Kanako Katai

822 total citations
20 papers, 642 citations indexed

About

Kanako Katai is a scholar working on Nephrology, Molecular Biology and Genetics. According to data from OpenAlex, Kanako Katai has authored 20 papers receiving a total of 642 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Nephrology, 8 papers in Molecular Biology and 8 papers in Genetics. Recurrent topics in Kanako Katai's work include Parathyroid Disorders and Treatments (14 papers), Magnesium in Health and Disease (6 papers) and Genetic Syndromes and Imprinting (5 papers). Kanako Katai is often cited by papers focused on Parathyroid Disorders and Treatments (14 papers), Magnesium in Health and Disease (6 papers) and Genetic Syndromes and Imprinting (5 papers). Kanako Katai collaborates with scholars based in Japan and France. Kanako Katai's co-authors include Sawako Tatsumi, Ken‐ichi Miyamoto, Kyoko Morita, Yutaka Taketani, Hiroko Segawa, Eiji Takeda, Tomoko Nii, Hironori Yamamoto, Hiromi Haga and Hidekazu Arai and has published in prestigious journals such as Journal of Biological Chemistry, Biochemical Journal and Endocrinology.

In The Last Decade

Kanako Katai

20 papers receiving 626 citations

Peers

Kanako Katai
J�rg Biber Switzerland
Thomas Knöpfel Switzerland
Hubert K. Zajicek United States
Viola Pomozi Hungary
Sunny Mahakena Netherlands
J�rg Biber Switzerland
Kanako Katai
Citations per year, relative to Kanako Katai Kanako Katai (= 1×) peers J�rg Biber

Countries citing papers authored by Kanako Katai

Since Specialization
Citations

This map shows the geographic impact of Kanako Katai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kanako Katai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kanako Katai more than expected).

Fields of papers citing papers by Kanako Katai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kanako Katai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kanako Katai. The network helps show where Kanako Katai may publish in the future.

Co-authorship network of co-authors of Kanako Katai

This figure shows the co-authorship network connecting the top 25 collaborators of Kanako Katai. A scholar is included among the top collaborators of Kanako Katai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kanako Katai. Kanako Katai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tatsumi, Sawako, Kanako Katai, Ichiro Kaneko, Hiroko Segawa, & Ken‐ichi Miyamoto. (2018). NAD metabolism and the SLC34 family: evidence for a liver-kidney axis regulating inorganic phosphate. Pflügers Archiv - European Journal of Physiology. 471(1). 109–122. 10 indexed citations
2.
Katai, Kanako, et al.. (2018). Seasonal Changes in Anthropometric, Physiological, Nutritional, and Performance Factors in Collegiate Rowers. The Journal of Strength and Conditioning Research. 34(11). 3225–3231. 3 indexed citations
3.
Katai, Kanako, et al.. (2015). Wakame (<i>Undaria pinnatifida </i>) modulates hyperphosphatemia in a rat model of chronic renal failure. The Journal of Medical Investigation. 62(1.2). 68–74. 2 indexed citations
4.
Yamamoto, Takehisa, Ken‐ichi Miyamoto, Kanako Katai, et al.. (2001). Hypophosphatemic rickets accompanying McCune?Albright syndrome: evidence that a humoral factor causes hypophosphatemia. Journal of Bone and Mineral Metabolism. 19(5-6). 287–295. 26 indexed citations
5.
Takeda, Eiji, Yutaka Taketani, Kyoko Morita, et al.. (2000). Molecular mechanisms of mammalian inorganic phosphate homeostasis. Advances in Enzyme Regulation. 40(1). 285–302. 34 indexed citations
6.
Katai, Kanako. (1999). Nicotinamide inhibits sodium-dependent phosphate cotransport activity in rat small intestine. Nephrology Dialysis Transplantation. 14(5). 1195–1201. 100 indexed citations
7.
Katai, Kanako, Ken‐ichi Miyamoto, Hiroko Segawa, et al.. (1999). Regulation of intestinal Na+-dependent phosphate co-transporters by a low-phosphate diet and 1,25-dihydroxyvitamin D3. Biochemical Journal. 343(3). 705–712. 136 indexed citations
8.
Tatsumi, Sawako, Ken‐ichi Miyamoto, Tomoko Kouda, et al.. (1998). Identification of Three Isoforms for the Na+-dependent Phosphate Cotransporter (NaPi-2) in Rat Kidney. Journal of Biological Chemistry. 273(44). 28568–28575. 23 indexed citations
9.
Tatsumi, Sawako, Hiroko Segawa, Kyoko Morita, et al.. (1998). Molecular Cloning and Hormonal Regulation of PiT-1, a Sodium-Dependent Phosphate Cotransporter from Rat Parathyroid Glands1. Endocrinology. 139(4). 1692–1699. 85 indexed citations
10.
Morita, Kyoko, Hiromi Haga, Hiroko Tanaka, et al.. (1998). Effect of dietary phosphate on Na+-dependent phosphate cotransporters function and expression in the rat kidney. Clinical and Experimental Nephrology. 2(2). 109–116. 5 indexed citations
11.
Morita, Kyoko, Kanako Katai, Hiroko Segawa, et al.. (1998). Effects of dietary Pi on the renal Na+-dependent Pi transporter NaPi-2 in thyroparathyroidectomized rats. Biochemical Journal. 333(1). 175–181. 31 indexed citations
12.
Miyamoto, Ken‐ichi, Yutaka Taketani, Kyoko Morita, et al.. (1998). Molecular and cellular regulation of renal phosphate transporters in X-linked hypophosphatemia. Clinical and Experimental Nephrology. 2(3). 178–182. 1 indexed citations
13.
Tanaka, Hiroko, Toshiyuki Shiraga, Kyoko Morita, et al.. (1997). REGULATION OF THE PEPTIDE TRANSPORTER PEPT1 IN THE RAT SMALL INTESTINE IN RESPONSE TO 5-FLUOROURACIL-INDUCED INJURY. 20(1). 82–84. 1 indexed citations
14.
Katai, Kanako, Hiroko Segawa, Hiromi Haga, et al.. (1997). Acute Regulation by Dietary Phosphate of the Sodium-Dependent Phosphate Transporter (NaP1-2) in Rat Kidney. The Journal of Biochemistry. 121(1). 50–55. 48 indexed citations
15.
Segawa, Hiroko, Ken‐ichi Miyamoto, Yoshio Ogura, et al.. (1997). Cloning, functional expression and dietary regulation of the mouse neutral and basic amino acid transporter (NBAT). Biochemical Journal. 328(2). 657–664. 14 indexed citations
16.
Takeda, Eiji, Kanako Katai, & Sawako Tatsumi. (1997). [Primary hypophosphatemic rickets].. PubMed. 656–9. 6 indexed citations
17.
Taketani, Yutaka, Ken‐ichi Miyamoto, Keiko Tanaka, et al.. (1997). Gene structure and functional analysis of the human Na+/phosphate co-transporter. Biochemical Journal. 324(3). 927–934. 39 indexed citations
18.
Miyamoto, Ken‐ichi, Hiroko Segawa, Sawako Tatsumi, et al.. (1996). Effects of Truncation of the COOH-terminal Region of a Na+-independent Neutral and Basic Amino Acid Transporter on Amino Acid Transport in Xenopus Oocytes. Journal of Biological Chemistry. 271(28). 16758–16763. 27 indexed citations
19.
Miyamoto, Ken‐ichi, Kanako Katai, Sawako Tatsumi, et al.. (1995). Mutations of the basic amino acid transporter gene associated with cystinuria. Biochemical Journal. 310(3). 951–955. 43 indexed citations
20.
MIYAMOTO, Κ., Sawako Tatsumi, Hiroyasu Yamamoto, et al.. (1995). Chromosome assignments of genes for human Na(+)-dependent phosphate co-transporters NaPi-3 and NPT-1.. PubMed. 42(1-2). 5–9. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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