Kostas Paschos

734 total citations
13 papers, 581 citations indexed

About

Kostas Paschos is a scholar working on Oncology, Molecular Biology and Immunology. According to data from OpenAlex, Kostas Paschos has authored 13 papers receiving a total of 581 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Oncology, 6 papers in Molecular Biology and 4 papers in Immunology. Recurrent topics in Kostas Paschos's work include Viral-associated cancers and disorders (11 papers), Immune Cell Function and Interaction (4 papers) and Epigenetics and DNA Methylation (3 papers). Kostas Paschos is often cited by papers focused on Viral-associated cancers and disorders (11 papers), Immune Cell Function and Interaction (4 papers) and Epigenetics and DNA Methylation (3 papers). Kostas Paschos collaborates with scholars based in United Kingdom, Netherlands and Germany. Kostas Paschos's co-authors include Martin J. Allday, Robert E. White, Gillian A. Parker, Emma Anderton, Quentin Bazot, Christine T. Styles, Jaap M. Middeldorp, Paul Smith, Lenka Skalska and Jens Kalchschmidt and has published in prestigious journals such as Nucleic Acids Research, The Journal of Experimental Medicine and Journal of Virology.

In The Last Decade

Kostas Paschos

13 papers receiving 580 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kostas Paschos United Kingdom 11 453 199 189 177 155 13 581
Stacy R. Hagemeier United States 11 395 0.9× 115 0.6× 120 0.6× 171 1.0× 227 1.5× 13 549
Toni Portis United States 11 337 0.7× 216 1.1× 291 1.5× 97 0.5× 89 0.6× 12 588
Jillian A. Bristol United States 13 276 0.6× 145 0.7× 121 0.6× 89 0.5× 128 0.8× 16 408
Eri Seto Japan 9 408 0.9× 112 0.6× 135 0.7× 202 1.1× 147 0.9× 12 596
Q Zhang United States 5 347 0.8× 100 0.5× 80 0.4× 121 0.7× 97 0.6× 8 403
Rémy Poirey Germany 13 289 0.6× 125 0.6× 103 0.5× 172 1.0× 97 0.6× 20 467
Anja M. Mehl United Kingdom 8 238 0.5× 124 0.6× 233 1.2× 73 0.4× 167 1.1× 11 482
Quentin Bazot United Kingdom 11 258 0.6× 120 0.6× 107 0.6× 92 0.5× 117 0.8× 16 343
Shidong Ma United States 10 264 0.6× 97 0.5× 189 1.0× 102 0.6× 125 0.8× 12 453
M Woisetschlaeger United States 8 584 1.3× 262 1.3× 140 0.7× 197 1.1× 232 1.5× 9 801

Countries citing papers authored by Kostas Paschos

Since Specialization
Citations

This map shows the geographic impact of Kostas Paschos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kostas Paschos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kostas Paschos more than expected).

Fields of papers citing papers by Kostas Paschos

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kostas Paschos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kostas Paschos. The network helps show where Kostas Paschos may publish in the future.

Co-authorship network of co-authors of Kostas Paschos

This figure shows the co-authorship network connecting the top 25 collaborators of Kostas Paschos. A scholar is included among the top collaborators of Kostas Paschos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kostas Paschos. Kostas Paschos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Paschos, Kostas, Quentin Bazot, Jonathan Lees, Paul J. Farrell, & Martin J. Allday. (2019). Requirement for PRC1 subunit BMI1 in host gene activation by Epstein–Barr virus protein EBNA3C. Nucleic Acids Research. 47(6). 2807–2821. 3 indexed citations
2.
Bazot, Quentin, Kostas Paschos, & Martin J. Allday. (2018). Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV. Journal of Virology. 92(7). 12 indexed citations
3.
Styles, Christine T., Kostas Paschos, Robert E. White, & Paul J. Farrell. (2018). The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency. Pathogens. 7(1). 31–31. 27 indexed citations
4.
Styles, Christine T., Quentin Bazot, Gillian A. Parker, et al.. (2017). EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency. PLoS Biology. 15(8). e2001992–e2001992. 50 indexed citations
5.
Kalchschmidt, Jens, et al.. (2016). EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells. PLoS Pathogens. 12(1). e1005383–e1005383. 22 indexed citations
6.
Paschos, Kostas, Quentin Bazot, Gillian A. Parker, et al.. (2016). Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression. Nucleic Acids Research. 45(5). 2368–2383. 17 indexed citations
7.
Kalchschmidt, Jens, Rachael Bashford-Rogers, Kostas Paschos, et al.. (2016). Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells. The Journal of Experimental Medicine. 213(6). 921–928. 59 indexed citations
8.
Bazot, Quentin, Kostas Paschos, Lenka Skalska, et al.. (2015). Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2. PLoS Pathogens. 11(7). e1005031–e1005031. 37 indexed citations
9.
Skalska, Lenka, Robert E. White, Gillian A. Parker, et al.. (2013). Induction of p16INK4a Is the Major Barrier to Proliferation when Epstein-Barr Virus (EBV) Transforms Primary B Cells into Lymphoblastoid Cell Lines. PLoS Pathogens. 9(2). e1003187–e1003187. 74 indexed citations
10.
Skalska, Lenka, Robert E. White, Gillian A. Parker, et al.. (2013). Correction: Induction of p16INK4aIs the Major Barrier to Proliferation when Epstein-Barr Virus (EBV) Transforms Primary B Cells into Lymphoblastoid Cell Lines. PLoS Pathogens. 9(3). 9 indexed citations
11.
Paschos, Kostas, et al.. (2012). BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV. Nucleic Acids Research. 40(15). 7233–7246. 95 indexed citations
12.
Paschos, Kostas, Paul Smith, Emma Anderton, et al.. (2009). Epstein-Barr Virus Latency in B Cells Leads to Epigenetic Repression and CpG Methylation of the Tumour Suppressor Gene Bim. PLoS Pathogens. 5(6). e1000492–e1000492. 135 indexed citations
13.
Young, Paul R., Emma Anderton, Kostas Paschos, Robert E. White, & Martin J. Allday. (2008). Epstein–Barr virus nuclear antigen (EBNA) 3A induces the expression of and interacts with a subset of chaperones and co-chaperones. Journal of General Virology. 89(4). 866–877. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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