Kerstin Klinkert

823 total citations
9 papers, 514 citations indexed

About

Kerstin Klinkert is a scholar working on Molecular Biology, Cell Biology and Surgery. According to data from OpenAlex, Kerstin Klinkert has authored 9 papers receiving a total of 514 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 6 papers in Cell Biology and 2 papers in Surgery. Recurrent topics in Kerstin Klinkert's work include Cellular transport and secretion (5 papers), Microtubule and mitosis dynamics (2 papers) and Photosynthetic Processes and Mechanisms (2 papers). Kerstin Klinkert is often cited by papers focused on Cellular transport and secretion (5 papers), Microtubule and mitosis dynamics (2 papers) and Photosynthetic Processes and Mechanisms (2 papers). Kerstin Klinkert collaborates with scholars based in France, Ireland and Germany. Kerstin Klinkert's co-authors include Arnaud Échard, Anne Houdusse, Murielle Rocancourt, Anne‐Laure Leblond, Noel M. Caplice, Arun Kumar, Kenneth Martin, Elizebeth C. Turner, Tara C. Browne and Stéphane Frémont and has published in prestigious journals such as Nature Communications, PLoS ONE and Cell Reports.

In The Last Decade

Kerstin Klinkert

9 papers receiving 512 citations

Peers

Kerstin Klinkert

MB

CB

IMMUN

PHYSI

CCM

Katherine R. Doherty United States

Gerolamo Lanfranchi Italy

Nicolas Figeac United Kingdom

Liam C. Hunt United States

Angela Lek United States

Frances J. Evesson Australia

Kayleigh A. Swaggart United States

Jennifer A. Schumacher United States

Csilla Pataki Denmark

Bhairab N. Singh United States

Katherine R. Doherty United States

Kerstin Klinkert

446 ×1.7

MB

150 ×0.6

CB

25 ×0.3

IMMUN

80 ×1.5

PHYSI

82 ×1.5

CCM

Citations per year, relative to Kerstin Klinkert Kerstin Klinkert (= 1×) peers Katherine R. Doherty

Countries citing papers authored by Kerstin Klinkert

Since Specialization

Citations

This map shows the geographic impact of Kerstin Klinkert's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kerstin Klinkert with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kerstin Klinkert more than expected).

Fields of papers citing papers by Kerstin Klinkert

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kerstin Klinkert. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kerstin Klinkert. The network helps show where Kerstin Klinkert may publish in the future.

Co-authorship network of co-authors of Kerstin Klinkert

This figure shows the co-authorship network connecting the top 25 collaborators of Kerstin Klinkert. A scholar is included among the top collaborators of Kerstin Klinkert based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kerstin Klinkert. Kerstin Klinkert is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Mellouk, Nora, Magdalena Gil, Camila Valenzuela, et al.. (2024). Post-translational targeting of Rab35 by the effector IcsB of Shigella determines intracellular bacterial niche formation. Cell Reports. 43(4). 114034–114034. 1 indexed citations

2.

Patiño‐López, Genaro, Kerstin Klinkert, Francina Langa‐Vives, et al.. (2020). Rab35-regulated lipid turnover by myotubularins represses mTORC1 activity and controls myelin growth. Nature Communications. 11(1). 2835–2835. 20 indexed citations

3.

Klinkert, Kerstin, Nicolas Levernier, Peter Groß, et al.. (2019). Aurora A depletion reveals centrosome-independent polarization mechanism in Caenorhabditis elegans. eLife. 8. 42 indexed citations

4.

Frémont, Stéphane, Jian Bai, Hugo Wioland, et al.. (2017). Oxidation of F-actin controls the terminal steps of cytokinesis. Nature Communications. 8(1). 14528–14528. 119 indexed citations

5.

Klinkert, Kerstin, et al.. (2017). Selective M2 Macrophage Depletion Leads to Prolonged Inflammation in Surgical Wounds. European Surgical Research. 58(3-4). 109–120. 56 indexed citations

6.

Klinkert, Kerstin & Arnaud Échard. (2016). Rab35 GTPase: A Central Regulator of Phosphoinositides and F‐actin in Endocytic Recycling and Beyond. Traffic. 17(10). 1063–1077. 106 indexed citations

7.

Klinkert, Kerstin, Murielle Rocancourt, Anne Houdusse, & Arnaud Échard. (2016). Rab35 GTPase couples cell division with initiation of epithelial apico-basal polarity and lumen opening. Nature Communications. 7(1). 11166–11166. 80 indexed citations

8.

Leblond, Anne‐Laure, Kerstin Klinkert, Kenneth Martin, et al.. (2015). Systemic and Cardiac Depletion of M2 Macrophage through CSF-1R Signaling Inhibition Alters Cardiac Function Post Myocardial Infarction. PLoS ONE. 10(9). e0137515–e0137515. 88 indexed citations

9.

Martin, Kenneth, Arun Kumar, Kerstin Klinkert, & Noel M. Caplice. (2013). Differential Endothelial Coverage, Response to Injury and Neointimal Integration of CX3CR1/Smooth Muscle-Like Cells after Carotid or Femoral Arterial Injury. Journal of Vascular Research. 50(3). 200–209. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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