathological structure recognition in IgA nephropathy. Journal of Clinical Pathology. 78(2). 128–134. 1 indexed citations
8.
Chen, Pei Pei, Liang Li, Liang Liu, et al.. (2023). Outer membrane vesicles derived from gut microbiota mediate tubulointerstitial inflammation: a potential new mechanism for diabetic kidney disease. Theranostics. 13(12). 3988–4003. 34 indexed citations
9.
Liu, Bi Cheng, et al.. (2022). Intestinal microbiota-derived membrane vesicles and their role in chronic kidney disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1868(10). 166478–166478. 7 indexed citations
10.
Lü, Jian, Pei Pei Chen, Gui Hua Wang, et al.. (2021). GPR43 deficiency protects against podocyte insulin resistance in diabetic nephropathy through the restoration of AMPKα activity. Theranostics. 11(10). 4728–4742. 66 indexed citations
11.
Lü, Jian, et al.. (2019). Urinary podocyte microparticles are associated with disease activity and renal injury in systemic lupus erythematosus. BMC Nephrology. 20(1). 303–303. 23 indexed citations
12.
Ling, Kun, Liang Liu, Yang Zhang, et al.. (2019). Aspirin attenuates podocyte injury in diabetic rats through overriding cyclooxygenase-2-mediated dysregulation of LDL receptor pathway. International Urology and Nephrology. 51(3). 551–558. 7 indexed citations
13.
Ling, Kun, Ze Bo Hu, Yang Zhang, et al.. (2018). Lipoprotein(a) accelerated the progression of atherosclerosis in patients with end-stage renal disease. BMC Nephrology. 19(1). 192–192. 16 indexed citations
14.
Lu, Chen, Kun Ling, Xiong Z. Ruan, & Bi Cheng Liu. (2017). The Emerging Roles of Microparticles in Diabetic Nephropathy. International Journal of Biological Sciences. 13(9). 1118–1125. 25 indexed citations
15.
16.
Liu, Jing, Kun Ling, Yang Zhang, et al.. (2015). Activation of mTORC1 disrupted LDL receptor pathway: A potential new mechanism for the progression of non-alcoholic fatty liver disease. The International Journal of Biochemistry & Cell Biology. 61. 8–19. 42 indexed citations
17.
Ling, Kun, Jing Liu, Jie Ni, et al.. (2013). Inflammatory Stress Exacerbates the Progression of Cardiac Fibrosis in High-Fat-Fed Apolipoprotein E Knockout Mice via Endothelial-Mesenchymal Transition. International Journal of Medical Sciences. 10(4). 420–426. 39 indexed citations
18.
Ling, Kun, et al.. (2013). Analysis of the spectrum and antibiotic resistance of uropathogens in vitro: Results based on a retrospective study from a tertiary hospital. American Journal of Infection Control. 41(7). 601–606. 19 indexed citations
19.
20.
Ling, Julia, Koon Wing Chan, Kun Ling, & G.L. French. (1988). BASIC programs to analyse minimal inhibitory concentration (MIC) antimicrobial susceptibility results. Computer Methods and Programs in Biomedicine. 26(1). 97–100. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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