Kazuyo Takeda

8.4k total citations · 2 hit papers
116 papers, 6.5k citations indexed

About

Kazuyo Takeda is a scholar working on Molecular Biology, Immunology and Epidemiology. According to data from OpenAlex, Kazuyo Takeda has authored 116 papers receiving a total of 6.5k indexed citations (citations by other indexed papers that have themselves been cited), including 58 papers in Molecular Biology, 29 papers in Immunology and 19 papers in Epidemiology. Recurrent topics in Kazuyo Takeda's work include Immune Cell Function and Interaction (10 papers), T-cell and B-cell Immunology (10 papers) and Cardiomyopathy and Myosin Studies (8 papers). Kazuyo Takeda is often cited by papers focused on Immune Cell Function and Interaction (10 papers), T-cell and B-cell Immunology (10 papers) and Cardiomyopathy and Myosin Studies (8 papers). Kazuyo Takeda collaborates with scholars based in United States, Japan and Australia. Kazuyo Takeda's co-authors include Victor J. Ferrans, Toren Finkel, Zu‐Xi Yu, Zu‐Xi Yu, Zu-Xi Yu, Jonathan W. Yewdell, Heather D. Hickman, Joel Moss, Jack R. Bennink and Shino Nemoto and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Kazuyo Takeda

113 papers receiving 6.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Wild Mouse Gut Microbiota Promotes Host Fitness and Improves Disease Resistance

2017 571 citations Stephan P. Rosshart, Brian G. Vassallo et al. Cell profile →
Ras Proteins Induce Senescence by Altering the Intracellular Levels of Reactive Oxygen Species

1999 523 citations Kazuyo Takeda, Zu-Xi Yu et al. Journal of Biological Chemistry profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kazuyo Takeda United States	45	3.2k	1.4k	1.1k	769	761	116	6.5k
Renato Longhi Italy	52	3.9k 1.2×	1.5k 1.1×	524 0.5×	761 1.0×	464 0.6×	210	8.2k
Werner Müller‐Esterl Germany	55	3.6k 1.1×	2.2k 1.5×	1.3k 1.2×	616 0.8×	1.0k 1.3×	185	10.6k
Fiorella Altruda Italy	50	5.1k 1.6×	2.0k 1.4×	816 0.8×	1.7k 2.2×	628 0.8×	156	9.0k
James F. Collawn United States	47	4.0k 1.3×	1.0k 0.7×	764 0.7×	1.7k 2.2×	367 0.5×	144	7.6k
Jun Miyoshi Japan	46	3.9k 1.2×	1.2k 0.9×	560 0.5×	1.1k 1.4×	261 0.3×	160	6.8k
Gethin Thomas Australia	43	3.4k 1.1×	1.3k 0.9×	576 0.5×	583 0.8×	387 0.5×	89	6.7k
Joseph Alroy United States	44	4.5k 1.4×	852 0.6×	1.9k 1.8×	1.1k 1.5×	498 0.7×	204	9.7k
Anne E. Willis United Kingdom	60	8.9k 2.8×	870 0.6×	604 0.6×	1.1k 1.4×	1.1k 1.5×	158	11.3k
Wilbert C. Boelens Netherlands	45	6.0k 1.9×	1.1k 0.7×	761 0.7×	1.1k 1.4×	268 0.4×	90	7.8k
Lawrence W. Stanton Singapore	53	6.9k 2.2×	1.3k 0.9×	542 0.5×	391 0.5×	456 0.6×	124	10.4k

Countries citing papers authored by Kazuyo Takeda

Since Specialization

Citations

This map shows the geographic impact of Kazuyo Takeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kazuyo Takeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kazuyo Takeda more than expected).

Fields of papers citing papers by Kazuyo Takeda

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kazuyo Takeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kazuyo Takeda. The network helps show where Kazuyo Takeda may publish in the future.

Co-authorship network of co-authors of Kazuyo Takeda

This figure shows the co-authorship network connecting the top 25 collaborators of Kazuyo Takeda. A scholar is included among the top collaborators of Kazuyo Takeda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kazuyo Takeda. Kazuyo Takeda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Yao, Xianglan, Meixia Gao, Zu‐Xi Yu, et al.. (2025). Human neutrophils are a cellular source of apolipoprotein A-I. Journal of Leukocyte Biology. 117(7).

Košík, Ivan, Tiansheng Li, Victoria Callahan, et al.. (2024). Recombinant OC43 SARS-CoV-2 spike replacement virus: An improved BSL-2 proxy virus for SARS-CoV-2 neutralization assays. Proceedings of the National Academy of Sciences. 121(29). e2310421121–e2310421121. 7 indexed citations

Wang, Shuibang, Li‐Yuan Chen, Mohammad Abdul Hai Siddique, et al.. (2024). Endothelial PHD2 deficiency induces apoptosis resistance and inflammation via AKT activation and AIP1 loss independent of HIF2α. American Journal of Physiology-Lung Cellular and Molecular Physiology. 327(4). L503–L519. 3 indexed citations

Subramaniam, Sakthivel, Rafaelle Fares‐Gusmao, Shinya Sato, et al.. (2023). Distinct disease features of acute and persistent genotype 3 hepatitis E virus infection in immunocompetent and immunosuppressed Mongolian gerbils. PLoS Pathogens. 19(9). e1011664–e1011664. 10 indexed citations

Nishio, Akira, Kazuyo Takeda, Zu‐Xi Yu, et al.. (2021). Clearance of pegylated interferon by Kupffer cells limits NK cell activation and therapy response of patients with HBV infection. Science Translational Medicine. 13(587). 35 indexed citations

Hickman, Heather D., Jacqueline W. Mays, James S. Gibbs, et al.. (2018). Influenza A Virus Negative Strand RNA Is Translated for CD8+ T Cell Immunosurveillance. The Journal of Immunology. 201(4). 1222–1228. 17 indexed citations

Rosshart, Stephan P., Brian G. Vassallo, Davide Angeletti, et al.. (2017). Wild Mouse Gut Microbiota Promotes Host Fitness and Improves Disease Resistance. Cell. 171(5). 1015–1028.e13. 571 indexed citations breakdown →

Takeda, Kazuyo, et al.. (2015). Hemin activation of innate cellular response blocks human immunodeficiency virus type-1-induced osteoclastogenesis. Biochemical and Biophysical Research Communications. 464(1). 7–12. 3 indexed citations

Wang, Xue, Yamei Gao, Krishnakumar Devadas, et al.. (2012). HIV-1 and HIV-2 infections induce autophagy in Jurkat and CD4+ T cells. Cellular Signalling. 24(7). 1414–1419. 44 indexed citations

10.

Dolan, Brian P., Lily Li, Kazuyo Takeda, Jack R. Bennink, & Jonathan W. Yewdell. (2009). Defective Ribosomal Products Are the Major Source of Antigenic Peptides Endogenously Generated from Influenza A Virus Neuraminidase. The Journal of Immunology. 184(3). 1419–1424. 33 indexed citations

11.

Grebe, Kristie M., Kazuyo Takeda, Heather D. Hickman, et al.. (2009). Cutting Edge: Sympathetic Nervous System Increases Proinflammatory Cytokines and Exacerbates Influenza A Virus Pathogenesis. The Journal of Immunology. 184(2). 540–544. 100 indexed citations

12.

Kostera‐Pruszczyk, Anna, Piotr Pruszczyk, Bertrand Goudeau, et al.. (2006). Skeletal myopathy and restrictive cardiomyopathy with atrioventricular conduction block resulting from desmin mutation. Neuromuscular Disorders. 16. 1 indexed citations

13.

Kumaki, Fumiyuki, Kazuyo Takeda, Zu‐Xi Yu, Joel Moss, & Victor J. Ferrans. (2002). Expression of Human Telomerase Reverse Transcriptase in Lymphangioleiomyomatosis. American Journal of Respiratory and Critical Care Medicine. 166(2). 187–191. 8 indexed citations

14.

Hu, Qinghua, Zu-Xi Yu, Victor J. Ferrans, et al.. (2002). Critical Role of NADPH Oxidase-derived Reactive Oxygen Species in Generating Ca2+ Oscillations in Human Aortic Endothelial Cells Stimulated by Histamine. Journal of Biological Chemistry. 277(36). 32546–32551. 71 indexed citations

15.

Yazawa, Hiroshi, Zu‐Xi Yu, Kazuyo Takeda, et al.. (2001). β Amyloid peptide (Aβ ₄₂ ) is internalized via the G‐protein‐coupled receptor FPRL1 and forms fibrillar aggregates in macrophages ¹. The FASEB Journal. 15(13). 2454–2462. 121 indexed citations

16.

Matsui, Kazuhiro, Kazuyo Takeda, Zu‐Xi Yu, et al.. (2000). Downregulation of Estrogen and Progesterone Receptors in the Abnormal Smooth Muscle Cells in Pulmonary Lymphangioleiomyomatosis Following Therapy: An Immunohistochemical Study. American Journal of Respiratory and Critical Care Medicine. 161(3). 1002–1009. 100 indexed citations

17.

Matsui, Kazuhiro, William Riemenschneider, Stephen L. Hilbert, et al.. (2000). Hyperplasia of Type II Pneumocytes in Pulmonary Lymphangioleiomyomatosis Immunohistochemical and Electron Microscopic Study. Archives of Pathology & Laboratory Medicine. 124(11). 1642–1648. 5 indexed citations

18.

Kim, Kyo Sang, Kazuyo Takeda, J.B. Pracyk, et al.. (1998). Protection from reoxygenation injury by inhibition of rac1.. Journal of Clinical Investigation. 101(9). 1821–1826. 129 indexed citations

19.

Nishikawa, Toshio, et al.. (1996). Hypertrophic cardiomyopathy in Noonan syndrome. Pediatrics International. 38(1). 91–98. 31 indexed citations

20.

Ishiyama, Shigeru, et al.. (1995). The effect of forskolin on the teratogenicity of methylxanthines in the chick embryo heart. Reproductive Toxicology. 9(2). 165–168. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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