Keith E. Bryan

611 total citations
8 papers, 428 citations indexed

About

Keith E. Bryan is a scholar working on Molecular Biology, Sensory Systems and Cell Biology. According to data from OpenAlex, Keith E. Bryan has authored 8 papers receiving a total of 428 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 3 papers in Sensory Systems and 3 papers in Cell Biology. Recurrent topics in Keith E. Bryan's work include Hearing, Cochlea, Tinnitus, Genetics (3 papers), Cellular Mechanics and Interactions (3 papers) and Photoreceptor and optogenetics research (2 papers). Keith E. Bryan is often cited by papers focused on Hearing, Cochlea, Tinnitus, Genetics (3 papers), Cellular Mechanics and Interactions (3 papers) and Photoreceptor and optogenetics research (2 papers). Keith E. Bryan collaborates with scholars based in United States, Germany and Denmark. Keith E. Bryan's co-authors include Peter A. Rubenstein, Tobias Moser, Tina Pangršič, Amy Lee, Benjamin Harke, Mark A. Rutherford, Thomas Frank, Alexander Egner, Andreas Neef and Dietmar Riedel and has published in prestigious journals such as Journal of Biological Chemistry, Neuron and Nature Neuroscience.

In The Last Decade

Keith E. Bryan

8 papers receiving 427 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Keith E. Bryan United States	7	242	177	148	119	63	8	428
Eric A. Stauffer United States	5	220 0.9×	283 1.6×	42 0.3×	70 0.6×	71 1.1×	6	436
Elise Pepermans France	9	334 1.4×	287 1.6×	48 0.3×	60 0.5×	48 0.8×	12	486
Runjia Cui United States	12	299 1.2×	360 2.0×	68 0.5×	20 0.2×	74 1.2×	16	559
Nora Overlack Germany	10	638 2.6×	244 1.4×	122 0.8×	92 0.8×	16 0.3×	13	726
Jan Reiners Germany	7	560 2.3×	419 2.4×	121 0.8×	91 0.8×	30 0.5×	8	720
Steve D.M. Brown United Kingdom	7	354 1.5×	316 1.8×	34 0.2×	16 0.1×	63 1.0×	8	522
G. P. Richardson United Kingdom	5	170 0.7×	411 2.3×	41 0.3×	30 0.3×	115 1.8×	6	522
Léa Lepelletier France	6	219 0.9×	155 0.9×	80 0.5×	93 0.8×	24 0.4×	6	353
Janet L. Cyr United States	7	226 0.9×	147 0.8×	167 1.1×	23 0.2×	33 0.5×	10	398
Karin Jürgens Germany	4	359 1.5×	232 1.3×	83 0.6×	65 0.5×	20 0.3×	6	450

Countries citing papers authored by Keith E. Bryan

Since Specialization

Citations

This map shows the geographic impact of Keith E. Bryan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keith E. Bryan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keith E. Bryan more than expected).

Fields of papers citing papers by Keith E. Bryan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keith E. Bryan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keith E. Bryan. The network helps show where Keith E. Bryan may publish in the future.

Co-authorship network of co-authors of Keith E. Bryan

This figure shows the co-authorship network connecting the top 25 collaborators of Keith E. Bryan. A scholar is included among the top collaborators of Keith E. Bryan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keith E. Bryan. Keith E. Bryan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Bryan, Keith E., et al.. (2011). Harmonin inhibits presynaptic Cav1.3 Ca2+ channels in mouse inner hair cells. Nature Neuroscience. 14(9). 1109–1111. 66 indexed citations

2.

Frank, Thomas, Mark A. Rutherford, Nicola Strenzke, et al.. (2010). Bassoon and the Synaptic Ribbon Organize Ca2+ Channels and Vesicles to Add Release Sites and Promote Refilling. Neuron. 68(4). 724–738. 209 indexed citations

3.

Frank, Thomas, Mark A. Rutherford, Nicola Strenzke, et al.. (2010). Bassoon and the Synaptic Ribbon Organize Ca2+ Channels and Vesicles to Add Release Sites and Promote Refilling. Neuron. 68(6). 1202–1202. 1 indexed citations

4.

Morin, Matías, Keith E. Bryan, Fernando Mayo, et al.. (2009). In vivo and in vitro effects of two novel gamma-actin (ACTG1) mutations that cause DFNA20/26 hearing impairment. Human Molecular Genetics. 18(16). 3075–3089. 59 indexed citations

5.

Bryan, Keith E. & Peter A. Rubenstein. (2009). Allele-specific Effects of Human Deafness γ-Actin Mutations (DFNA20/26) on the Actin/Cofilin Interaction. Journal of Biological Chemistry. 284(27). 18260–18269. 16 indexed citations

6.

Bryan, Keith E., Kuo‐Kuang Wen, Mei Zhu, et al.. (2006). Effects of Human Deafness γ-Actin Mutations (DFNA20/26) on Actin Function. Journal of Biological Chemistry. 281(29). 20129–20139. 37 indexed citations

7.

Lü, Xiaoying, et al.. (2005). Role of the N‐terminal negative charges of actin in force generation and cross‐bridge kinetics in reconstituted bovine cardiac muscle fibres. The Journal of Physiology. 564(1). 65–82. 22 indexed citations

8.

Bryan, Keith E. & Peter A. Rubenstein. (2004). An Intermediate Form of ADP-F-actin. Journal of Biological Chemistry. 280(2). 1696–1703. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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