Klaus Dittmann

4.4k total citations
61 papers, 3.3k citations indexed

About

Klaus Dittmann is a scholar working on Molecular Biology, Oncology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Klaus Dittmann has authored 61 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Molecular Biology, 27 papers in Oncology and 12 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Klaus Dittmann's work include DNA Repair Mechanisms (18 papers), Cancer-related Molecular Pathways (14 papers) and Lung Cancer Treatments and Mutations (9 papers). Klaus Dittmann is often cited by papers focused on DNA Repair Mechanisms (18 papers), Cancer-related Molecular Pathways (14 papers) and Lung Cancer Treatments and Mutations (9 papers). Klaus Dittmann collaborates with scholars based in Germany, United States and Japan. Klaus Dittmann's co-authors include H. Peter Rodemann, Claus Mayer, H. Peter Rodemann, Rainer Kehlbach, Mahmoud Toulany, Michaël Baumann, Klaus Bayreuther, Pal I. Francz, Martin Schaller and Birgit Fehrenbacher and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Klaus Dittmann

60 papers receiving 3.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Klaus Dittmann Germany 29 1.8k 1.3k 911 512 439 61 3.3k
H. Peter Rodemann Germany 29 1.8k 1.0× 979 0.7× 663 0.7× 560 1.1× 358 0.8× 56 3.0k
Esther Zwick Germany 11 2.3k 1.3× 1.2k 0.9× 302 0.3× 388 0.8× 390 0.9× 12 3.7k
Carlo Alberto Angeletti Italy 32 2.1k 1.2× 1.5k 1.2× 1.2k 1.3× 930 1.8× 219 0.5× 67 4.0k
Marja T. Nevalainen United States 37 1.8k 1.0× 1.7k 1.3× 1.0k 1.1× 876 1.7× 165 0.4× 77 3.7k
Gregory M. Springett United States 30 1.9k 1.0× 2.0k 1.5× 836 0.9× 694 1.4× 217 0.5× 86 4.4k
Elena A. Komarova United States 20 2.3k 1.3× 1.7k 1.3× 306 0.3× 686 1.3× 362 0.8× 24 3.8k
Desiree Ehleiter United States 15 2.2k 1.2× 609 0.5× 418 0.5× 415 0.8× 775 1.8× 18 3.6k
R.R. Weichselbaum United States 28 1.8k 1.0× 1.2k 0.9× 625 0.7× 659 1.3× 724 1.6× 64 3.5k
Julie L. Boerner United States 27 1.3k 0.7× 1.2k 0.9× 414 0.5× 438 0.9× 191 0.4× 63 2.4k
Ruoxiang Wang United States 35 1.6k 0.9× 1.1k 0.8× 904 1.0× 777 1.5× 146 0.3× 86 3.2k

Countries citing papers authored by Klaus Dittmann

Since Specialization
Citations

This map shows the geographic impact of Klaus Dittmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Klaus Dittmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Klaus Dittmann more than expected).

Fields of papers citing papers by Klaus Dittmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Klaus Dittmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Klaus Dittmann. The network helps show where Klaus Dittmann may publish in the future.

Co-authorship network of co-authors of Klaus Dittmann

This figure shows the co-authorship network connecting the top 25 collaborators of Klaus Dittmann. A scholar is included among the top collaborators of Klaus Dittmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Klaus Dittmann. Klaus Dittmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sonanini, Dominik, Christoph M. Griessinger, Philipp Knopf, et al.. (2021). Low-dose total body irradiation facilitates antitumoral Th1 immune responses. Theranostics. 11(16). 7700–7714. 8 indexed citations
2.
Freudenmann, Lena Katharina, Claus Mayer, H. Peter Rodemann, & Klaus Dittmann. (2019). Reduced exosomal L-Plastin is responsible for radiation-induced bystander effect. Experimental Cell Research. 383(1). 111498–111498. 7 indexed citations
3.
4.
Dittmann, Klaus, Claus Mayer, Stefan Czemmel, Stephan M. Huber, & H. Peter Rodemann. (2017). New roles for nuclear EGFR in regulating the stability and translation of mRNAs associated with VEGF signaling. PLoS ONE. 12(12). e0189087–e0189087. 21 indexed citations
5.
Rodemann, H. Peter, et al.. (2017). Glucose starvation impairs DNA repair in tumour cells selectively by blocking histone acetylation. Radiotherapy and Oncology. 126(3). 465–470. 22 indexed citations
6.
Dittmann, Klaus, Claus Mayer, H. Peter Rodemann, & Stephan M. Huber. (2013). EGFR cooperates with glucose transporter SGLT1 to enable chromatin remodeling in response to ionizing radiation. Radiotherapy and Oncology. 107(2). 247–251. 28 indexed citations
7.
Capalbo, Gianni, Klaus Dittmann, Christian Weiß, et al.. (2010). Radiation-Induced Survivin Nuclear Accumulation is Linked to DNA Damage Repair. International Journal of Radiation Oncology*Biology*Physics. 77(1). 226–234. 54 indexed citations
8.
Dittmann, Klaus, Claus Mayer, Rainer Kehlbach, & H. Peter Rodemann. (2008). Radiation-induced caveolin-1 associated EGFR internalization is linked with nuclear EGFR transport and activation of DNA-PK. Molecular Cancer. 7(1). 69–69. 157 indexed citations
9.
10.
Dittmann, Klaus, Claus Mayer, Rainer Kehlbach, & H. Peter Rodemann. (2008). The radioprotector Bowman–Birk proteinase inhibitor stimulates DNA repair via epidermal growth factor receptor phosphorylation and nuclear transport. Radiotherapy and Oncology. 86(3). 375–382. 37 indexed citations
11.
Ohneseit, Petra, et al.. (2007). Inhibition of cyclooxygenase-2 activity by celecoxib does not lead to radiosensitization of human prostate cancer cells in vitro. Radiotherapy and Oncology. 82(2). 229–238. 15 indexed citations
12.
Baumann, Michaël, Mechthild Krause, Ekkehard Dikomey, et al.. (2007). EGFR-targeted anti-cancer drugs in radiotherapy: Preclinical evaluation of mechanisms. Radiotherapy and Oncology. 83(3). 238–248. 147 indexed citations
13.
Mayer, Claus, et al.. (2007). Activation of protein kinase Cε stimulates DNA-repair via epidermal growth factor receptor nuclear accumulation. Radiotherapy and Oncology. 86(3). 383–390. 46 indexed citations
14.
Dittmann, Klaus, Claus Mayer, Birgit Fehrenbacher, et al.. (2005). Radiation-induced Epidermal Growth Factor Receptor Nuclear Import Is Linked to Activation of DNA-dependent Protein Kinase. Journal of Biological Chemistry. 280(35). 31182–31189. 433 indexed citations
15.
Dittmann, Klaus, Claus Mayer, & H. Peter Rodemann. (2005). Inhibition of radiation-induced EGFR nuclear import by C225 (Cetuximab) suppresses DNA-PK activity. Radiotherapy and Oncology. 76(2). 157–161. 234 indexed citations
16.
Baumann, Michaël, Mechthild Krause, Daniel Zips, et al.. (2004). Molecular targeting in radiotherapy of lung cancer. Lung Cancer. 45. S187–S197. 20 indexed citations
17.
Dittmann, Klaus, Claus Mayer, & H. Peter Rodemann. (2001). O-phospho-L-tyrosine protects TP53 wild-type cells against ionizing radiation. International Journal of Cancer. 96(S1). 1–1. 8 indexed citations
18.
Dittmann, Klaus, et al.. (2001). Bowman-Birk proteinase inhibitor-mediated radioprotection against UV irradiation is TP53-dependent and associated with stimulation of nucleotide excision repair. Radiation and Environmental Biophysics. 40(2). 163–167. 5 indexed citations
19.
Gueven, Nuri, Klaus Dittmann, Claus Mayer, & H. Peter Rodemann. (1998). The radioprotective potential of the Bowman–Birk protease inhibitor is independent of its secondary structure. Cancer Letters. 125(1-2). 77–82. 16 indexed citations
20.
Dittmann, Klaus, Heidi Löffler, Michael Bamberg, & H. Peter Rodemann. (1995). Bowman-Birk proteinase inhibitor (BBI) modulates radiosensitivity and radiation-induced differentiation of human fibroblasts in culture. Radiotherapy and Oncology. 34(2). 137–143. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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