Kenjiro Seki

1.3k total citations
32 papers, 997 citations indexed

About

Kenjiro Seki is a scholar working on Cellular and Molecular Neuroscience, Behavioral Neuroscience and Cognitive Neuroscience. According to data from OpenAlex, Kenjiro Seki has authored 32 papers receiving a total of 997 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Cellular and Molecular Neuroscience, 8 papers in Behavioral Neuroscience and 8 papers in Cognitive Neuroscience. Recurrent topics in Kenjiro Seki's work include Stress Responses and Cortisol (8 papers), Neuroscience and Neuropharmacology Research (6 papers) and Tryptophan and brain disorders (6 papers). Kenjiro Seki is often cited by papers focused on Stress Responses and Cortisol (8 papers), Neuroscience and Neuropharmacology Research (6 papers) and Tryptophan and brain disorders (6 papers). Kenjiro Seki collaborates with scholars based in Japan, United States and Slovakia. Kenjiro Seki's co-authors include Richard Lee, Shoji Sanada, Matthew L. Steinhauser, Anastacia Y. Kudinova, Joseph Gannon, Satomi Yoshida, Manoj K. Jaiswal, Masaharu Kudoh, Katsuei Shibuki and Motomichi Torisu and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and The Journal of Immunology.

In The Last Decade

Kenjiro Seki

28 papers receiving 976 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kenjiro Seki Japan 15 310 218 174 173 135 32 997
James P. Halper United States 19 317 1.0× 156 0.7× 48 0.3× 240 1.4× 58 0.4× 26 1.4k
Thierry Amédée France 22 152 0.5× 488 2.2× 88 0.5× 782 4.5× 193 1.4× 30 1.6k
Anja Finn Sweden 18 64 0.2× 241 1.1× 153 0.9× 183 1.1× 26 0.2× 30 1.2k
Fumihiko Okada Japan 19 107 0.3× 256 1.2× 98 0.6× 642 3.7× 115 0.9× 48 1.4k
G.P. Hemmings United Kingdom 17 137 0.4× 199 0.9× 62 0.4× 404 2.3× 28 0.2× 44 1.2k
Gabriel Rusanescu United States 12 73 0.2× 225 1.0× 68 0.4× 454 2.6× 121 0.9× 14 1.1k
Anita Patel United States 16 439 1.4× 156 0.7× 61 0.4× 401 2.3× 24 0.2× 21 1.6k
X. Lin United States 13 141 0.5× 83 0.4× 57 0.3× 206 1.2× 54 0.4× 22 828
Martina Svensson Sweden 14 273 0.9× 92 0.4× 65 0.4× 393 2.3× 39 0.3× 19 1.1k
Teresa Wierzba‐Bobrowicz Poland 21 125 0.4× 228 1.0× 51 0.3× 430 2.5× 16 0.1× 109 1.4k

Countries citing papers authored by Kenjiro Seki

Since Specialization
Citations

This map shows the geographic impact of Kenjiro Seki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kenjiro Seki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kenjiro Seki more than expected).

Fields of papers citing papers by Kenjiro Seki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kenjiro Seki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kenjiro Seki. The network helps show where Kenjiro Seki may publish in the future.

Co-authorship network of co-authors of Kenjiro Seki

This figure shows the co-authorship network connecting the top 25 collaborators of Kenjiro Seki. A scholar is included among the top collaborators of Kenjiro Seki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kenjiro Seki. Kenjiro Seki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Takahashi, H., et al.. (2024). GABAergic circuit interaction between central amygdala and bed nucleus of the stria terminalis in lipopolysaccharide-induced despair-like behavior. Physiology & Behavior. 288. 114753–114753. 1 indexed citations
2.
Yoshida, Satomi, et al.. (2020). The α1-adrenergic receptors in the amygdala regulate the induction of learned despair through protein kinase C-beta signaling. Behavioural Pharmacology. 32(1). 73–85. 5 indexed citations
3.
Yoshida, Satomi, et al.. (2020). Gαq protein signaling in the bed nucleus of the stria terminalis regulate the lipopolysaccharide-induced despair-like behavior in mice. SHILAP Revista de lepidopterología. 7(4). 438–458. 3 indexed citations
4.
Jaiswal, Manoj K., Kenjiro Seki, & Satomi Yoshida. (2018). Molecular mechanism of noradrenaline during the stress-induced major depressive disorder. Neural Regeneration Research. 13(7). 1159–1159. 76 indexed citations
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Ohtake, Nobuaki, Mieko Saito, Masaaki Eto, & Kenjiro Seki. (2014). Exendin-4 promotes the membrane trafficking of the AMPA receptor GluR1 subunit and ADAM10 in the mouse neocortex. Regulatory Peptides. 190-191. 1–11. 35 indexed citations
9.
Kaieda, Shinjiro, Kichul Shin, Peter A. Nigrović, et al.. (2010). Synovial Fibroblasts Promote the Expression and Granule Accumulation of Tryptase via Interleukin-33 and Its Receptor ST-2 (IL1RL1). Journal of Biological Chemistry. 285(28). 21478–21486. 53 indexed citations
10.
Araya, Runa, Takanori Noguchi, Naohito Kitamura, et al.. (2006). Loss of M5 muscarinic acetylcholine receptors leads to cerebrovascular and neuronal abnormalities and cognitive deficits in mice. Neurobiology of Disease. 24(2). 334–344. 69 indexed citations
11.
Kamiya, Atsunori, Toru Kawada, Masaki Mizuno, et al.. (2006). Baroreflex Increases Correlation and Coherence of Muscle Sympathetic Nerve Activity (SNA) with Renal and Cardiac SNAs. The Journal of Physiological Sciences. 56(5). 325–333. 4 indexed citations
12.
Kudoh, Masaharu, Kenjiro Seki, & Katsuei Shibuki. (2004). Sound sequence discrimination learning is dependent on cholinergic inputs to the rat auditory cortex. Neuroscience Research. 50(1). 113–123. 22 indexed citations
13.
Seki, Kenjiro, Masaharu Kudoh, & Katsuei Shibuki. (2001). Sequence dependence of post‐tetanic potentiation after sequential heterosynaptic stimulation in the rat auditory cortex. The Journal of Physiology. 533(2). 503–518. 24 indexed citations
14.
Seki, Kenjiro, Masaharu Kudoh, & Katsuei Shibuki. (1999). Long-term potentiation of Ca2+ signal in the rat auditory cortex. Neuroscience Research. 34(3). 187–197. 17 indexed citations
15.
Furuya, Kenichi, Yoshifumi Mizumoto, Manabu Murakami, et al.. (1995). Gene Expressions of Oxytocin and Oxytocin Receptor in Cumulus Cells of Human Ovary. Hormone Research. 44(2). 47–49. 18 indexed citations
16.
Seki, Kenjiro, et al.. (1991). A case of microcystic adnexal carcinoma.. Skin Cancer. 6(1). 95–97. 1 indexed citations
17.
Morita, Makoto, et al.. (1991). Three Different Adnexal Tumors Arising in Organoid Nevus.. The Nishinihon Journal of Dermatology. 53(5). 960–964.
18.
Suzuki, Hidemi, et al.. (1990). An unusual case of sporotrichosis and a statistical survey 1974-1987 at our university hospital.. The Nishinihon Journal of Dermatology. 52(1). 36–41.
19.
Seki, Kenjiro, et al.. (1989). Primary cutaneous aspergillosis associated with leukemia: Report of 3 cases.. The Nishinihon Journal of Dermatology. 51(4). 695–700. 1 indexed citations
20.
Ishida, M., et al.. (1982). [Changes in the criteria for planned delivery in the National Defense Medical College Hospital with statistical analysis].. PubMed. 34(12). 2123–32. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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