Kestis Vaitkus

459 citations
22 papers · 343 indexed · h-index 12
Topics
Hemoglobinopathies and Related Disorders (22 papers)Epigenetics and DNA Methylation (21 papers)Cancer-related gene regulation (8 papers)
Partner nations
United States

In The Last Decade

Kestis Vaitkus

21 papers receiving 338 citations

Peers

Kestis Vaitkus
Comparison fields: 5 of 32
  • Molecular Biology 269
  • Genetics 208
  • Hematology 113
  • Pediatrics, Perinatology and Child Health 60
  • Physiology 28
Replace Rosalba Di Marzo with:
Rosalba Di Marzo Italy
Maria Suciu United Kingdom
Manoussos N. Papadakis Greece
Anne Boland France
AE Kulozik United Kingdom
Alireza Ghamari United States
Wattanan Makarasara Thailand
Kalin Mayberry United States
Amy Y. Y. Chan Hong Kong
G. V. Sciarratta Italy
Kestis Vaitkus relative to Rosalba Di Marzo Italy Rosalba Di Marzo's profile →
Citations per field
00.5×4.7×
Rosalba Di Marzo · 1×
Citations per year

Countries citing papers authored by Kestis Vaitkus

Since Specialization
Citations

This map shows the geographic impact of Kestis Vaitkus's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kestis Vaitkus with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kestis Vaitkus more than expected).

Fields of papers citing papers by Kestis Vaitkus

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kestis Vaitkus. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kestis Vaitkus. The network helps show where Kestis Vaitkus may publish in the future.

Co-authorship network of co-authors of Kestis Vaitkus

This figure shows the co-authorship network connecting the top 25 collaborators of Kestis Vaitkus. A scholar is included among the top collaborators of Kestis Vaitkus based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kestis Vaitkus. Kestis Vaitkus is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 5
3 18
4 1
5 23
6 37
7 3
8 1
9 72
10 21
11 23
12 22
13 22
14 1
15 7
16 28
17 26
18 19
19 11
20 1

About Kestis Vaitkus

Kestis Vaitkus is a scholar working on Genetics, Pediatrics, Perinatology and Child Health and Molecular Biology, having authored 22 papers that have together received 343 indexed citations. Recurring topics across this work include Hemoglobinopathies and Related Disorders (22 papers), Epigenetics and DNA Methylation (21 papers) and Cancer-related gene regulation (8 papers). The work is most often cited by research in Genetics (208 citations), Hematology (113 citations) and Molecular Biology (269 citations). Kestis Vaitkus has collaborated with scholars based in United States. Frequent co-authors include Donald Lavelle, Joseph DeSimone, Vinzon Ibañez, Mahipal Singh, Maria Armila Ruiz, Yogen Saunthararajah, Angela Rivers, Nadim Mahmud, Tatiana Kouznetsova and Ramasamy Jagadeeswaran. Their work appears in journals such as Blood, PLoS ONE and Journal of Translational Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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