Jun‐Jun Kang

641 total citations
22 papers, 392 citations indexed

About

Jun‐Jun Kang is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Endocrine and Autonomic Systems. According to data from OpenAlex, Jun‐Jun Kang has authored 22 papers receiving a total of 392 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 7 papers in Endocrine and Autonomic Systems. Recurrent topics in Jun‐Jun Kang's work include Neuroscience of respiration and sleep (7 papers), Neuroscience and Neuropharmacology Research (6 papers) and Neonatal and fetal brain pathology (5 papers). Jun‐Jun Kang is often cited by papers focused on Neuroscience of respiration and sleep (7 papers), Neuroscience and Neuropharmacology Research (6 papers) and Neonatal and fetal brain pathology (5 papers). Jun‐Jun Kang collaborates with scholars based in China, United States and Hong Kong. Jun‐Jun Kang's co-authors include Yingying Liu, Jihua Chen, Ling Zhang, Shengxi Wu, Xiaoyang Liu, Yazhou Wang, Kun Zhang, Caiyong Yu, Huan Yu and Dingding Yang and has published in prestigious journals such as Neuron, Cell Metabolism and Brain Research.

In The Last Decade

Jun‐Jun Kang

22 papers receiving 389 citations

Peers

Jun‐Jun Kang
Jun‐Jun Kang
Citations per year, relative to Jun‐Jun Kang Jun‐Jun Kang (= 1×) peers Akie Honmura

Countries citing papers authored by Jun‐Jun Kang

Since Specialization
Citations

This map shows the geographic impact of Jun‐Jun Kang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jun‐Jun Kang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jun‐Jun Kang more than expected).

Fields of papers citing papers by Jun‐Jun Kang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jun‐Jun Kang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jun‐Jun Kang. The network helps show where Jun‐Jun Kang may publish in the future.

Co-authorship network of co-authors of Jun‐Jun Kang

This figure shows the co-authorship network connecting the top 25 collaborators of Jun‐Jun Kang. A scholar is included among the top collaborators of Jun‐Jun Kang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jun‐Jun Kang. Jun‐Jun Kang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xian, Panpan, Mengmeng Wang, Rou‐Gang Xie, et al.. (2025). Mitochondrial dysfunction reveals H2S-mediated synaptic sulfhydration as a potential mechanism for autism-associated social defects. Cell Metabolism. 37(10). 2076–2092.e8. 1 indexed citations
2.
Guo, Baolin, Honghui Mao, Keke Ren, et al.. (2023). CB1R dysfunction of inhibitory synapses in the ACC drives chronic social isolation stress-induced social impairments in male mice. Neuron. 112(3). 441–457.e6. 20 indexed citations
3.
Kang, Jun‐Jun, Shou Jing Yang, Baolin Guo, et al.. (2023). Alterations in synapses and mitochondria induced by acute or chronic intermittent hypoxia in the pre-Bötzinger complex of rats: an ultrastructural triple-labeling study with immunocytochemistry and histochemistry. Frontiers in Cellular Neuroscience. 17. 1132241–1132241. 1 indexed citations
4.
Yan, Jianfei, Minjuan Shen, Bing‐Dong Sui, et al.. (2022). Autophagic LC3 + calcified extracellular vesicles initiate cartilage calcification in osteoarthritis. Science Advances. 8(19). eabn1556–eabn1556. 54 indexed citations
5.
Liang, Lirong, Ze Fan, Youyi Zhao, et al.. (2022). Sevoflurane-Induced Neurotoxicity in the Developing Hippocampus via HIPK2/AKT/mTOR Signaling. Neurotoxicity Research. 40(3). 803–813. 8 indexed citations
6.
Xi, Zihan, Huan Yu, Kun Zhang, et al.. (2021). Mitochondria transplantation protects traumatic brain injury via promoting neuronal survival and astrocytic BDNF. Translational research. 235. 102–114. 64 indexed citations
7.
Liu, Rongrong, Ziyu Liu, Haifeng Hu, et al.. (2021). HTNV infection of CD8+ T cells is associated with disease progression in HFRS patients. Communications Biology. 4(1). 652–652. 13 indexed citations
8.
Chen, Zhe, Hai‐Bin Tang, Jun‐Jun Kang, et al.. (2021). Necroptotic astrocytes induced neuronal apoptosis partially through EVs-derived pro-BDNF. Brain Research Bulletin. 177. 73–80. 17 indexed citations
9.
Song, Changgeng, Fang Yang, Jiajia Zhang, et al.. (2021). Endocannabinoid system in the neurodevelopment of GABAergic interneurons: implications for neurological and psychiatric disorders. Reviews in the Neurosciences. 32(8). 803–831. 6 indexed citations
10.
Fang, Zongping, Di Wu, Jiao Deng, et al.. (2021). An MD2-perturbing peptide has therapeutic effects in rodent and rhesus monkey models of stroke. Science Translational Medicine. 13(597). 36 indexed citations
11.
Xian, Hang, Jun‐Jun Kang, Yingying Liu, et al.. (2020). CCL2 facilitates spinal synaptic transmission and pain via interaction with presynaptic CCR2 in spinal nociceptor terminals. Molecular Brain. 13(1). 161–161. 25 indexed citations
12.
Kang, Jun‐Jun, Xiaoyan Wei, & Yingying Liu. (2020). [Chronic intermittent hypoxia induces expression of phospho-PKC substrates in rat pre-Bötzinger complex].. PubMed. 72(5). 559–565. 1 indexed citations
13.
Wang, Mengmeng, Xinyan Liu, Yilin Hou, et al.. (2019). Decrease of GSK-3β Activity in the Anterior Cingulate Cortex of Shank3b−/− Mice Contributes to Synaptic and Social Deficiency. Frontiers in Cellular Neuroscience. 13. 447–447. 18 indexed citations
14.
Kang, Jun‐Jun, Baolin Guo, Chun Sing Lam, et al.. (2018). Daily acute intermittent hypoxia induced dynamic changes in dendritic mitochondrial ultrastructure and cytochrome oxidase activity in the pre-Bötzinger complex of rats. Experimental Neurology. 313. 124–134. 9 indexed citations
15.
Kang, Jun‐Jun, Chun Sing Lam, Xiaofeng Huang, et al.. (2016). Catecholaminergic neurons in synaptic connections with pre-Bötzinger complex neurons in the rostral ventrolateral medulla in normoxic and daily acute intermittent hypoxic rats. Experimental Neurology. 287(Pt 2). 165–175. 18 indexed citations
16.
Meng, Xiandong, Dong Wei, Juan Li, et al.. (2014). Astrocytic expression of cannabinoid type 1 receptor in rat and human sclerotic hippocampi.. PubMed. 7(6). 2825–37. 14 indexed citations
17.
Heng, Lijun, Jie Ma, Lijuan Qu, et al.. (2013). Increased expression of cannabinoid receptor 1 in the nucleus accumbens core in a rat model with morphine withdrawal. Brain Research. 1531. 102–112. 13 indexed citations
18.
Liu, Ning, Fang Li, Yujiang Chen, et al.. (2013). The inhibitory effect of a polymerisable cationic monomer on functional matrix metalloproteinases. Journal of Dentistry. 41(11). 1101–1108. 12 indexed citations
19.
Kang, Jun‐Jun, Xiaoyan Wei, Jinping Liu, et al.. (2013). Expression of phospho‐Ca2+/calmodulin‐dependent protein kinase II in the pre‐Bötzinger complex of rats. Journal of Neurochemistry. 126(3). 349–359. 2 indexed citations
20.
Chae, Han–Jung, et al.. (1999). Mechanism of mitogenic effect of fluoride on fetal rat osteoblastic cells: evidence for Shc, Grb2 and P-CREB-dependent pathways.. PubMed. 105(3). 185–99. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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