Kaouther Snoussi

474 total citations
7 papers, 399 citations indexed

About

Kaouther Snoussi is a scholar working on Oncology, Molecular Biology and Epidemiology. According to data from OpenAlex, Kaouther Snoussi has authored 7 papers receiving a total of 399 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Oncology, 2 papers in Molecular Biology and 2 papers in Epidemiology. Recurrent topics in Kaouther Snoussi's work include Cytokine Signaling Pathways and Interactions (4 papers), Chemokine receptors and signaling (2 papers) and Glycosylation and Glycoproteins Research (1 paper). Kaouther Snoussi is often cited by papers focused on Cytokine Signaling Pathways and Interactions (4 papers), Chemokine receptors and signaling (2 papers) and Glycosylation and Glycoproteins Research (1 paper). Kaouther Snoussi collaborates with scholars based in Tunisia, Qatar and United States. Kaouther Snoussi's co-authors include Noureddine Bouaouina, Lotfi Chouchane, Slim Ben Ahmed, Ahmed Noureddine Helal, A. Donny Strosberg, Wijden Mahfoudh, Hédi Khaı̈ri, M. Fékih, Hela Ben Nasr and Souhir Mestiri and has published in prestigious journals such as BMC Cancer, Journal of Translational Medicine and Human Immunology.

In The Last Decade

Kaouther Snoussi

7 papers receiving 387 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kaouther Snoussi Tunisia	6	183	121	107	96	78	7	399
Sweena M. Chaudhari Germany	8	101 0.6×	255 2.1×	86 0.8×	40 0.4×	51 0.7×	8	444
Gisela Sanchez‐Williams United States	8	159 0.9×	56 0.5×	261 2.4×	45 0.5×	108 1.4×	8	601
Anke Neuwirth United States	4	201 1.1×	82 0.7×	152 1.4×	152 1.6×	55 0.7×	6	539
Nathan Richoz United Kingdom	9	88 0.5×	254 2.1×	161 1.5×	47 0.5×	16 0.2×	14	444
Luz Agana United States	8	100 0.5×	79 0.7×	127 1.2×	40 0.4×	14 0.2×	8	393
Guoqiang Bao China	12	74 0.4×	37 0.3×	147 1.4×	50 0.5×	38 0.5×	35	437
Songcheng Yin China	14	246 1.3×	149 1.2×	215 2.0×	118 1.2×	17 0.2×	32	634
Zhijie Yu China	10	78 0.4×	108 0.9×	326 3.0×	203 2.1×	13 0.2×	17	644
Sylwia Grodecka-Gazdecka Poland	11	144 0.8×	53 0.4×	182 1.7×	105 1.1×	11 0.1×	35	376
Yih‐Hsin Chang Taiwan	8	55 0.3×	65 0.5×	68 0.6×	51 0.5×	9 0.1×	18	241

Countries citing papers authored by Kaouther Snoussi

Since Specialization

Citations

This map shows the geographic impact of Kaouther Snoussi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kaouther Snoussi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kaouther Snoussi more than expected).

Fields of papers citing papers by Kaouther Snoussi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kaouther Snoussi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kaouther Snoussi. The network helps show where Kaouther Snoussi may publish in the future.

Co-authorship network of co-authors of Kaouther Snoussi

This figure shows the co-authorship network connecting the top 25 collaborators of Kaouther Snoussi. A scholar is included among the top collaborators of Kaouther Snoussi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kaouther Snoussi. Kaouther Snoussi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Snoussi, Kaouther, et al.. (2024). Atypical Cogan syndrome: a case report. Journal of Medical Case Reports. 18(1). 551–551. 1 indexed citations

2.

Mahfoudh, Wijden, Kaouther Snoussi, Sallouha Gabbouj, et al.. (2019). Contribution of BRCA1 5382insC mutation in triple negative breast cancer in Tunisia. Journal of Translational Medicine. 17(1). 123–123. 18 indexed citations

3.

Snoussi, Kaouther, Wijden Mahfoudh, Noureddine Bouaouina, et al.. (2010). Combined effects of IL-8 and CXCR2gene polymorphisms on breast cancer susceptibility and aggressiveness. BMC Cancer. 10(1). 283–283. 88 indexed citations

4.

Nasr, Hela Ben, Karim Chahed, Souhir Mestiri, et al.. (2007). Association of IL-8 (−251)T/A polymorphism with susceptibility to and aggressiveness of nasopharyngeal carcinoma. Human Immunology. 68(9). 761–769. 32 indexed citations

5.

Snoussi, Kaouther, Wijden Mahfoudh, Noureddine Bouaouina, et al.. (2006). Genetic Variation in IL-8 Associated with Increased Risk and Poor Prognosis of Breast Carcinoma. Human Immunology. 67(1-2). 13–21. 62 indexed citations

6.

Snoussi, Kaouther, A. Donny Strosberg, Noureddine Bouaouina, et al.. (2006). Leptin and leptin receptor polymorphisms are associated with increased risk and poor prognosis of breast carcinoma. BMC Cancer. 6(1). 38–38. 124 indexed citations

7.

Snoussi, Kaouther, A. Donny Strosberg, Noureddine Bouaouina, Slim Ben Ahmed, & Lotfi Chouchane. (2005). Genetic variation in pro-inflammatory cytokines (interleukin-1beta, interleukin-1alpha and interleukin-6) associated with the aggressive forms, survival, and relapse prediction of breast carcinoma.. PubMed. 16(4). 253–60. 74 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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