Katsuhiro Uto

695 total citations
8 papers, 501 citations indexed

About

Katsuhiro Uto is a scholar working on Molecular Biology, Cell Biology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Katsuhiro Uto has authored 8 papers receiving a total of 501 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Cell Biology and 3 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Katsuhiro Uto's work include Microtubule and mitosis dynamics (6 papers), Reproductive Biology and Fertility (3 papers) and DNA Repair Mechanisms (2 papers). Katsuhiro Uto is often cited by papers focused on Microtubule and mitosis dynamics (6 papers), Reproductive Biology and Fertility (3 papers) and DNA Repair Mechanisms (2 papers). Katsuhiro Uto collaborates with scholars based in Japan and Germany. Katsuhiro Uto's co-authors include Noriyuki Sagata, Nobushige Nakajo, Yoshinori Kanemori, Daigo Inoue, Jun Iwashita, Kengo Okamoto, Shuichi Ueno, Tomoya Oe, Andreas Schnapp and Eva Wex and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Genes & Development and The EMBO Journal.

In The Last Decade

Katsuhiro Uto

8 papers receiving 497 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Katsuhiro Uto Japan	8	415	270	128	90	34	8	501
Dan Chase United States	11	546 1.3×	349 1.3×	182 1.4×	32 0.4×	14 0.4×	11	684
Gyosuke Sakashita Japan	12	365 0.9×	227 0.8×	92 0.7×	20 0.2×	18 0.5×	17	439
Zaira García Spain	7	390 0.9×	144 0.5×	112 0.9×	15 0.2×	32 0.9×	7	477
Marı́a Ana Gómez-Ferrerı́a United States	12	416 1.0×	288 1.1×	59 0.5×	21 0.2×	27 0.8×	13	541
María Guillamot United States	9	475 1.1×	272 1.0×	150 1.2×	21 0.2×	39 1.1×	10	603
Claudio Alfieri United Kingdom	12	717 1.7×	478 1.8×	106 0.8×	25 0.3×	16 0.5×	15	810
Wytse Bruinsma Netherlands	9	515 1.2×	312 1.2×	212 1.7×	18 0.2×	14 0.4×	9	640
Mary E. Gagou United Kingdom	11	639 1.5×	285 1.1×	156 1.2×	21 0.2×	18 0.5×	12	701
Dominik Boos Germany	13	749 1.8×	446 1.7×	165 1.3×	60 0.7×	10 0.3×	20	852
Linda Roy United States	8	315 0.8×	217 0.8×	122 1.0×	139 1.5×	5 0.1×	9	419

Countries citing papers authored by Katsuhiro Uto

Since Specialization

Citations

This map shows the geographic impact of Katsuhiro Uto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katsuhiro Uto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katsuhiro Uto more than expected).

Fields of papers citing papers by Katsuhiro Uto

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katsuhiro Uto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katsuhiro Uto. The network helps show where Katsuhiro Uto may publish in the future.

Co-authorship network of co-authors of Katsuhiro Uto

This figure shows the co-authorship network connecting the top 25 collaborators of Katsuhiro Uto. A scholar is included among the top collaborators of Katsuhiro Uto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katsuhiro Uto. Katsuhiro Uto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Sanderson, Michael P., Eva Wex, Takeshi Kono, Katsuhiro Uto, & Andreas Schnapp. (2010). Syk and Lyn mediate distinct Syk phosphorylation events in FcɛRI-signal transduction: Implications for regulation of IgE-mediated degranulation. Molecular Immunology. 48(1-3). 171–178. 30 indexed citations

2.

Kanemori, Yoshinori, Katsuhiro Uto, & Noriyuki Sagata. (2005). β-TrCP recognizes a previously undescribed nonphosphorylated destruction motif in Cdc25A and Cdc25B phosphatases. Proceedings of the National Academy of Sciences. 102(18). 6279–6284. 102 indexed citations

3.

Uto, Katsuhiro, et al.. (2004). Chk1, but not Chk2, inhibits Cdc25 phosphatases by a novel common mechanism. The EMBO Journal. 23(16). 3386–3396. 94 indexed citations

4.

Uto, Katsuhiro. (2000). Nek2B, a novel maternal form of Nek2 kinase, is essential for the assembly or maintenance of centrosomes in early Xenopus embryos. The EMBO Journal. 19(8). 1816–1826. 75 indexed citations

5.

Nakajo, Nobushige, Jun Iwashita, Katsuhiro Uto, et al.. (2000). Absence of Wee1 ensures the meiotic cell cycle in Xenopus oocytes. Genes & Development. 14(3). 328–338. 108 indexed citations

6.

Nakajo, Nobushige, Jun Iwashita, Katsuhiro Uto, et al.. (2000). Nakajo, N. et al. Absence of Wee1 ensures the meiotic cell cycle in Xenopus oocytes. Genes Dev. 14, 328-338. 11 indexed citations

7.

Nakajo, Nobushige, Tomoya Oe, Katsuhiro Uto, & Noriyuki Sagata. (1999). Involvement of Chk1 Kinase in Prophase I Arrest of Xenopus Oocytes. Developmental Biology. 207(2). 432–444. 41 indexed citations

8.

Uto, Katsuhiro, Nobushige Nakajo, & Noriyuki Sagata. (1999). Two Structural Variants of Nek2 Kinase, Termed Nek2A and Nek2B, Are Differentially Expressed inXenopusTissues and Development. Developmental Biology. 208(2). 456–464. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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