Keita Kanki

1.3k total citations
50 papers, 994 citations indexed

About

Keita Kanki is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, Keita Kanki has authored 50 papers receiving a total of 994 indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Molecular Biology, 10 papers in Cancer Research and 6 papers in Genetics. Recurrent topics in Keita Kanki's work include Retinoids in leukemia and cellular processes (8 papers), Carcinogens and Genotoxicity Assessment (8 papers) and Genomics, phytochemicals, and oxidative stress (7 papers). Keita Kanki is often cited by papers focused on Retinoids in leukemia and cellular processes (8 papers), Carcinogens and Genotoxicity Assessment (8 papers) and Genomics, phytochemicals, and oxidative stress (7 papers). Keita Kanki collaborates with scholars based in Japan, South Korea and United States. Keita Kanki's co-authors include Akiyoshi Nishikawa, Takashi Umemura, Masao Hirose, Yasuki Kitamura, Goshi Shiota, Yuichi Kuroiwa, Hiroki Shimizu, Takayoshi Imazawa, Yuji Ishii and Yukio Kodama and has published in prestigious journals such as PLoS ONE, Cancer Research and Biochemical and Biophysical Research Communications.

In The Last Decade

Keita Kanki

49 papers receiving 961 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Keita Kanki Japan 19 535 255 125 101 97 50 994
Anna Kakehashi Japan 20 463 0.9× 250 1.0× 151 1.2× 125 1.2× 94 1.0× 89 1.0k
Min He China 22 602 1.1× 322 1.3× 105 0.8× 151 1.5× 97 1.0× 76 1.1k
Min Tong United States 20 535 1.0× 237 0.9× 185 1.5× 71 0.7× 57 0.6× 35 1.5k
Janis Fleming United Kingdom 22 768 1.4× 276 1.1× 205 1.6× 77 0.8× 89 0.9× 31 1.4k
Toyohiko Aoki Japan 14 394 0.7× 295 1.2× 95 0.8× 39 0.4× 101 1.0× 38 757
Aline de Conti United States 23 694 1.3× 290 1.1× 77 0.6× 230 2.3× 66 0.7× 57 1.4k
Brian N. Chorley United States 21 835 1.6× 262 1.0× 86 0.7× 100 1.0× 201 2.1× 53 1.4k
Ana Calcagnotto United States 14 556 1.0× 247 1.0× 101 0.8× 47 0.5× 36 0.4× 28 1.2k
Karen Brauers Netherlands 17 498 0.9× 235 0.9× 125 1.0× 38 0.4× 143 1.5× 23 898
Liquan Wang China 21 699 1.3× 152 0.6× 101 0.8× 102 1.0× 89 0.9× 85 1.6k

Countries citing papers authored by Keita Kanki

Since Specialization
Citations

This map shows the geographic impact of Keita Kanki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keita Kanki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keita Kanki more than expected).

Fields of papers citing papers by Keita Kanki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keita Kanki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keita Kanki. The network helps show where Keita Kanki may publish in the future.

Co-authorship network of co-authors of Keita Kanki

This figure shows the co-authorship network connecting the top 25 collaborators of Keita Kanki. A scholar is included among the top collaborators of Keita Kanki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keita Kanki. Keita Kanki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Harada, Kensuke, et al.. (2020). Hedgehog Signal Inhibitor GANT61 Inhibits the Malignant Behavior of Undifferentiated Hepatocellular Carcinoma Cells by Targeting Non-Canonical GLI Signaling. International Journal of Molecular Sciences. 21(9). 3126–3126. 28 indexed citations
2.
Matsumura, Takuro, Yasuharu Ohta, Akihiko Taguchi, et al.. (2020). Liver-specific dysregulation of clock-controlled output signal impairs energy metabolism in liver and muscle. Biochemical and Biophysical Research Communications. 534. 415–421. 9 indexed citations
3.
4.
Kanki, Keita, et al.. (2015). Involvement of glucocorticoid in induction of lingual T1R3 in rodents. European Journal of Pharmacology. 761. 262–267. 4 indexed citations
5.
Kanki, Keita, Yuji Akechi, Hiroyuki Tsuchiya, et al.. (2013). Biological and clinical implications of retinoic acid-responsive genes in human hepatocellular carcinoma cells. Journal of Hepatology. 59(5). 1037–1044. 23 indexed citations
6.
Hoshikawa, Yoshiko, Keita Kanki, Toshihiro Yasui, et al.. (2011). c‐Jun N‐terminal kinase activation by oxidative stress suppresses retinoid signaling through proteasomal degradation of retinoic acid receptor α protein in hepatic cells. Cancer Science. 102(5). 934–941. 19 indexed citations
7.
Tsuchiya, Hiroyuki, Toshihiro Yasui, Keita Kanki, et al.. (2010). Disrupted plasma membrane localization of equilibrative nucleoside transporter 2 in the chemoresistance of human pancreatic cells to gemcitabine (dFdCyd). Cancer Science. 102(3). 622–629. 14 indexed citations
8.
9.
Kuroiwa, Yuichi, Toshiya Okamura, Yuji Ishii, et al.. (2007). Enhancement of esophageal carcinogenesis in acid reflux model rats treated with ascorbic acid and sodium nitrite in combination with or without initiation. Cancer Science. 99(1). 7–13. 10 indexed citations
10.
Ishii, Yuji, Takashi Umemura, Keita Kanki, et al.. (2006). Possible involvement of NO-mediated oxidative stress in induction of rat forestomach damage and cell proliferation by combined treatment with catechol and sodium nitrite. Archives of Biochemistry and Biophysics. 447(2). 127–135. 13 indexed citations
11.
Kitamura, Yasuki, Takashi Umemura, Keita Kanki, et al.. (2006). Increased susceptibility to hepatocarcinogenicity of Nrf2‐deficient mice exposed to 2‐amino‐3‐methylimidazo[4,5‐f]quinoline. Cancer Science. 98(1). 19–24. 58 indexed citations
12.
Umemura, Takashi, Keita Kanki, Yuichi Kuroiwa, et al.. (2006). In vivo mutagenicity and initiation following oxidative DNA lesion in the kidneys of rats given potassium bromate. Cancer Science. 97(9). 829–835. 45 indexed citations
13.
Kitamura, Yasuki, Takashi Umemura, Keita Kanki, et al.. (2006). Lung as a new target in rats of 2‐amino‐3‐methylimidazo[4,5‐f]quinoline carcinogenesis: Results of a two‐stage model initiated with N‐bis(2‐hydroxypropyl)nitrosamine. Cancer Science. 97(5). 368–373. 11 indexed citations
14.
Umemura, Takashi, Yukio Kodama, Akiyoshi Nishikawa, et al.. (2005). Nine-week detection of six genotoxic lung carcinogens using the rasH2/BHT mouse model. Cancer Letters. 231(2). 314–318. 14 indexed citations
15.
16.
Son, Hwa–Young, Akiyoshi Nishikawa, Kazushi Okazaki, et al.. (2004). Specificity of Co-Promoting Effects of Caffeine on Thyroid Carcinogenesis in Rats Pretreated with N-Bis(2-hydroxypropyl)nitrosamine. Toxicologic Pathology. 32(3). 338–344. 5 indexed citations
17.
Son, Hwa–Young, Akiyoshi Nishikawa, Keita Kanki, et al.. (2003). Synergistic interaction between excess caffeine and deficient iodine on the promotion of thyroid carcinogenesis in rats pretreated with N‐bis(2‐hydroxypropyl)nitrosamine. Cancer Science. 94(4). 334–337. 10 indexed citations
18.
Kanki, Keita, Akiyoshi Nishikawa, Fumio Furukawa, et al.. (2003). A 13-week subchronic toxicity study of paprika color in F344 rats. Food and Chemical Toxicology. 41(10). 1337–1343. 17 indexed citations
19.
Kanki, Keita, et al.. (2001). Differentiation behavior of pituitary cells in normal and metamorphosis-arrested larvae of the salamander Hynobius retardatus. The International Journal of Developmental Biology. 45(8). 903–906. 3 indexed citations
20.
Kanki, Keita & Masami Wakahara. (2000). Spatio-Temporal Expression of TSHβ and FSHβ Genes in Normally Metamorphosing, Metamorphosed, and Metamorphosis-Arrested Hynobius retardatus. General and Comparative Endocrinology. 119(3). 276–286. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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