K.T. Lee

1.4k total citations
55 papers, 1.2k citations indexed

About

K.T. Lee is a scholar working on Surgery, Molecular Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, K.T. Lee has authored 55 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Surgery, 18 papers in Molecular Biology and 12 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in K.T. Lee's work include Cholesterol and Lipid Metabolism (14 papers), Peroxisome Proliferator-Activated Receptors (12 papers) and Atherosclerosis and Cardiovascular Diseases (10 papers). K.T. Lee is often cited by papers focused on Cholesterol and Lipid Metabolism (14 papers), Peroxisome Proliferator-Activated Receptors (12 papers) and Atherosclerosis and Cardiovascular Diseases (10 papers). K.T. Lee collaborates with scholars based in United States, Uganda and Nigeria. K.T. Lee's co-authors include W.A. Thomas, D.N. Kim, Johannes Reiner, R.A. Florentin, S.C. Nam, H. Imai, Josef Schmee, J. Jarmolych, R.F. Scott and A.S. Daoud and has published in prestigious journals such as The Lancet, SHILAP Revista de lepidopterología and Journal of Lipid Research.

In The Last Decade

K.T. Lee

54 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
K.T. Lee United States 20 390 316 251 240 181 55 1.2k
W.A. Thomas United States 26 620 1.6× 475 1.5× 443 1.8× 315 1.3× 387 2.1× 88 1.9k
Johannes Reiner United States 19 241 0.6× 296 0.9× 164 0.7× 76 0.3× 163 0.9× 59 885
Noriyuki Homma Japan 18 168 0.4× 413 1.3× 36 0.1× 225 0.9× 84 0.5× 44 1.2k
Meritxell Nus Spain 19 119 0.3× 281 0.9× 422 1.7× 157 0.7× 132 0.7× 38 965
Hiroshi Kanamori Japan 20 266 0.7× 555 1.8× 601 2.4× 102 0.4× 42 0.2× 44 1.6k
Fons K. Verheyen Netherlands 14 195 0.5× 380 1.2× 117 0.5× 224 0.9× 68 0.4× 17 902
Norman A. Granholm United States 17 278 0.7× 281 0.9× 233 0.9× 40 0.2× 75 0.4× 34 918
Vanessa Legry Belgium 13 311 0.8× 254 0.8× 40 0.2× 85 0.4× 107 0.6× 25 970
P G Quinn United States 15 225 0.6× 768 2.4× 140 0.6× 93 0.4× 31 0.2× 17 1.5k
Patricia G. Phillips United States 20 72 0.2× 346 1.1× 168 0.7× 72 0.3× 46 0.3× 40 952

Countries citing papers authored by K.T. Lee

Since Specialization
Citations

This map shows the geographic impact of K.T. Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K.T. Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K.T. Lee more than expected).

Fields of papers citing papers by K.T. Lee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K.T. Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K.T. Lee. The network helps show where K.T. Lee may publish in the future.

Co-authorship network of co-authors of K.T. Lee

This figure shows the co-authorship network connecting the top 25 collaborators of K.T. Lee. A scholar is included among the top collaborators of K.T. Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K.T. Lee. K.T. Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ashton, Katie A., K.T. Lee, Omkar L. Patkar, et al.. (2024). Biallelic SLC13A1 loss-of-function variants result in impaired sulfate transport and skeletal phenotypes, including short stature, scoliosis, and skeletal dysplasia. SHILAP Revista de lepidopterología. 3. 101958–101958.
3.
Lee, K.T., et al.. (1988). Clonal nature of atherosclerotic plaques. Experimental and Molecular Pathology. 48(3). 391–402. 4 indexed citations
4.
Kim, D.N., H. Imai, Josef Schmee, K.T. Lee, & W.A. Thomas. (1985). Intimal cell mass-derived atherosclerotic lesions in the abdominal aorta of hyperlipidemic swine Part 1. Cell of origin, cell divisions and cell losses in first 90 days on diet. Atherosclerosis. 56(2). 169–188. 29 indexed citations
5.
Kim, D.N., K.T. Lee, Josef Schmee, & W.A. Thomas. (1984). Quantification of intimal cell masses and atherosclerotic lesions in coronary arteries of control and hyperlipidemic swine. Atherosclerosis. 52(1). 115–122. 15 indexed citations
6.
Thomas, W.A., R.F. Scott, R.A. Florentin, Johannes Reiner, & K.T. Lee. (1981). Population dynamics of arterial cells during atherogenesis. Experimental and Molecular Pathology. 35(2). 153–162. 14 indexed citations
7.
Kim, D.N., et al.. (1980). Effect of clofibrate, cholestyramine, cholesterol and feeding pattern on the diurnal variation of cholesterol 7 alpha-hydroxylation in swine. Experimental and Molecular Pathology. 32(1). 52–60. 4 indexed citations
9.
Kim, D.N., David Rogers, Johannes Reiner, K.T. Lee, & W.A. Thomas. (1976). Lack of effect of clofibrate on hepatic HMG-CoA reductase activity in young swine in the postabsorptive state. Experimental and Molecular Pathology. 25(3). 301–310. 7 indexed citations
10.
Thomas, W.A., et al.. (1976). Alterations in population dynamics of arterial smooth muscle cells during atherogenesis. Experimental and Molecular Pathology. 24(2). 244–260. 34 indexed citations
11.
Kim, D.N., et al.. (1976). Effects of lipids on the stability of lysosomes in vitro. Experimental and Molecular Pathology. 25(2). 131–141. 4 indexed citations
12.
Lee, K.T., et al.. (1975). Advanced coronary atherosclerosis in swine produced by combination of balloon-catheter injury and cholesterol feeding. Experimental and Molecular Pathology. 23(3). 491–499. 55 indexed citations
13.
Kim, D.N., K.T. Lee, Johannes Reiner, & W.A. Thomas. (1975). An evaluation of some of the potential immediate sources of cholesterol for bile acid synthesis in swine. Experimental and Molecular Pathology. 22(2). 284–293. 2 indexed citations
14.
Kim, D.N., et al.. (1975). Effects of lipids on the stability of lysosomes in vitro. Experimental and Molecular Pathology. 23(2). 266–275. 5 indexed citations
15.
Kim, D.N., K.T. Lee, Johannes Reiner, & W.A. Thomas. (1974). Restraint of cholesterol accumulation in tissue pools associated with drastic short-term lowering of serum cholesterol levels by clofibrate or cholestyramine in hypercholesterolemic swine. Journal of Lipid Research. 15(4). 326–331. 37 indexed citations
16.
Nam, S.C., R.A. Florentin, K. Janakidevi, et al.. (1974). Population dynamics of arterial smooth muscle cells. Experimental and Molecular Pathology. 21(2). 259–267. 19 indexed citations
17.
Nam, S.C., et al.. (1973). Rapid production of advanced atherosclerosis in swine by a combination of endothelial injury and cholesterol feeding. Experimental and Molecular Pathology. 18(3). 369–379. 68 indexed citations
18.
Lee, K.T., J. Jarmolych, D.N. Kim, et al.. (1971). Production of advanced coronary atherosclerosis, myocardial infarction and “sudden death” in swine. Experimental and Molecular Pathology. 15(2). 170–190. 45 indexed citations
20.
Goodale, F., A.S. Daoud, R.A. Florentin, K.T. Lee, & Alan M. Gittelsohn. (1962). Chemico-anatomic studies of arteriosclerosis and thrombosis in diabetics. Experimental and Molecular Pathology. 1(4). 353–363. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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