Keiko Miyadera

651 total citations
34 papers, 449 citations indexed

About

Keiko Miyadera is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Keiko Miyadera has authored 34 papers receiving a total of 449 indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 15 papers in Genetics and 8 papers in Cellular and Molecular Neuroscience. Recurrent topics in Keiko Miyadera's work include Retinal Development and Disorders (20 papers), Photoreceptor and optogenetics research (7 papers) and Virus-based gene therapy research (5 papers). Keiko Miyadera is often cited by papers focused on Retinal Development and Disorders (20 papers), Photoreceptor and optogenetics research (7 papers) and Virus-based gene therapy research (5 papers). Keiko Miyadera collaborates with scholars based in United States, Japan and United Kingdom. Keiko Miyadera's co-authors include Gustavo D. Aguirre, Gregory M. Acland, Cathryn S. Mellersh, David R. Sargan, M. E. G. Boursnell, Simone Iwabe, Kumiko Kato, Hiroyuki Ogawa, Evelyn Santana and András M. Komáromy and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and PLoS ONE.

In The Last Decade

Keiko Miyadera

31 papers receiving 435 citations

Peers

Keiko Miyadera

GENET

OPHTH

CMN

Ji‐Neng Lv China

Birgit Budde Germany

Tamara Martı́nez Spain

Yanrong Shi United States

Catherine Willis United States

Marek Pacal Canada

Alicia María United States

Irene A. Aligianis United Kingdom

Katsuhiro Hosono Japan

Zhangyong Wei United States

Ji‐Neng Lv China

Keiko Miyadera

551 ×1.6

110 ×0.6

GENET

121 ×1.2

OPHTH

108 ×1.8

CMN

52 ×0.9

Citations per year, relative to Keiko Miyadera Keiko Miyadera (= 1×) peers Ji‐Neng Lv

Countries citing papers authored by Keiko Miyadera

Since Specialization

Citations

This map shows the geographic impact of Keiko Miyadera's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keiko Miyadera with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keiko Miyadera more than expected).

Fields of papers citing papers by Keiko Miyadera

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keiko Miyadera. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keiko Miyadera. The network helps show where Keiko Miyadera may publish in the future.

Co-authorship network of co-authors of Keiko Miyadera

This figure shows the co-authorship network connecting the top 25 collaborators of Keiko Miyadera. A scholar is included among the top collaborators of Keiko Miyadera based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keiko Miyadera. Keiko Miyadera is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bagel, Jessica, Keiko Miyadera, Gary P. Swain, et al.. (2025). Age-sensitive response of systemic AAV-mediated gene therapy in a newly characterized feline model of mucolipidosis II. Molecular Therapy. 33(8). 3808–3821.

Murgiano, Leonardo, et al.. (2024). A naturally occurring canine model of syndromic congenital microphthalmia. G3 Genes Genomes Genetics. 14(6).

Dufour, Valérie, et al.. (2024). Clinical descriptive and long‐term outcome of melanocytic uveal lesions in young dogs: 40 cases (45 eyes) including 13 cases of sector iridectomy. Veterinary Ophthalmology. 28(2). 371–385. 1 indexed citations

Takahashi, Kei, et al.. (2023). Extended functional rescue following AAV gene therapy in a canine model of LRIT3-congenital stationary night blindness. Vision Research. 209. 108260–108260. 1 indexed citations

Bradbury, Allison M., Jessica Bagel, Gary P. Swain, et al.. (2023). Combination HSCT and intravenous AAV-mediated gene therapy in a canine model proves pivotal for translation of Krabbe disease therapy. Molecular Therapy. 32(1). 44–58. 4 indexed citations

Takahashi, Kei, et al.. (2023). Molecular characterization of MAP9 in the photoreceptor sensory cilia as a modifier in canine RPGRIP1-associated cone-rod dystrophy. Frontiers in Cellular Neuroscience. 17. 1226603–1226603. 2 indexed citations

Miyadera, Keiko, et al.. (2022). Novel insights into chorioretinal and juxtapapillary colobomas by optical coherence tomography. Veterinary Ophthalmology. 25(S1). 136–143. 1 indexed citations

Miyadera, Keiko, Evelyn Santana, Meike Visel, et al.. (2022). Targeting ON-bipolar cells by AAV gene therapy stably reverses LRIT3 -congenital stationary night blindness. Proceedings of the National Academy of Sciences. 119(13). e2117038119–e2117038119. 19 indexed citations

Miyadera, Keiko, Evelyn Santana, Meike Visel, et al.. (2020). AAV gene therapy restores ON-bipolar cell function in a canine model of LRIT3-congenital stationary night blindness. Investigative Ophthalmology & Visual Science. 61(7). 2298–2298. 1 indexed citations

10.

Murgiano, Leonardo, Doreen Becker, Evelyn Santana, et al.. (2020). CCDC66 frameshift variant associated with a new form of early-onset progressive retinal atrophy in Portuguese Water Dogs. Scientific Reports. 10(1). 21162–21162. 11 indexed citations

11.

Bradbury, Allison M., Jessica Bagel, Duc Nguyen, et al.. (2020). Krabbe disease successfully treated via monotherapy of intrathecal gene therapy. Journal of Clinical Investigation. 130(9). 4906–4920. 44 indexed citations

12.

Miyadera, Keiko, Leonardo Murgiano, Valérie Dufour, et al.. (2018). Isolated population helps tease out a third locus underlying a multigenic form of canine RPGRIP1 cone-rod dystrophy. Investigative Ophthalmology & Visual Science. 59(9). 1438–1438. 2 indexed citations

13.

Iwabe, Simone, et al.. (2017). Variabilities in retinal function and structure in a canine model of cone-rod dystrophy associated with RPGRIP1 support multigenic etiology. Scientific Reports. 7(1). 12823–12823. 12 indexed citations

14.

Forman, Oliver P., et al.. (2016). Canine genome assembly correction facilitates identification of a MAP9 deletion as a potential age of onset modifier for RPGRIP1-associated canine retinal degeneration. Mammalian Genome. 27(5-6). 237–245. 21 indexed citations

15.

Kondo, Mineo, Evelyn Santana, Kumiko Kato, et al.. (2015). A Naturally Occurring Canine Model of Autosomal Recessive Congenital Stationary Night Blindness. PLoS ONE. 10(9). e0137072–e0137072. 21 indexed citations

16.

Miyadera, Keiko, et al.. (2014). Molecular and immunohistochemical characterization of a canine model of complete congenital stationary night blindness (CSNB). Investigative Ophthalmology & Visual Science. 55(13). 3288–3288. 1 indexed citations

17.

Miyadera, Keiko, et al.. (2012). Multiple Mechanisms Contribute to Leakiness of a Frameshift Mutation in Canine Cone-Rod Dystrophy. PLoS ONE. 7(12). e51598–e51598. 10 indexed citations

18.

Miyadera, Keiko, et al.. (2012). Investigating the inheritance of prolapsed nictitating membrane glands in a large canine pedigree. Veterinary Ophthalmology. 16(6). 416–422. 18 indexed citations

19.

Miyadera, Keiko, Kumiko Kato, M. E. G. Boursnell, Cathryn S. Mellersh, & David R. Sargan. (2011). Genome-wide association study in RPGRIP1 −/− dogs identifies a modifier locus that determines the onset of retinal degeneration. Mammalian Genome. 23(1-2). 212–223. 26 indexed citations

20.

Tsuji, Takehito, et al.. (2005). An insertion mutation of the bovine F11 gene is responsible for factor XI deficiency in Japanese black cattle. Mammalian Genome. 16(5). 383–389. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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