Kari R. Hoyt

2.5k total citations
42 papers, 2.1k citations indexed

About

Kari R. Hoyt is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Endocrine and Autonomic Systems. According to data from OpenAlex, Kari R. Hoyt has authored 42 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Cellular and Molecular Neuroscience, 22 papers in Molecular Biology and 10 papers in Endocrine and Autonomic Systems. Recurrent topics in Kari R. Hoyt's work include Neuroscience and Neuropharmacology Research (16 papers), Mitochondrial Function and Pathology (12 papers) and Circadian rhythm and melatonin (10 papers). Kari R. Hoyt is often cited by papers focused on Neuroscience and Neuropharmacology Research (16 papers), Mitochondrial Function and Pathology (12 papers) and Circadian rhythm and melatonin (10 papers). Kari R. Hoyt collaborates with scholars based in United States, South Korea and United Kingdom. Kari R. Hoyt's co-authors include Karl Obrietan, Ian J. Reynolds, Elias Aizenman, Teresa G. Hastings, Boyoung Lee, Yun‐Sik Choi, Hee‐Yeon Cho, Greg Q. Butcher, Sydney Aten and C. Thong and has published in prestigious journals such as Journal of Neuroscience, Molecular Cell and PLoS ONE.

In The Last Decade

Kari R. Hoyt

41 papers receiving 2.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kari R. Hoyt United States 25 1.1k 1.0k 390 317 205 42 2.1k
Elisenda Sanz Spain 19 1.4k 1.3× 647 0.6× 411 1.1× 330 1.0× 372 1.8× 36 2.7k
Angela Ho Canada 28 1.4k 1.3× 953 0.9× 631 1.6× 361 1.1× 152 0.7× 88 2.5k
Fulvio Florenzano Italy 32 1.0k 0.9× 820 0.8× 898 2.3× 292 0.9× 389 1.9× 73 2.9k
C. Gabriel France 31 569 0.5× 673 0.7× 473 1.2× 506 1.6× 291 1.4× 59 2.4k
Geraldine J. Kress United States 17 966 0.9× 831 0.8× 271 0.7× 127 0.4× 243 1.2× 19 1.9k
Juan José Garrido Spain 26 1.1k 1.0× 960 1.0× 299 0.8× 111 0.4× 211 1.0× 50 2.2k
Marco Milanese Italy 30 900 0.8× 888 0.9× 295 0.8× 104 0.3× 329 1.6× 88 2.5k
Ross D. O’Shea Australia 22 703 0.6× 783 0.8× 237 0.6× 167 0.5× 243 1.2× 40 1.7k
Emil C. Toescu United Kingdom 32 1.5k 1.4× 1.3k 1.3× 591 1.5× 124 0.4× 443 2.2× 63 2.9k
Rugao Liu United States 24 744 0.7× 433 0.4× 576 1.5× 395 1.2× 244 1.2× 28 2.0k

Countries citing papers authored by Kari R. Hoyt

Since Specialization
Citations

This map shows the geographic impact of Kari R. Hoyt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kari R. Hoyt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kari R. Hoyt more than expected).

Fields of papers citing papers by Kari R. Hoyt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kari R. Hoyt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kari R. Hoyt. The network helps show where Kari R. Hoyt may publish in the future.

Co-authorship network of co-authors of Kari R. Hoyt

This figure shows the co-authorship network connecting the top 25 collaborators of Kari R. Hoyt. A scholar is included among the top collaborators of Kari R. Hoyt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kari R. Hoyt. Kari R. Hoyt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hoyt, Kari R. & Karl Obrietan. (2022). Circadian clocks, cognition, and Alzheimer’s disease: synaptic mechanisms, signaling effectors, and chronotherapeutics. Molecular Neurodegeneration. 17(1). 35–35. 44 indexed citations
2.
Aten, Sydney, et al.. (2021). Light-induced changes in the suprachiasmatic nucleus transcriptome regulated by the ERK/MAPK pathway. PLoS ONE. 16(6). e0249430–e0249430. 10 indexed citations
3.
Aten, Sydney, et al.. (2020). SynGAP is expressed in the murine suprachiasmatic nucleus and regulates circadian‐gated locomotor activity and light‐entrainment capacity. European Journal of Neuroscience. 53(3). 732–749. 7 indexed citations
4.
Aten, Sydney, et al.. (2018). miR-132/212 is induced by stress and its dysregulation triggers anxiety-related behavior. Neuropharmacology. 144. 256–270. 32 indexed citations
5.
Kumar, Sanjay, Christopher C. Pan, Nirav Shah, et al.. (2016). Activation of Mitofusin2 by Smad2-RIN1 Complex during Mitochondrial Fusion. Molecular Cell. 62(4). 520–531. 43 indexed citations
6.
Karelina, Kate, et al.. (2014). Mitogen and stress-activated kinases 1/2 regulate ischemia-induced hippocampal progenitor cell proliferation and neurogenesis. Neuroscience. 285. 292–302. 25 indexed citations
7.
Godshalk, Sirie E., Trupti Paranjape, Sunitha Nallur, et al.. (2010). A Variant in a MicroRNA complementary site in the 3′ UTR of the KIT oncogene increases risk of acral melanoma. Oncogene. 30(13). 1542–1550. 32 indexed citations
8.
Choi, Yun‐Sik, Boyoung Lee, Hee‐Yeon Cho, et al.. (2009). CREB is a key regulator of striatal vulnerability in chemical and genetic models of Huntington's disease. Neurobiology of Disease. 36(2). 259–268. 50 indexed citations
9.
Lee, Boyoung, Ruifeng Cao, Yun‐Sik Choi, et al.. (2008). The CREB/CRE transcriptional pathway: protection against oxidative stress‐mediated neuronal cell death. Journal of Neurochemistry. 108(5). 1251–1265. 139 indexed citations
10.
Thong, C., Michael J. Mihm, John Anthony Bauer, & Kari R. Hoyt. (2007). Bioenergetic and oxidative effects of free 3‐nitrotyrosine in culture: selective vulnerability of dopaminergic neurons and increased sensitivity of non‐dopaminergic neurons to dopamine oxidation. Journal of Neurochemistry. 103(1). 131–144. 9 indexed citations
11.
Mihm, Michael J., et al.. (2006). Cardiac dysfunction in the R6/2 mouse model of Huntington’s disease. Neurobiology of Disease. 25(2). 297–308. 116 indexed citations
12.
Lee, Boyoung, Greg Q. Butcher, Kari R. Hoyt, Soren Impey, & Karl Obrietan. (2005). Activity-Dependent Neuroprotection and cAMP Response Element-Binding Protein (CREB): Kinase Coupling, Stimulus Intensity, and Temporal Regulation of CREB Phosphorylation at Serine 133. Journal of Neuroscience. 25(5). 1137–1148. 144 indexed citations
13.
Obrietan, Karl & Kari R. Hoyt. (2004). CRE-Mediated Transcription Is Increased in Huntington's Disease Transgenic Mice. Journal of Neuroscience. 24(4). 791–796. 82 indexed citations
14.
Dziema, Heather, et al.. (2003). The ERK/MAP kinase pathway couples light to immediate‐early gene expression in the suprachiasmatic nucleus. European Journal of Neuroscience. 17(8). 1617–1627. 98 indexed citations
15.
16.
Hoyt, Kari R., et al.. (1997). Characterization of Hydrogen Peroxide Toxicity in Cultured Rat Forebrain Neurons. Neurochemical Research. 22(3). 333–340. 101 indexed citations
17.
Hoyt, Kari R., et al.. (1997). Trifluoperazine and dibucaine‐induced inhibition of glutamate‐induced mitochondrial depolarization in rat cultured forebrain neurones. British Journal of Pharmacology. 122(5). 803–808. 44 indexed citations
18.
Hoyt, Kari R. & Ian J. Reynolds. (1996). Localization of D1 dopamine receptors on live cultured striatal neurons by quantitative fluorescence microscopy. Brain Research. 731(1-2). 21–30. 7 indexed citations
19.
Hoyt, Kari R., Sunita Rajdev, Cheryl L. Fattman, & Ian J. Reynolds. (1995). Cyclothiazide Modulates AMPA Receptor‐Mediated Increases in Intracellular Free Ca2+ and Mg2+ in Cultured Neurons from Rat Brain. Journal of Neurochemistry. 64(5). 2049–2056. 31 indexed citations
20.
Hoyt, Kari R., et al.. (1992). Nitric oxide modulates NMDA-induced increases in intracellular Ca2+ in cultured rat forebrain neurons. Brain Research. 592(1-2). 310–316. 124 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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