Jun‐ichi Akagi

1.2k total citations
38 papers, 992 citations indexed

About

Jun‐ichi Akagi is a scholar working on Molecular Biology, Cancer Research and Surgery. According to data from OpenAlex, Jun‐ichi Akagi has authored 38 papers receiving a total of 992 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 15 papers in Cancer Research and 7 papers in Surgery. Recurrent topics in Jun‐ichi Akagi's work include Carcinogens and Genotoxicity Assessment (15 papers), DNA Repair Mechanisms (14 papers) and Genomics, phytochemicals, and oxidative stress (6 papers). Jun‐ichi Akagi is often cited by papers focused on Carcinogens and Genotoxicity Assessment (15 papers), DNA Repair Mechanisms (14 papers) and Genomics, phytochemicals, and oxidative stress (6 papers). Jun‐ichi Akagi collaborates with scholars based in Japan, Sri Lanka and Netherlands. Jun‐ichi Akagi's co-authors include Fumio Hanaoka, Takeshi Toyoda, Young‐Man Cho, Kumiko Ogawa, Yasuko Mizuta, Haruo Ohmori, Eiji Ohashi, Chikahide Masutani, Shigenori Iwai and Yoshiki Murakumo and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Cell Biology and Molecular Cell.

In The Last Decade

Jun‐ichi Akagi

37 papers receiving 978 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jun‐ichi Akagi Japan 14 602 240 171 87 76 38 992
Han-Oh Park South Korea 19 1.2k 2.0× 147 0.6× 178 1.0× 159 1.8× 60 0.8× 42 1.7k
Yuqi Guo China 24 589 1.0× 181 0.8× 130 0.8× 85 1.0× 120 1.6× 51 1.2k
Sanjay Gupta India 18 556 0.9× 115 0.5× 99 0.6× 38 0.4× 114 1.5× 87 1.1k
Dong-Hyun Lee South Korea 17 718 1.2× 160 0.7× 58 0.3× 95 1.1× 128 1.7× 47 1.1k
Yili Liu China 21 642 1.1× 286 1.2× 90 0.5× 59 0.7× 39 0.5× 48 961
Xin Bing China 16 254 0.4× 95 0.4× 114 0.7× 35 0.4× 78 1.0× 34 724
Liye Zhu China 20 657 1.1× 206 0.9× 107 0.6× 339 3.9× 32 0.4× 38 1.1k
Yujing He China 20 575 1.0× 199 0.8× 170 1.0× 217 2.5× 132 1.7× 60 1.5k
Xiangyang Bai China 15 327 0.5× 133 0.6× 144 0.8× 35 0.4× 217 2.9× 29 920
Zhipeng Zhang China 15 500 0.8× 210 0.9× 164 1.0× 20 0.2× 36 0.5× 29 750

Countries citing papers authored by Jun‐ichi Akagi

Since Specialization
Citations

This map shows the geographic impact of Jun‐ichi Akagi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jun‐ichi Akagi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jun‐ichi Akagi more than expected).

Fields of papers citing papers by Jun‐ichi Akagi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jun‐ichi Akagi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jun‐ichi Akagi. The network helps show where Jun‐ichi Akagi may publish in the future.

Co-authorship network of co-authors of Jun‐ichi Akagi

This figure shows the co-authorship network connecting the top 25 collaborators of Jun‐ichi Akagi. A scholar is included among the top collaborators of Jun‐ichi Akagi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jun‐ichi Akagi. Jun‐ichi Akagi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Akagi, Jun‐ichi, Masayuki Yokoi, Tsuyoshi Shirai, et al.. (2023). A formamidopyrimidine derivative from the deoxyguanosine adduct produced by food contaminant acrylamide induces DNA replication block and mutagenesis. Journal of Biological Chemistry. 299(8). 105002–105002. 3 indexed citations
2.
Akagi, Jun‐ichi, Young‐Man Cho, Takeshi Toyoda, Yasuko Mizuta, & Kumiko Ogawa. (2023). EpCAM and APN expression in combination with γ‐H2AX as biomarkers for detecting hepatocarcinogens in rats. Cancer Science. 114(12). 4763–4769. 1 indexed citations
3.
Toyoda, Takeshi, Kohei Matsushita, Hirotoshi Akane, et al.. (2023). Early detection of hepatocarcinogens in rats by immunohistochemistry of γ-H2AX. The Journal of Toxicological Sciences. 48(6). 323–332. 1 indexed citations
4.
Akagi, Jun‐ichi, Yasuko Mizuta, Hirotoshi Akane, Takeshi Toyoda, & Kumiko Ogawa. (2023). Oral toxicological study of titanium dioxide nanoparticles with a crystallite diameter of 6 nm in rats. Particle and Fibre Toxicology. 20(1). 23–23. 15 indexed citations
5.
Yamada, Takanori, Takeshi Toyoda, Kohei Matsushita, et al.. (2020). Expression of stem cell markers as useful complementary factors in the early detection of urinary bladder carcinogens by immunohistochemistry for γ-H2AX. Archives of Toxicology. 95(2). 715–726. 7 indexed citations
6.
Sakai, Wataru, Masayuki Kusakabe, Takeshi Matsui, et al.. (2020). Functional impacts of the ubiquitin–proteasome system on DNA damage recognition in global genome nucleotide excision repair. Scientific Reports. 10(1). 19704–19704. 18 indexed citations
7.
Mizuta, Yasuko, Jun‐ichi Akagi, Naoki Sugimoto, et al.. (2020). A 90-day repeated oral dose toxicity study of four stereoisomers of 2,4-dimethyl-4-phenyltetrahydrofuran, a synthetic flavoring substance, in F344 rats. Regulatory Toxicology and Pharmacology. 114. 104664–104664. 3 indexed citations
8.
Akagi, Jun‐ichi, et al.. (2018). Subchronic toxicity evaluation of isoeugenyl methyl ether in F344/DuCrj rats by 13-week oral administration. Regulatory Toxicology and Pharmacology. 102. 34–39. 4 indexed citations
9.
Akagi, Jun‐ichi, Young‐Man Cho, Yasuko Mizuta, Takeshi Toyoda, & Kumiko Ogawa. (2018). Subchronic toxicity evaluation of 5-hexenyl isothiocyanate, a nature identical flavoring substance from Wasabia japonica, in F344/DuCrj rats. Food and Chemical Toxicology. 122. 80–86. 6 indexed citations
10.
Hirata, Tadashi, Young‐Man Cho, Isamu Suzuki, et al.. (2017). 4-Methylthio-3-butenyl isothiocyanate mediates nuclear factor (erythroid-derived 2)-like 2 activation by regulating reactive oxygen species production in human esophageal epithelial cells. Food and Chemical Toxicology. 111. 295–301. 5 indexed citations
12.
Cho, Young‐Man, Takeshi Toyoda, Jun‐ichi Akagi, et al.. (2015). Orally administered glycidol and its fatty acid esters as well as 3-MCPD fatty acid esters are metabolized to 3-MCPD in the F344 rat. Regulatory Toxicology and Pharmacology. 73(3). 726–731. 30 indexed citations
13.
Toyoda, Takeshi, Young‐Man Cho, Jun‐ichi Akagi, et al.. (2015). Early Detection of Genotoxic Urinary Bladder Carcinogens by Immunohistochemistry for γ-H2AX. Toxicological Sciences. 148(2). 400–408. 30 indexed citations
14.
Toyoda, Takeshi, Young‐Man Cho, Yasuko Mizuta, Jun‐ichi Akagi, & Kumiko Ogawa. (2014). A 13-week subchronic toxicity study of ferric citrate in F344 rats. Food and Chemical Toxicology. 74. 68–75. 13 indexed citations
15.
Hashimoto, Keiji, Jun‐ichi Akagi, Eiji Ohashi, et al.. (2012). The Vital Role of Polymerase zeta and REV1 in Mutagenic, but Not Correct, DNA Synthesis across Benzo[a]pyrene-dG and Recruitment of Polymerase zeta by REV1 to Replication-stalled Site. Data Archiving and Networked Services (DANS).
16.
Yokoi, Masayuki, et al.. (2012). Stalled Polη at its cognate substrate initiates an alternative translesion synthesis pathway via interaction with REV1. Genes to Cells. 17(2). 98–108. 13 indexed citations
17.
Hashimoto, Keiji, Youngjin Cho, In‐Young Yang, et al.. (2012). The Vital Role of Polymerase ζ and REV1 in Mutagenic, but Not Correct, DNA Synthesis across Benzo[a]pyrene-dG and Recruitment of Polymerase ζ by REV1 to Replication-stalled Site. Journal of Biological Chemistry. 287(12). 9613–9622. 42 indexed citations
18.
Akagi, Jun‐ichi, Chikahide Masutani, Yuki Kataoka, et al.. (2009). Interaction with DNA polymerase η is required for nuclear accumulation of REV1 and suppression of spontaneous mutations in human cells. DNA repair. 8(5). 585–599. 50 indexed citations
19.
Sugasawa, Kaoru, Jun‐ichi Akagi, Ryotaro Nishi, Shigenori Iwai, & Fumio Hanaoka. (2009). Two-Step Recognition of DNA Damage for Mammalian Nucleotide Excision Repair: Directional Binding of the XPC Complex and DNA Strand Scanning. Molecular Cell. 36(4). 642–653. 153 indexed citations
20.
Ohashi, Eiji, Yoshiki Murakumo, Jun‐ichi Akagi, et al.. (2004). Interaction of hREV1 with three human Y‐family DNA polymerases. Genes to Cells. 9(6). 523–531. 222 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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