Jun‐ichi Hanai

8.4k total citations · 3 hit papers
40 papers, 6.9k citations indexed

About

Jun‐ichi Hanai is a scholar working on Molecular Biology, Cancer Research and Nephrology. According to data from OpenAlex, Jun‐ichi Hanai has authored 40 papers receiving a total of 6.9k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 8 papers in Cancer Research and 5 papers in Nephrology. Recurrent topics in Jun‐ichi Hanai's work include TGF-β signaling in diseases (10 papers), Angiogenesis and VEGF in Cancer (5 papers) and Cancer-related gene regulation (4 papers). Jun‐ichi Hanai is often cited by papers focused on TGF-β signaling in diseases (10 papers), Angiogenesis and VEGF in Cancer (5 papers) and Cancer-related gene regulation (4 papers). Jun‐ichi Hanai collaborates with scholars based in United States, Japan and Canada. Jun‐ichi Hanai's co-authors include Vikas P. Sukhatme, Tadanori Mammoto, Kohei Miyazono, Masahiro Kawabata, Ayako Nishihara, David M. Charytan, Frank Strutz, Raghu Kalluri, Michael Zeisberg and Hikaru Sugimoto and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Jun‐ichi Hanai

40 papers receiving 6.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Soluble endoglin contributes to the pathogenesis of preeclampsia

2006 1.4k citations Shivalingappa Venkatesha, Jun‐ichi Hanai et al. Nature Medicine profile →
BMP-7 counteracts TGF-β1–induced epithelial-to-mesenchymal transition and reverses chronic renal injury

2003 1.1k citations Michael Zeisberg, Jun‐ichi Hanai et al. Nature Medicine profile →
Smad6 inhibits signalling by the TGF-β superfamily

1997 869 citations Takeshi Imamura, Ayako Nishihara et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jun‐ichi Hanai United States	30	4.0k	1.2k	1.1k	1.0k	901	40	6.9k
N. Ferrara United States	20	2.5k 0.6×	420 0.3×	746 0.7×	742 0.7×	810 0.9×	21	5.1k
Hans J. Baelde Netherlands	41	1.8k 0.5×	590 0.5×	498 0.5×	523 0.5×	1.1k 1.2×	160	5.8k
Hans-Peter Gerber United States	13	4.7k 1.2×	454 0.4×	1.9k 1.8×	1.3k 1.3×	1.5k 1.7×	16	8.9k
Sharon D. Ricardo Australia	40	2.5k 0.6×	304 0.2×	406 0.4×	295 0.3×	917 1.0×	109	5.5k
Yeon S. Ahn United States	44	2.8k 0.7×	367 0.3×	702 0.7×	553 0.5×	1.6k 1.8×	131	6.9k
Nam Keun Kim South Korea	36	2.6k 0.6×	560 0.5×	1.5k 1.4×	508 0.5×	887 1.0×	305	5.6k
Bin Yu China	33	2.6k 0.6×	604 0.5×	1.4k 1.3×	277 0.3×	671 0.7×	176	4.9k
Bertrand Knebelmann France	46	3.0k 0.8×	162 0.1×	831 0.8×	519 0.5×	593 0.7×	123	6.2k
Vera Eremina Canada	23	2.3k 0.6×	422 0.3×	201 0.2×	504 0.5×	631 0.7×	25	4.9k
Pamela F. Jones United Kingdom	22	7.2k 1.8×	423 0.3×	2.1k 2.0×	1.6k 1.5×	734 0.8×	45	9.5k

Countries citing papers authored by Jun‐ichi Hanai

Since Specialization

Citations

This map shows the geographic impact of Jun‐ichi Hanai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jun‐ichi Hanai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jun‐ichi Hanai more than expected).

Fields of papers citing papers by Jun‐ichi Hanai

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jun‐ichi Hanai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jun‐ichi Hanai. The network helps show where Jun‐ichi Hanai may publish in the future.

Co-authorship network of co-authors of Jun‐ichi Hanai

This figure shows the co-authorship network connecting the top 25 collaborators of Jun‐ichi Hanai. A scholar is included among the top collaborators of Jun‐ichi Hanai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jun‐ichi Hanai. Jun‐ichi Hanai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ren, Jian-Guo, Pankaj Seth, Huihui Ye, et al.. (2017). Citrate Suppresses Tumor Growth in Multiple Models through Inhibition of Glycolysis, the Tricarboxylic Acid Cycle and the IGF-1R Pathway. Scientific Reports. 7(1). 4537–4537. 111 indexed citations

Fan, Yi, Jun‐ichi Hanai, Phuong Le, et al.. (2017). Parathyroid Hormone Directs Bone Marrow Mesenchymal Cell Fate. Cell Metabolism. 25(3). 661–672. 305 indexed citations

Jernigan, Finith E., Jun‐ichi Hanai, Vikas P. Sukhatme, & Lijun Sun. (2017). Discovery of furan carboxylate derivatives as novel inhibitors of ATP-citrate lyase via virtual high-throughput screening. Bioorganic & Medicinal Chemistry Letters. 27(4). 929–935. 19 indexed citations

Ide, Noriko, Hannes Olauson, Tadatoshi Sato, et al.. (2016). In vivo evidence for a limited role of proximal tubular Klotho in renal phosphate handling. Kidney International. 90(2). 348–362. 57 indexed citations

Koerner, Steffi, Jun‐ichi Hanai, Sha Bai, et al.. (2016). Design and synthesis of emodin derivatives as novel inhibitors of ATP-citrate lyase. European Journal of Medicinal Chemistry. 126. 920–928. 71 indexed citations

Xie, Han, Jun‐ichi Hanai, Jian-Guo Ren, et al.. (2014). Targeting Lactate Dehydrogenase-A Inhibits Tumorigenesis and Tumor Progression in Mouse Models of Lung Cancer and Impacts Tumor-Initiating Cells. Cell Metabolism. 19(5). 795–809. 374 indexed citations

Kishi, Shuji, Peter Bayliss, & Jun‐ichi Hanai. (2014). A prospective epigenetic paradigm between cellular senescence and epithelial-mesenchymal transition in organismal development and aging. Translational research. 165(1). 241–249. 15 indexed citations

Koshimizu, Eriko, Shintaro Imamura, Jie Qi, et al.. (2011). Embryonic Senescence and Laminopathies in a Progeroid Zebrafish Model. PLoS ONE. 6(3). e17688–e17688. 43 indexed citations

Hanai, Jun‐ichi, Atsuo T. Sasaki, Susumu Kobayashi, et al.. (2011). Inhibition of lung cancer growth: ATP citrate lyase knockdown and statin treatment leads to dual blockade of mitogen‐activated protein Kinase (MAPK) and Phosphatidylinositol‐3‐kinase (PI3K)/AKT pathways. Journal of Cellular Physiology. 227(4). 1709–1720. 134 indexed citations

10.

Seth, Pankaj, Yanfeng Lin, Jun‐ichi Hanai, et al.. (2005). Magic roundabout, a tumor endothelial marker: Expression and signaling. Biochemical and Biophysical Research Communications. 332(2). 533–541. 90 indexed citations

11.

Hanai, Jun‐ichi, Tadanori Mammoto, Pankaj Seth, et al.. (2005). Lipocalin 2 Diminishes Invasiveness and Metastasis of Ras-transformed Cells. Journal of Biological Chemistry. 280(14). 13641–13647. 103 indexed citations

12.

Zeisberg, Michael, Jun‐ichi Hanai, Hikaru Sugimoto, et al.. (2003). BMP-7 counteracts TGF-β1–induced epithelial-to-mesenchymal transition and reverses chronic renal injury. Nature Medicine. 9(7). 964–968. 1126 indexed citations breakdown →

13.

Karumanchi, S. Ananth, et al.. (2002). Cellular Actions and Signaling by Endostatin. Critical Reviews in Eukaryotic Gene Expression. 12(3). 175–192. 17 indexed citations

14.

Hanai, Jun‐ichi, S. Ananth Karumanchi, Chris Albanese, et al.. (2002). Endostatin Causes G1 Arrest of Endothelial Cells through Inhibition of Cyclin D1. Journal of Biological Chemistry. 277(19). 16464–16469. 182 indexed citations

15.

Hanai, Jun‐ichi, Keiichiro Tada, Nicole C. Grieder, et al.. (2000). Schnurri interacts with Mad in a Dpp‐dependent manner. Genes to Cells. 5(5). 359–369. 29 indexed citations

16.

Nishihara, Ayako, Jun‐ichi Hanai, Takeshi Imamura, Kohei Miyazono, & Masahiro Kawabata. (1999). E1A Inhibits Transforming Growth Factor-β Signaling through Binding to Smad Proteins. Journal of Biological Chemistry. 274(40). 28716–28723. 91 indexed citations

17.

Inoue, Hirofumi, et al.. (1999). c-Ski Acts as a Transcriptional Co-repressor in Transforming Growth Factor-β Signaling through Interaction with Smads. Journal of Biological Chemistry. 274(49). 35269–35277. 318 indexed citations

18.

Takaichi, Kenmei, et al.. (1999). Expression of transfected human Na+/H+ exchanger (NHE-1) in the basolateral membrane of opossum kidney cells. Journal of Cellular Physiology. 178(1). 44–50. 2 indexed citations

19.

Nishihara, Ayako, Jun‐ichi Hanai, Nobuaki Okamoto, et al.. (1998). Role of p300, a transcriptional coactivator, in signalling of TGF‐β. Genes to Cells. 3(9). 613–623. 137 indexed citations

20.

Tanemoto, Masayuki, et al.. (1995). Myeloperoxidase-antineutrophil cytoplasmic antibody-positive crescentic glomerulonephritis complicating the course of Graves' disease: Report of three adult cases. American Journal of Kidney Diseases. 26(5). 774–780. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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