Kenneth E. Viar

3.2k total citations
14 papers, 683 citations indexed

About

Kenneth E. Viar is a scholar working on Endocrine and Autonomic Systems, Cognitive Neuroscience and Cellular and Molecular Neuroscience. According to data from OpenAlex, Kenneth E. Viar has authored 14 papers receiving a total of 683 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Endocrine and Autonomic Systems, 9 papers in Cognitive Neuroscience and 3 papers in Cellular and Molecular Neuroscience. Recurrent topics in Kenneth E. Viar's work include Neuroscience of respiration and sleep (12 papers), Sleep and Wakefulness Research (9 papers) and Circadian rhythm and melatonin (5 papers). Kenneth E. Viar is often cited by papers focused on Neuroscience of respiration and sleep (12 papers), Sleep and Wakefulness Research (9 papers) and Circadian rhythm and melatonin (5 papers). Kenneth E. Viar collaborates with scholars based in United States, Lebanon and Czechia. Kenneth E. Viar's co-authors include Ruth L. Stornetta, Patrice G. Guyenet, Peter G.R. Burke, Patrice G. Guyenet, Chikara Abe, Benjamin B. Holloway, Roy Kanbar, Tyler Basting, Melissa B. Coates and Daniel S. Stornetta and has published in prestigious journals such as Journal of Neuroscience, Genes & Development and Nature Neuroscience.

In The Last Decade

Kenneth E. Viar

14 papers receiving 682 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kenneth E. Viar United States 14 430 293 153 148 134 14 683
Ian C. Wenker United States 17 546 1.3× 292 1.0× 138 0.9× 167 1.1× 43 0.3× 30 822
Genrieta Bochorishvili United States 9 372 0.9× 332 1.1× 138 0.9× 120 0.8× 62 0.5× 10 747
Melissa B. Coates United States 10 620 1.4× 421 1.4× 163 1.1× 215 1.5× 33 0.2× 14 768
Melissa M.J. Farnham Australia 17 299 0.7× 87 0.3× 137 0.9× 95 0.6× 106 0.8× 39 640
Olga Dergacheva United States 19 522 1.2× 267 0.9× 210 1.4× 97 0.7× 31 0.2× 45 741
Clément Menuet France 18 410 1.0× 199 0.7× 176 1.2× 118 0.8× 45 0.3× 32 904
Marc Stefan Dawid-Milner Spain 13 314 0.7× 166 0.6× 179 1.2× 100 0.7× 31 0.2× 35 556
Seth D. DePuy United States 13 869 2.0× 449 1.5× 307 2.0× 293 2.0× 65 0.5× 13 1.2k
T Thomas United Kingdom 19 593 1.4× 119 0.4× 175 1.1× 120 0.8× 56 0.4× 24 885
Anthony D. Shafton Australia 15 421 1.0× 156 0.5× 179 1.2× 157 1.1× 72 0.5× 28 846

Countries citing papers authored by Kenneth E. Viar

Since Specialization
Citations

This map shows the geographic impact of Kenneth E. Viar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kenneth E. Viar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kenneth E. Viar more than expected).

Fields of papers citing papers by Kenneth E. Viar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kenneth E. Viar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kenneth E. Viar. The network helps show where Kenneth E. Viar may publish in the future.

Co-authorship network of co-authors of Kenneth E. Viar

This figure shows the co-authorship network connecting the top 25 collaborators of Kenneth E. Viar. A scholar is included among the top collaborators of Kenneth E. Viar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kenneth E. Viar. Kenneth E. Viar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Viar, Kenneth E., et al.. (2020). Sarm1 knockout protects against early but not late axonal degeneration in experimental allergic encephalomyelitis. PLoS ONE. 15(6). e0235110–e0235110. 15 indexed citations
2.
Wenker, Ian C., Chikara Abe, Kenneth E. Viar, et al.. (2017). Blood Pressure Regulation by the Rostral Ventrolateral Medulla in Conscious Rats: Effects of Hypoxia, Hypercapnia, Baroreceptor Denervation, and Anesthesia. Journal of Neuroscience. 37(17). 4565–4583. 54 indexed citations
3.
Abe, Chikara, Tsuyoshi Inoue, Kenneth E. Viar, et al.. (2017). C1 neurons mediate a stress-induced anti-inflammatory reflex in mice. Nature Neuroscience. 20(5). 700–707. 146 indexed citations
4.
Basting, Tyler, Chikara Abe, Kenneth E. Viar, Ruth L. Stornetta, & Patrice G. Guyenet. (2016). Is plasticity within the retrotrapezoid nucleus responsible for the recovery of the set‐point after carotid body denervation in rats?. The Journal of Physiology. 594(12). 3371–3390. 17 indexed citations
5.
Burke, Peter G.R., Roy Kanbar, Tyler Basting, et al.. (2015). State‐dependent control of breathing by the retrotrapezoid nucleus. The Journal of Physiology. 593(13). 2909–2926. 67 indexed citations
6.
Burke, Peter G.R., Roy Kanbar, Kenneth E. Viar, Ruth L. Stornetta, & Patrice G. Guyenet. (2015). Selective optogenetic stimulation of the retrotrapezoid nucleus in sleeping rats activates breathing without changing blood pressure or causing arousal or sighs. Journal of Applied Physiology. 118(12). 1491–1501. 26 indexed citations
7.
Stornetta, Ruth L., et al.. (2015). Afferent and efferent connections of C1 cells with spinal cord or hypothalamic projections in mice. Brain Structure and Function. 221(8). 4027–4044. 33 indexed citations
8.
Basting, Tyler, Peter G.R. Burke, Roy Kanbar, et al.. (2015). Hypoxia Silences Retrotrapezoid Nucleus Respiratory Chemoreceptors via Alkalosis. Journal of Neuroscience. 35(2). 527–543. 56 indexed citations
9.
Holloway, Benjamin B., Kenneth E. Viar, Ruth L. Stornetta, & Patrice G. Guyenet. (2015). The retrotrapezoid nucleus stimulates breathing by releasing glutamate in adult conscious mice. European Journal of Neuroscience. 42(6). 2271–2282. 28 indexed citations
10.
Burke, Peter G.R., et al.. (2014). Optogenetic Stimulation of Adrenergic C1 Neurons Causes Sleep State–Dependent Cardiorespiratory Stimulation and Arousal with Sighs in Rats. American Journal of Respiratory and Critical Care Medicine. 190(11). 1301–1310. 68 indexed citations
11.
Lim, Chae‐Seok, et al.. (2014). Pharmacological rescue of Ras signaling, GluA1-dependent synaptic plasticity, and learning deficits in a fragile X model. Genes & Development. 28(3). 273–289. 48 indexed citations
12.
Bochorishvili, Genrieta, Thành Nguyễn, Melissa B. Coates, et al.. (2014). The orexinergic neurons receive synaptic input from C1 cells in rats. The Journal of Comparative Neurology. 522(17). 3834–3846. 41 indexed citations
13.
Holloway, Benjamin B., Ruth L. Stornetta, Genrieta Bochorishvili, et al.. (2013). Monosynaptic Glutamatergic Activation of Locus Coeruleus and Other Lower Brainstem Noradrenergic Neurons by the C1 Cells in Mice. Journal of Neuroscience. 33(48). 18792–18805. 51 indexed citations
14.
Abbott, Stephen B.G., Benjamin B. Holloway, Kenneth E. Viar, & Patrice G. Guyenet. (2013). Vesicular glutamate transporter 2 is required for the respiratory and parasympathetic activation produced by optogenetic stimulation of catecholaminergic neurons in the rostral ventrolateral medulla of mice in vivo. European Journal of Neuroscience. 39(1). 98–106. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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