Kenji Iwakabe

1.1k total citations · 1 hit paper
16 papers, 977 citations indexed

About

Kenji Iwakabe is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Kenji Iwakabe has authored 16 papers receiving a total of 977 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Immunology, 4 papers in Oncology and 3 papers in Molecular Biology. Recurrent topics in Kenji Iwakabe's work include Immune Cell Function and Interaction (12 papers), Immunotherapy and Immune Responses (10 papers) and T-cell and B-cell Immunology (8 papers). Kenji Iwakabe is often cited by papers focused on Immune Cell Function and Interaction (12 papers), Immunotherapy and Immune Responses (10 papers) and T-cell and B-cell Immunology (8 papers). Kenji Iwakabe collaborates with scholars based in Japan, United States and Ireland. Kenji Iwakabe's co-authors include Takashi Nishimura, Takashi Yahata, Marimo Sato, Akio Ohta, Yasushi Ohmi, Tetsu Kawano, Hidemitsu Kitamura, Masaru Taniguchi, Shin‐Ichiro Nishimura and Luc Van Kaer and has published in prestigious journals such as The Journal of Experimental Medicine, European Journal of Immunology and Journal of Neuroimmunology.

In The Last Decade

Kenji Iwakabe

16 papers receiving 960 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The Natural Killer T (NKT) Cell Ligand α-Galactosylceramide Demonstrates Its Immunopotentiating Effect by Inducing Interleukin (IL)-12 Production by Dendritic Cells and IL-12 Receptor Expression on NKT Cells

1999 527 citations Hidemitsu Kitamura, Kenji Iwakabe et al. The Journal of Experimental Medicine profile →

Peers

Kenji Iwakabe

IMMUN

ONCOL

EPIDE

PHYSI

Yasushi Ohmi Japan

S Habu Japan

Egle Šimelyte Finland

Lada Paul Israel

Mamata De United States

T Oku Japan

Z Amraoui Belgium

Wilfred Lim Canada

John J. Seeley United States

Siobhan Blankson Ireland

Yasushi Ohmi Japan

Kenji Iwakabe

669 ×0.8

IMMUN

165 ×0.7

ONCOL

93 ×1.0

58 ×0.9

EPIDE

53 ×1.0

PHYSI

Citations per year, relative to Kenji Iwakabe Kenji Iwakabe (= 1×) peers Yasushi Ohmi

Countries citing papers authored by Kenji Iwakabe

Since Specialization

Citations

This map shows the geographic impact of Kenji Iwakabe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kenji Iwakabe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kenji Iwakabe more than expected).

Fields of papers citing papers by Kenji Iwakabe

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kenji Iwakabe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kenji Iwakabe. The network helps show where Kenji Iwakabe may publish in the future.

Co-authorship network of co-authors of Kenji Iwakabe

This figure shows the co-authorship network connecting the top 25 collaborators of Kenji Iwakabe. A scholar is included among the top collaborators of Kenji Iwakabe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kenji Iwakabe. Kenji Iwakabe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Chamoto, Kenji, Akemi Kosaka, Takemasa Tsuji, et al.. (2003). Critical role of the Th1/Tc1 circuit for the generation of tumor‐specific CTL during tumor eradication in vivo by Th1‐cell therapy. Cancer Science. 94(10). 924–928. 55 indexed citations

Yahata, Takashi, Akio Ohta, Masashi Sekimoto, et al.. (2000). Interleukin-4-dependent induction of preproenkephalin in antigen-specific T helper-type 2 (Th2) cells. Journal of Neuroimmunology. 105(2). 103–108. 10 indexed citations

Ohta, Akio, Masashi Sekimoto, Marimo Sato, et al.. (2000). Potentiation of antitumor effect of NKT cell ligand, α-galactosylceramide by combination with IL-12 on lung metastasis of malignant melanoma cells1. Clinical & Experimental Metastasis. 18(2). 147–153. 36 indexed citations

Kitamura, Hidemitsu, Akio Ohta, Masashi Sekimoto, et al.. (2000). α-Galactosylceramide Induces Early B-Cell Activation through IL-4 Production by NKT Cells. Cellular Immunology. 199(1). 37–42. 121 indexed citations

Sato, Marimo, Kenji Iwakabe, Akio Ohta, et al.. (2000). Self-priming cell culture system for monitoring genetically-controlled spontaneous cytokine-producing ability in mice. Immunology Letters. 70(3). 173–178. 1 indexed citations

Nishimura, T., Hidemitsu Kitamura, Kenji Iwakabe, et al.. (2000). The interface between innate and acquired immunity: glycolipid antigen presentation by CD1d-expressing dendritic cells to NKT cells induces the differentiation of antigen-specific cytotoxic T lymphocytes. International Immunology. 12(7). 987–994. 1 indexed citations

Nishimura, Takashi, M. Sato, Kenji Iwakabe, et al.. (2000). The critical role of Th1-dominant immunity in tumor immunology. Cancer Chemotherapy and Pharmacology. 46(S1). S52–S61. 1 indexed citations

Iwakabe, Kenji, Akio Ohta, Masashi Sekimoto, et al.. (1999). Natural Killer T Cell Ligand α‐Galactosylceramide Inhibited Lymph Node Metastasis of Highly Metastatic Melanoma Cells. Japanese Journal of Cancer Research. 90(8). 801–804. 19 indexed citations

Yahata, Takashi, Naoko Abe, Yasushi Ohmi, et al.. (1999). The essential role of phorbol ester-sensitive protein kinase C isoforms in activation-induced cell death of Th1 cells. European Journal of Immunology. 29(3). 727–732. 20 indexed citations

10.

Nishimura, Takashi, Kenji Iwakabe, Masashi Sekimoto, et al.. (1999). Distinct Role of Antigen-Specific T Helper Type 1 (Th1) and Th2 Cells in Tumor Eradication in Vivo. The Journal of Experimental Medicine. 190(5). 617–628. 18 indexed citations

11.

Sato, Marimo, Kenji Iwakabe, Shuichi Kimura, & Takashi Nishimura. (1999). The influence of dietary protein antigen on Th1/Th2 balance and cellular immunity. Immunology Letters. 70(1). 29–35. 8 indexed citations

12.

Sato, Marimo, Kenji Iwakabe, Shuichi Kimura, & Takashi Nishimura. (1999). Functional skewing of bone marrow-derived dendritic cells by Th1- or Th2-inducing cytokines. Immunology Letters. 67(1). 63–68. 50 indexed citations

13.

Kitamura, Hidemitsu, Kenji Iwakabe, Takashi Yahata, et al.. (1999). The Natural Killer T (NKT) Cell Ligand α-Galactosylceramide Demonstrates Its Immunopotentiating Effect by Inducing Interleukin (IL)-12 Production by Dendritic Cells and IL-12 Receptor Expression on NKT Cells. The Journal of Experimental Medicine. 189(7). 1121–1128. 527 indexed citations breakdown →

14.

Iwakabe, Kenji, Masako Shimada, Akio Ohta, et al.. (1998). The restraint stress drives a shift in Th1/Th2 balance toward Th2-dominant immunity in mice. Immunology Letters. 62(1). 39–43. 104 indexed citations

15.

Yahata, Takashi, Kiyoshi Ando, Kazuhito Watanabe, et al.. (1998). Reconstitution of immune systems in RAG2−/− mice by transfer with interleukin-12-induced splenic hematopoietic progenitor cells. Immunology Letters. 62(3). 165–170. 1 indexed citations

16.

Yahata, Takashi, Kazuhito Watanabe, Akio Ohta, et al.. (1998). Accumulation of IL-12-activated antitumor effector cells into lymph nodes of tumor-bearing mice. Immunology Letters. 61(2-3). 127–133. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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