Ke-Yu Deng

667 total citations
8 papers, 527 citations indexed

About

Ke-Yu Deng is a scholar working on Molecular Biology, Physiology and Genetics. According to data from OpenAlex, Ke-Yu Deng has authored 8 papers receiving a total of 527 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Molecular Biology, 3 papers in Physiology and 3 papers in Genetics. Recurrent topics in Ke-Yu Deng's work include Calcium signaling and nucleotide metabolism (2 papers), Adenosine and Purinergic Signaling (1 paper) and Autophagy in Disease and Therapy (1 paper). Ke-Yu Deng is often cited by papers focused on Calcium signaling and nucleotide metabolism (2 papers), Adenosine and Purinergic Signaling (1 paper) and Autophagy in Disease and Therapy (1 paper). Ke-Yu Deng collaborates with scholars based in China, United States and Canada. Ke-Yu Deng's co-authors include Tsutomu Kume, Douglas B. Gould, Brigid L.M. Hogan, Virginia P. Winfrey, Michael A. Walter, Hong‐Bo Xin, Guangju Ji, Michael I. Kotlikoff, Mark Rishniw and Lei Gu and has published in prestigious journals such as Cell, Scientific Reports and Signal Transduction and Targeted Therapy.

In The Last Decade

Ke-Yu Deng

8 papers receiving 520 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ke-Yu Deng China	6	354	120	48	46	44	8	527
Annabel Christ Germany	14	321 0.9×	101 0.8×	40 0.8×	71 1.5×	39 0.9×	18	556
Nisha Patel Saudi Arabia	17	428 1.2×	267 2.2×	60 1.3×	52 1.1×	28 0.6×	39	727
Pelin Özlem Şimşek‐Kiper Türkiye	14	377 1.1×	326 2.7×	29 0.6×	48 1.0×	87 2.0×	99	662
Esther Kinning United Kingdom	13	321 0.9×	256 2.1×	94 2.0×	35 0.8×	28 0.6×	28	556
Kit San Yeung Hong Kong	13	252 0.7×	245 2.0×	30 0.6×	40 0.9×	32 0.7×	29	529
Fatema Alzahrani Saudi Arabia	14	430 1.2×	386 3.2×	47 1.0×	33 0.7×	43 1.0×	25	724
Fabian Tetzlaff Germany	11	248 0.7×	36 0.3×	74 1.5×	86 1.9×	42 1.0×	13	434
Go Hun Seo South Korea	11	256 0.7×	204 1.7×	21 0.4×	31 0.7×	20 0.5×	82	527
Soo‐Mi Park United Kingdom	15	464 1.3×	258 2.1×	79 1.6×	52 1.1×	39 0.9×	39	794
Lanying Song United States	16	557 1.6×	205 1.7×	67 1.4×	80 1.7×	23 0.5×	22	749

Countries citing papers authored by Ke-Yu Deng

Since Specialization

Citations

This map shows the geographic impact of Ke-Yu Deng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ke-Yu Deng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ke-Yu Deng more than expected).

Fields of papers citing papers by Ke-Yu Deng

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ke-Yu Deng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ke-Yu Deng. The network helps show where Ke-Yu Deng may publish in the future.

Co-authorship network of co-authors of Ke-Yu Deng

This figure shows the co-authorship network connecting the top 25 collaborators of Ke-Yu Deng. A scholar is included among the top collaborators of Ke-Yu Deng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ke-Yu Deng. Ke-Yu Deng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Zhang, Yating, Ke Wen, Qi Ding, et al.. (2025). Myeloid but not hepatocytic CD38 is a key driver for hepatic ischemia/reperfusion injury. Signal Transduction and Targeted Therapy. 10(1). 150–150. 1 indexed citations

2.

Huang, Qiming, et al.. (2024). Long-term hypoxic atmosphere enhances the stemness, immunoregulatory functions, and therapeutic application of human umbilical cord mesenchymal stem cells. Bone and Joint Research. 13(12). 763–777. 1 indexed citations

3.

He, Jing, et al.. (2024). Myeloid Deletion of Cdc42 Protects Liver From Hepatic Ischemia-Reperfusion Injury via Inhibiting Macrophage-Mediated Inflammation in Mice. Cellular and Molecular Gastroenterology and Hepatology. 17(6). 965–981. 6 indexed citations

4.

He, Xiangqin, Ning Wang, Dan Wang, et al.. (2020). Specific deletion of CDC42 in pancreatic β cells attenuates glucose-induced insulin expression and secretion in mice. Molecular and Cellular Endocrinology. 518. 111004–111004. 10 indexed citations

5.

Qian, Yisong, Ziwei Wang, Li Zuo, et al.. (2018). CD38 Deficiency Promotes Inflammatory Response through Activating Sirt1/NF-κB-Mediated Inhibition of TLR2 Expression in Macrophages. Mediators of Inflammation. 2018. 1–13. 23 indexed citations

6.

Yuan, Qi, Zheng Chen, Gaetano Santulli, et al.. (2014). Functional Role of Calstabin2 in Age-related Cardiac Alterations. Scientific Reports. 4(1). 7425–7425. 58 indexed citations

7.

Xin, Hong‐Bo, Ke-Yu Deng, Mark Rishniw, Guangju Ji, & Michael I. Kotlikoff. (2002). Smooth muscle expression of Cre recombinase and eGFP in transgenic mice. Physiological Genomics. 10(3). 211–215. 121 indexed citations

8.

Kume, Tsutomu, Ke-Yu Deng, Virginia P. Winfrey, et al.. (1998). The Forkhead/Winged Helix Gene Mf1 Is Disrupted in the Pleiotropic Mouse Mutation congenital hydrocephalus. Cell. 93(6). 985–996. 307 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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