Katalin Hantó

467 total citations
10 papers, 404 citations indexed

About

Katalin Hantó is a scholar working on Cardiology and Cardiovascular Medicine, Pathology and Forensic Medicine and Oncology. According to data from OpenAlex, Katalin Hantó has authored 10 papers receiving a total of 404 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cardiology and Cardiovascular Medicine, 4 papers in Pathology and Forensic Medicine and 4 papers in Oncology. Recurrent topics in Katalin Hantó's work include Cardiac electrophysiology and arrhythmias (5 papers), Cardiac Ischemia and Reperfusion (4 papers) and PARP inhibition in cancer therapy (4 papers). Katalin Hantó is often cited by papers focused on Cardiac electrophysiology and arrhythmias (5 papers), Cardiac Ischemia and Reperfusion (4 papers) and PARP inhibition in cancer therapy (4 papers). Katalin Hantó collaborates with scholars based in Hungary. Katalin Hantó's co-authors include Balázs Sümegi, Ferenc Gallyas, Antal Tapodi, Zita Bognár, Gábor Várbiró, Balázs Debreceni, István Wittmann, Kálmán Hideg, Tamás Kálai and Anita Pálfi and has published in prestigious journals such as Journal of Biological Chemistry, Free Radical Biology and Medicine and Journal of Pharmacology and Experimental Therapeutics.

In The Last Decade

Katalin Hantó

10 papers receiving 401 citations

Peers

Katalin Hantó
Comparison fields: 5 of 71
  • Molecular Biology 216
  • Oncology 184
  • Cardiology and Cardiovascular Medicine 88
  • Pathology and Forensic Medicine 76
  • Immunology 30
Krisztián Erős Hungary
Kyle T. Keyes United States
Huiquan Lou Canada
Lingling Yao China
Ramaneeya Nithipongvanitch Thailand
Jian Cao China
Roberto Bolli United States
Xiaoyang Huang China
Guangping Lu China
Boquan Wu China
Krisztián Erős Hungary View profile →
Citations per field, relative to Katalin Hantó
Katalin Hantó · 1×
Citations per year, relative to Katalin Hantó
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Countries citing papers authored by Katalin Hantó

Since Specialization
Citations

This map shows the geographic impact of Katalin Hantó's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katalin Hantó with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katalin Hantó more than expected).

Fields of papers citing papers by Katalin Hantó

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katalin Hantó. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katalin Hantó. The network helps show where Katalin Hantó may publish in the future.

Co-authorship network of co-authors of Katalin Hantó

This figure shows the co-authorship network connecting the top 25 collaborators of Katalin Hantó. A scholar is included among the top collaborators of Katalin Hantó based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katalin Hantó. Katalin Hantó is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
# Title Journal Authors Indexed citations
1 Regulation of MKP-1 expression and MAPK activation by PARP-1 in oxidative stress: A new mechanism for the cytoplasmic effect of PARP-1 activation Free Radical Biology and Medicine Bóglárka Rácz, Katalin Hantó et al. 49
2 The effect of carotid stenting on rheological parameters, free radical production and platelet aggregation Clinical Hemorheology and Microcirculation László Szapáry, Ferenc Kövér et al. 10
3 Prevalent role of Akt and ERK activation in cardioprotective effect of Ca2+ channel- and beta-adrenergic receptor blockers Molecular and Cellular Biochemistry Krisztina Kovács, Katalin Hantó et al. 24
4 The effect of carotid stenting on rheological parameters, free radical production and platelet aggregation Orvosi Hetilap Gergely Fehér, Ferenc Kövér et al. 1
5 A novel SOD-mimetic permeability transition inhibitor agent protects ischemic heart by inhibiting both apoptotic and necrotic cell death Free Radical Biology and Medicine Zita Bognár, Tamás Kálai et al. 35
6 PARP inhibition prevents postinfarction myocardial remodeling and heart failure via the protein kinase C/glycogen synthase kinase-3β pathway☆ Journal of Molecular and Cellular Cardiology Anita Pálfi, Ambrus Tóth et al. 54
7 Synthesis and evaluation of the permeability transition inhibitory characteristics of paramagnetic and diamagnetic amiodarone derivatives Bioorganic & Medicinal Chemistry Tamás Kálai, Gábor Várbiró et al. 24
8 Pivotal Role of Akt Activation in Mitochondrial Protection and Cell Survival by Poly(ADP-ribose)polymerase-1 Inhibition in Oxidative Stress Journal of Biological Chemistry Antal Tapodi, Balázs Debreceni et al. 148
9 The Role of Akt and Mitogen-Activated Protein Kinase Systems in the Protective Effect of Poly(ADP-Ribose) Polymerase Inhibition in Langendorff Perfused and in Isoproterenol-Damaged Rat Hearts Journal of Pharmacology and Experimental Therapeutics Anita Pálfi, Ambrus Tóth et al. 43
10 Impact of a novel cardioprotective agent on the ischaemia-reperfusion-induced Akt kinase activation Biochemical Pharmacology Ambrus Tóth, Krisztina Kovács et al. 16

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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