Khadija Khan

5.1k total citations
12 papers, 206 citations indexed

About

Khadija Khan is a scholar working on Infectious Diseases, Molecular Biology and Virology. According to data from OpenAlex, Khadija Khan has authored 12 papers receiving a total of 206 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Infectious Diseases, 2 papers in Molecular Biology and 2 papers in Virology. Recurrent topics in Khadija Khan's work include SARS-CoV-2 and COVID-19 Research (4 papers), COVID-19 Clinical Research Studies (3 papers) and HIV Research and Treatment (2 papers). Khadija Khan is often cited by papers focused on SARS-CoV-2 and COVID-19 Research (4 papers), COVID-19 Clinical Research Studies (3 papers) and HIV Research and Treatment (2 papers). Khadija Khan collaborates with scholars based in South Africa, United States and United Kingdom. Khadija Khan's co-authors include Wajihullah Khan, Farina Karim, Alex Sigal, Sandile Cele, Helen Y. Chu, Rachel Eguia, Jennifer K. Logue, Allison J. Greaney, Jesse D. Bloom and Tyler N. Starr and has published in prestigious journals such as Frontiers in Immunology, PLoS Pathogens and Transplantation.

In The Last Decade

Khadija Khan

12 papers receiving 198 citations

Peers

Khadija Khan

PARAS

EPIDE

VIROL

Terence C. Theisen United States

Wesley Freppel Canada

Faustina Pappoe Ghana

Luz Belinda Ortiz-Alegría Mexico

Yudai Kaneko Japan

Irma Cañedo-Solares Mexico

Héctor Luna-Pastén Mexico

Claudia Patricia Rico-Torres Mexico

Cynthia Azeredo Cordeiro Brazil

Jacob K. Frenkel United States

Terence C. Theisen United States

Khadija Khan

152 ×1.5

PARAS

49 ×0.6

178 ×2.6

EPIDE

16 ×0.3

VIROL

71 ×1.4

Citations per year, relative to Khadija Khan Khadija Khan (= 1×) peers Terence C. Theisen

Countries citing papers authored by Khadija Khan

Since Specialization

Citations

This map shows the geographic impact of Khadija Khan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Khadija Khan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Khadija Khan more than expected).

Fields of papers citing papers by Khadija Khan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Khadija Khan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Khadija Khan. The network helps show where Khadija Khan may publish in the future.

Co-authorship network of co-authors of Khadija Khan

This figure shows the co-authorship network connecting the top 25 collaborators of Khadija Khan. A scholar is included among the top collaborators of Khadija Khan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Khadija Khan. Khadija Khan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

Krause, Robert, Paul Ogongo, Liku B. Tezera, et al.. (2024). B cell heterogeneity in human tuberculosis highlights compartment-specific phenotype and functional roles. Communications Biology. 7(1). 584–584. 9 indexed citations

Mandolo, Jonathan, Farina Karim, Khadija Khan, et al.. (2024). Low pre-existing endemic human coronavirus (HCoV-NL63)-specific T cell frequencies are associated with impaired SARS-CoV-2-specific T cell responses in people living with HIV. Frontiers in Immunology. 14. 1291048–1291048. 2 indexed citations

Lustig, Gila, Yashica Ganga, Hylton Rodel, et al.. (2023). SARS-CoV-2 infection in immunosuppression evolves sub-lineages which independently accumulate neutralization escape mutations. Virus Evolution. 10(1). vead075–vead075. 5 indexed citations

Greaney, Allison J., Tyler N. Starr, Rachel Eguia, et al.. (2022). A SARS-CoV-2 variant elicits an antibody response with a shifted immunodominance hierarchy. PLoS Pathogens. 18(2). e1010248–e1010248. 28 indexed citations

Greaney, Allison J., Rachel Eguia, Tyler N. Starr, et al.. (2022). The SARS-CoV-2 Delta variant induces an antibody response largely focused on class 1 and 2 antibody epitopes. PLoS Pathogens. 18(6). e1010592–e1010592. 10 indexed citations

Khan, Khadija, et al.. (2022). Immunohistopathological changes in the placenta of malaria-infected women in unstable transmission setting of Aligarh. Placenta. 127. 52–61. 2 indexed citations

Lustig, Gila, Sandile Cele, Farina Karim, et al.. (2021). T cell derived HIV-1 is present in the CSF in the face of suppressive antiretroviral therapy. PLoS Pathogens. 17(9). e1009871–e1009871. 28 indexed citations

Khan, Khadija, et al.. (2020). Seroprevalence of low avidity anti-Toxoplasma IgG in pregnant women and its relationship with their age and contact with cats. Tropical biomedicine. 37(4). 1038–1049. 2 indexed citations

Khan, Khadija & Wajihullah Khan. (2018). Congenital toxoplasmosis: An overview of the neurological and ocular manifestations. Parasitology International. 67(6). 715–721. 111 indexed citations

10.

Jackson, Laurelle, Sandile Cele, Isabella A.T.M. Ferreira, et al.. (2018). Incomplete inhibition of HIV infection results in more HIV infected lymph node cells by reducing cell death. eLife. 7. 6 indexed citations

11.

Lau, Arthur, Khadija Khan, Xiaoyi Huang, et al.. (2014). Endogenous Expression of SPI-6 (Serpin Protease Inhibitor-6) in Renal Tubular Cells Inhibits Granzyme B Mediated Injury and Promotes Renal Allograft Survival.. Transplantation. 98. 328–328. 1 indexed citations

12.

Khan, Khadija, et al.. (2007). Helminth Parasitic Load in Soil of Northern Lahore. 1(3). 248–251. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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