Junyan Shu

809 total citations
10 papers, 731 citations indexed

About

Junyan Shu is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Junyan Shu has authored 10 papers receiving a total of 731 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Oncology and 4 papers in Immunology. Recurrent topics in Junyan Shu's work include NF-κB Signaling Pathways (2 papers), Cell death mechanisms and regulation (2 papers) and Immune Response and Inflammation (2 papers). Junyan Shu is often cited by papers focused on NF-κB Signaling Pathways (2 papers), Cell death mechanisms and regulation (2 papers) and Immune Response and Inflammation (2 papers). Junyan Shu collaborates with scholars based in United States, India and Bulgaria. Junyan Shu's co-authors include Genhong Cheng, Hajir Dadgostar, Ho H. Lee, Qingwen Cheng, Dennis W. Stacey, John R. Stevenson, Xiaolan Zhou, Masahiro Hitomi, Guan Chen and John H. Kehrl and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Molecular and Cellular Biology.

In The Last Decade

Junyan Shu

10 papers receiving 721 citations

Peers

Junyan Shu

MB

IMMUN

CR

ONCOL

EPIDE

J X Lin United States

Elmar Wegener Germany

Ernestina Saulle Italy

Antonio Ruiz‐Vela Spain

Gaspard Cretenet Netherlands

Laurent Susini France

Maria Buxadé Spain

David Allanic France

Fumiko Kihara‐Negishi Japan

Chika Sakai Japan

J X Lin United States

Junyan Shu

263 ×0.6

MB

328 ×1.0

IMMUN

123 ×0.5

CR

174 ×1.0

ONCOL

43 ×0.8

EPIDE

Citations per year, relative to Junyan Shu Junyan Shu (= 1×) peers J X Lin

Countries citing papers authored by Junyan Shu

Since Specialization

Citations

This map shows the geographic impact of Junyan Shu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Junyan Shu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Junyan Shu more than expected).

Fields of papers citing papers by Junyan Shu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Junyan Shu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Junyan Shu. The network helps show where Junyan Shu may publish in the future.

Co-authorship network of co-authors of Junyan Shu

This figure shows the co-authorship network connecting the top 25 collaborators of Junyan Shu. A scholar is included among the top collaborators of Junyan Shu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Junyan Shu. Junyan Shu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Kuhns, Scott, et al.. (2020). Differential influence on antibody dependent cellular phagocytosis by different glycoforms on therapeutic Monoclonal antibodies. Journal of Biotechnology. 317. 5–15. 12 indexed citations

2.

Shu, Junyan, et al.. (1999). Adenovirus-mediated Gene Transfer of Dominant Negative Ha-Ras Inhibits Proliferation of Primary Meningioma Cells. Neurosurgery. 44(3). 579–587. 28 indexed citations

3.

Chin, Arnold I., et al.. (1999). TANK Potentiates Tumor Necrosis Factor Receptor-Associated Factor-Mediated c-Jun N-Terminal Kinase/Stress-Activated Protein Kinase Activation through the Germinal Center Kinase Pathway. Molecular and Cellular Biology. 19(10). 6665–6672. 37 indexed citations

4.

Lee, Ho H., Hajir Dadgostar, Qingwen Cheng, Junyan Shu, & Genhong Cheng. (1999). NF-κB-mediated up-regulation of Bcl-x and Bfl-1/A1 is required for CD40 survival signaling in B lymphocytes. Proceedings of the National Academy of Sciences. 96(16). 9136–9141. 463 indexed citations

5.

Hitomi, Masahiro, et al.. (1998). p21Waf1 inhibits the activity of cyclin dependent Kinase 2 by preventing its activating phosphorylation. Oncogene. 17(8). 959–969. 26 indexed citations

6.

Chen, Guan, Junyan Shu, & Dennis W. Stacey. (1997). Oncogenic transformation potentiates apoptosis, S-phase arrest and stress-kinase activation by etoposide. Oncogene. 15(14). 1643–1651. 55 indexed citations

7.

Hitomi, Masahiro, Junyan Shu, David K. Strom, et al.. (1996). Prostaglandin A2 Blocks the Activation of G1 Phase Cyclin-dependent Kinase without Altering Mitogen-activated Protein Kinase Stimulation. Journal of Biological Chemistry. 271(16). 9376–9383. 27 indexed citations

8.

Shu, Junyan, Masahiro Hitomi, & Dennis W. Stacey. (1996). Activation of JNK/SAPK pathway is not directly inhibitory for cell cycle progression in NIH3T3 cells.. PubMed. 13(11). 2421–30. 24 indexed citations

9.

Shu, Junyan, Stephen H. Benedict, & Marcia A. Chan. (1994). Anti-IgM and Anti-IgG Modulate c-fos RNA Levels Differently in Human B Lymphocytes. Cellular Immunology. 159(2). 170–183. 1 indexed citations

10.

Shu, Junyan, John R. Stevenson, & Xiaolan Zhou. (1993). Modulation of cellular immune responses by cold water swim stress in the rat. Developmental & Comparative Immunology. 17(4). 357–371. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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