Kate E. Gilling

578 total citations
17 papers, 461 citations indexed

About

Kate E. Gilling is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Kate E. Gilling has authored 17 papers receiving a total of 461 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Cellular and Molecular Neuroscience, 11 papers in Molecular Biology and 3 papers in Cognitive Neuroscience. Recurrent topics in Kate E. Gilling's work include Neuroscience and Neuropharmacology Research (13 papers), Ion channel regulation and function (7 papers) and Receptor Mechanisms and Signaling (4 papers). Kate E. Gilling is often cited by papers focused on Neuroscience and Neuropharmacology Research (13 papers), Ion channel regulation and function (7 papers) and Receptor Mechanisms and Signaling (4 papers). Kate E. Gilling collaborates with scholars based in Germany, United States and Russia. Kate E. Gilling's co-authors include Chris G. Parsons, Claudia Jatzke, Mirko Hechenberger, Gerhard Rammes, Rainer Rupprecht, Gerald Gimpl, W. Zieglgänsberger, Gerhard Hapfelmeier, B Eisensamer and Brigitta Bondy and has published in prestigious journals such as Neuroscience, Molecular Psychiatry and Antioxidants and Redox Signaling.

In The Last Decade

Kate E. Gilling

17 papers receiving 454 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kate E. Gilling Germany 11 257 219 93 62 55 17 461
Katarína Lichnerová Czechia 11 346 1.3× 290 1.3× 81 0.9× 63 1.0× 35 0.6× 11 532
Bruce Ladenheim United States 9 403 1.6× 161 0.7× 105 1.1× 46 0.7× 47 0.9× 11 542
Takeo Funakoshi Japan 15 301 1.2× 321 1.5× 84 0.9× 84 1.4× 30 0.5× 22 763
David M. Otte Germany 11 189 0.7× 192 0.9× 170 1.8× 75 1.2× 33 0.6× 16 492
Takahide Shuto Japan 13 344 1.3× 421 1.9× 184 2.0× 45 0.7× 43 0.8× 24 697
Michael Di Palma Italy 14 311 1.2× 327 1.5× 40 0.4× 81 1.3× 56 1.0× 33 643
Bernadett K. Szász Hungary 10 212 0.8× 153 0.7× 72 0.8× 70 1.1× 34 0.6× 11 361
Maria S. Quinton United States 13 297 1.2× 204 0.9× 87 0.9× 47 0.8× 21 0.4× 21 543
M. Amin Khan United States 13 185 0.7× 205 0.9× 149 1.6× 70 1.1× 22 0.4× 16 579
Joana M. Marques Portugal 13 313 1.2× 264 1.2× 60 0.6× 75 1.2× 47 0.9× 19 655

Countries citing papers authored by Kate E. Gilling

Since Specialization
Citations

This map shows the geographic impact of Kate E. Gilling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kate E. Gilling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kate E. Gilling more than expected).

Fields of papers citing papers by Kate E. Gilling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kate E. Gilling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kate E. Gilling. The network helps show where Kate E. Gilling may publish in the future.

Co-authorship network of co-authors of Kate E. Gilling

This figure shows the co-authorship network connecting the top 25 collaborators of Kate E. Gilling. A scholar is included among the top collaborators of Kate E. Gilling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kate E. Gilling. Kate E. Gilling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Grosser, Sabine, Karl‐Heinz Braunewell, Kate E. Gilling, et al.. (2020). Loss of Long-Term Potentiation at Hippocampal Output Synapses in Experimental Temporal Lobe Epilepsy. Frontiers in Molecular Neuroscience. 13. 143–143. 13 indexed citations
2.
Wagner, Lisa Katharina, Kate E. Gilling, Peter M. Kloetzel, et al.. (2017). Immunoproteasome deficiency alters microglial cytokine response and improves cognitive deficits in Alzheimer’s disease-like APPPS1 mice. Acta Neuropathologica Communications. 5(1). 52–52. 47 indexed citations
3.
Sotzny, Franziska, Annett Koch, Anja Brehm, et al.. (2016). TCF11/Nrf1-Mediated Induction of Proteasome Expression Prevents Cytotoxicity by Rotenone. Antioxidants and Redox Signaling. 25(16). 870–885. 30 indexed citations
4.
Gruber, David, Kate E. Gilling, Anne Albrecht, et al.. (2015). 5-HT receptor-mediated modulation of granule cell inhibition after juvenile stress recovers after a second exposure to adult stress. Neuroscience. 293. 67–79. 15 indexed citations
5.
Grosser, Sabine, et al.. (2014). Gating of hippocampal output by β-adrenergic receptor activation in the pilocarpine model of epilepsy. Neuroscience. 286. 325–337. 6 indexed citations
6.
7.
Gilling, Kate E., Frank Oltmanns, & Joachim Behr. (2012). Impaired maturation of serotonergic function in the dentate gyrus associated with epilepsy. Neurobiology of Disease. 50. 86–95. 6 indexed citations
8.
Nothdurfter, Caroline, Barbara Di Benedetto, Manfred Uhr, et al.. (2012). Lipid raft integrity affects GABAA receptor, but not NMDA receptor modulation by psychopharmacological compounds. The International Journal of Neuropsychopharmacology. 16(6). 1361–1371. 29 indexed citations
9.
10.
Parsons, Chris G., Kate E. Gilling, & Claudia Jatzke. (2008). Blocking kinetics of memantine on NR1a/2A receptors recorded in inside-out and outside-out patches from Xenopus oocytes. Journal of Neural Transmission. 115(10). 1367–1373. 6 indexed citations
11.
Parsons, Chris G., Kate E. Gilling, & Claudia Jatzke. (2008). Memantine does not show intracellular block of the NMDA receptor channel. European Journal of Pharmacology. 587(1-3). 99–103. 14 indexed citations
12.
Parsons, Chris G. & Kate E. Gilling. (2007). Memantine as an Example of a Fast, Voltage-Dependent, Open Channel N-Methyl-d-Aspartate Receptor Blocker. Methods in molecular biology. 403. 15–36. 35 indexed citations
13.
Gilling, Kate E., Claudia Jatzke, & Chris G. Parsons. (2007). Agonist concentration dependency of blocking kinetics but not equilibrium block of N-methyl-d-aspartate receptors by memantine. Neuropharmacology. 53(3). 415–420. 24 indexed citations
14.
Gilling, Kate E., et al.. (2007). A novel class of amino-alkylcyclohexanes as uncompetitive, fast, voltage-dependent, N-methyl-D-aspartate (NMDA) receptor antagonists – in vitro characterization. Journal of Neural Transmission. 114(12). 1529–1537. 10 indexed citations
15.
Rammes, Gerhard, B Eisensamer, Uta Ferrari, et al.. (2004). Antipsychotic drugs antagonize human serotonin type 3 receptor currents in a noncompetitive manner. Molecular Psychiatry. 9(9). 846–858. 55 indexed citations
16.
Eisensamer, B, Gerhard Rammes, Gerald Gimpl, et al.. (2004). Antidepressants are functional antagonists at the serotonin type 3 (5-HT3) receptor. Pharmacopsychiatry. 36(5). 1 indexed citations
17.
Eisensamer, B, Gerhard Rammes, Gerald Gimpl, et al.. (2003). Antidepressants are functional antagonists at the serotonin type 3 (5-HT3) receptor. Molecular Psychiatry. 8(12). 994–1007. 105 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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