Julia Steinbacher

506 total citations
6 papers, 105 citations indexed

About

Julia Steinbacher is a scholar working on Oncology, Immunology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Julia Steinbacher has authored 6 papers receiving a total of 105 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Oncology, 5 papers in Immunology and 3 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Julia Steinbacher's work include Immune Cell Function and Interaction (5 papers), CAR-T cell therapy research (5 papers) and Monoclonal and Polyclonal Antibodies Research (3 papers). Julia Steinbacher is often cited by papers focused on Immune Cell Function and Interaction (5 papers), CAR-T cell therapy research (5 papers) and Monoclonal and Polyclonal Antibodies Research (3 papers). Julia Steinbacher collaborates with scholars based in Germany, Switzerland and United Kingdom. Julia Steinbacher's co-authors include Helmut R. Salih, Ludger Große‐Hovest, Benjamin Joachim Schmiedel, Gundram Jung, Alexander Steinle, Tina Nuebling, Philaretos C. Kousis, Stefanie Raab, Maya C. André and Miyuki Azuma and has published in prestigious journals such as Blood, The Journal of Immunology and International Journal of Cancer.

In The Last Decade

Julia Steinbacher

6 papers receiving 105 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Julia Steinbacher Germany	4	85	81	18	15	14	6	105
Bettina Brauchle Germany	4	71 0.8×	62 0.8×	30 1.7×	25 1.7×	29 2.1×	12	107
Anetta Marcinek Germany	4	74 0.9×	74 0.9×	29 1.6×	28 1.9×	36 2.6×	16	122
Robert Godal United States	2	58 0.7×	104 1.3×	5 0.3×	17 1.1×	28 2.0×	3	115
Véronique Vergé France	6	40 0.5×	81 1.0×	6 0.3×	47 3.1×	21 1.5×	20	125
Guillaume Habif France	5	125 1.5×	164 2.0×	11 0.6×	34 2.3×	8 0.6×	6	199
Gerulf Hänel Germany	5	39 0.5×	53 0.7×	17 0.9×	21 1.4×	20 1.4×	15	92
Kaitlyn A. Lagattuta United States	4	39 0.5×	96 1.2×	19 1.1×	40 2.7×	7 0.5×	7	143
Seth J. Welsh United States	4	59 0.7×	52 0.6×	42 2.3×	42 2.8×	63 4.5×	10	135
Miguel Caballero‐Baños Spain	5	60 0.7×	72 0.9×	3 0.2×	23 1.5×	3 0.2×	9	100
Daniel J. Hunt United States	3	36 0.4×	89 1.1×	5 0.3×	8 0.5×	47 3.4×	7	125

Countries citing papers authored by Julia Steinbacher

Since Specialization

Citations

This map shows the geographic impact of Julia Steinbacher's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Julia Steinbacher with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Julia Steinbacher more than expected).

Fields of papers citing papers by Julia Steinbacher

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Julia Steinbacher. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Julia Steinbacher. The network helps show where Julia Steinbacher may publish in the future.

Co-authorship network of co-authors of Julia Steinbacher

This figure shows the co-authorship network connecting the top 25 collaborators of Julia Steinbacher. A scholar is included among the top collaborators of Julia Steinbacher based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Julia Steinbacher. Julia Steinbacher is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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6 of 6 papers shown

Paczulla, Anna, Kathrin Rothfelder, Simon Raffel, et al.. (2018). Absence of NKG2D Ligands Defines Human Acute Myeloid Leukaemia Stem Cells and Mediates Their Immune Evasion. Blood. 132(Supplement 1). 769–769. 2 indexed citations

Raab, Stefanie, Julia Steinbacher, Benjamin Joachim Schmiedel, et al.. (2014). Fc-Optimized NKG2D–Fc Constructs Induce NK Cell Antibody-Dependent Cellular Cytotoxicity against Breast Cancer Cells Independently of HER2/neu Expression Status. The Journal of Immunology. 193(8). 4261–4272. 30 indexed citations

Steinbacher, Julia, Benjamin Joachim Schmiedel, Alexander Steinle, et al.. (2014). An Fc‐optimized NKG2D‐immunoglobulin G fusion protein for induction of natural killer cell reactivity against leukemia. International Journal of Cancer. 136(5). 1073–1084. 27 indexed citations

Schmiedel, Benjamin Joachim, Julia Steinbacher, Tina Nuebling, et al.. (2013). Generation and Preclinical Characterization of a Fc-optimized GITR-Ig Fusion Protein for Induction of NK Cell Reactivity Against Leukemia. Molecular Therapy. 21(4). 877–886. 16 indexed citations

Schmiedel, Benjamin Joachim, Tina Nuebling, Julia Steinbacher, et al.. (2012). Receptor Activator for NF-κB Ligand in Acute Myeloid Leukemia: Expression, Function, and Modulation of NK Cell Immunosurveillance. The Journal of Immunology. 190(2). 821–831. 29 indexed citations

Steinbacher, Julia, Benjamin Joachim Schmiedel, Tina Nuebling, et al.. (2012). Bimodal Induction of NK Cell Reactivity Against Acute Myeloid (AML) and Chronic Lymphoid Leukemia (CLL) by Fc-Engineered GITR-Fc Fusion Proteins.. Blood. 120(21). 2143–2143. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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