Julia S. Brunner

2.0k total citations · 1 hit paper
36 papers, 972 citations indexed

About

Julia S. Brunner is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, Julia S. Brunner has authored 36 papers receiving a total of 972 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 13 papers in Immunology and 7 papers in Cancer Research. Recurrent topics in Julia S. Brunner's work include Amino Acid Enzymes and Metabolism (4 papers), Cancer, Hypoxia, and Metabolism (4 papers) and Mast cells and histamine (4 papers). Julia S. Brunner is often cited by papers focused on Amino Acid Enzymes and Metabolism (4 papers), Cancer, Hypoxia, and Metabolism (4 papers) and Mast cells and histamine (4 papers). Julia S. Brunner collaborates with scholars based in Austria, United States and Germany. Julia S. Brunner's co-authors include Lydia W.S. Finley, Gernot Schabbauer, Michael Bramberger, Bernhard Rinner, Helmut Schwabach, Omar Sharif, Andrea Vogel, Emine Sahin, Victoria Saferding and Stephan Blüml and has published in prestigious journals such as Nature, Molecular Cell and The Journal of Immunology.

In The Last Decade

Julia S. Brunner

32 papers receiving 955 citations

Hit Papers

A non-canonical tricarboxylic acid cycle underlies cellul... 2022 2026 2023 2024 2022 50 100 150

Peers

Julia S. Brunner
Kai Song China
Lipei Liu China
Ying Fan China
Mohamed Abouelhoda Saudi Arabia
Julia S. Brunner
Citations per year, relative to Julia S. Brunner Julia S. Brunner (= 1×) peers Yifang Chen

Countries citing papers authored by Julia S. Brunner

Since Specialization
Citations

This map shows the geographic impact of Julia S. Brunner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Julia S. Brunner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Julia S. Brunner more than expected).

Fields of papers citing papers by Julia S. Brunner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Julia S. Brunner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Julia S. Brunner. The network helps show where Julia S. Brunner may publish in the future.

Co-authorship network of co-authors of Julia S. Brunner

This figure shows the co-authorship network connecting the top 25 collaborators of Julia S. Brunner. A scholar is included among the top collaborators of Julia S. Brunner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Julia S. Brunner. Julia S. Brunner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Brunner, Julia S., et al.. (2026). Aspartate availability drives differential engagement of the malate-aspartate shuttle. Molecular Cell. 86(5). 954–967.e7.
2.
Yashinskie, Jossie J., Grace H. McGregor, Katrina I. Paras, et al.. (2026). p53 increases phospholipid headgroup scavenging in senescence. Nature Cell Biology. 28(2). 296–306.
3.
Jackson, Benjamin T., et al.. (2025). Intracellular metabolic gradients dictate dependence on exogenous pyruvate. Nature Metabolism. 7(6). 1168–1182. 2 indexed citations
4.
Ros, Manon, Makiko Hayashi, Julia S. Brunner, et al.. (2025). Cooperative nutrient scavenging is an evolutionary advantage in cancer. Nature. 640(8058). 534–542. 4 indexed citations
5.
Jackson, Benjamin T., Vidur Garg, Katrina I. Paras, et al.. (2024). Amino acid intake strategies define pluripotent cell states. Nature Metabolism. 6(1). 127–140. 7 indexed citations
6.
Puchner, Antonia, Elisabeth Simader, Victoria Saferding, et al.. (2023). Bona fide dendritic cells are pivotal precursors for osteoclasts. Annals of the Rheumatic Diseases. 83(4). 518–528. 8 indexed citations
7.
Brunner, Julia S. & Lydia W.S. Finley. (2022). Metabolic determinants of tumour initiation. Nature Reviews Endocrinology. 19(3). 134–150. 40 indexed citations
8.
Vogel, Andrea, Klara Soukup, Angela Halfmann, et al.. (2022). JAK1 signaling in dendritic cells promotes peripheral tolerance in autoimmunity through PD-L1-mediated regulatory T cell induction. Cell Reports. 38(8). 110420–110420. 25 indexed citations
9.
Arnold, Paige K., Benjamin T. Jackson, Katrina I. Paras, et al.. (2022). A non-canonical tricarboxylic acid cycle underlies cellular identity. Nature. 603(7901). 477–481. 172 indexed citations breakdown →
10.
Vogel, Andrea, et al.. (2021). Lipid scavenging macrophages and inflammation. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1867(1). 159066–159066. 55 indexed citations
11.
Capellades, Jordi, Alexandra Junza, Sara Samino, et al.. (2020). Exploring the Use of Gas Chromatography Coupled to Chemical Ionization Mass Spectrometry (GC-CI-MS) for Stable Isotope Labeling in Metabolomics. Analytical Chemistry. 93(3). 1242–1248. 13 indexed citations
12.
Saferding, Victoria, Melanie Hofmann, Julia S. Brunner, et al.. (2020). microRNA‐146a controls age‐related bone loss. Aging Cell. 19(11). e13244–e13244. 28 indexed citations
13.
Whitham, Martin, Martin Pál, Tim Petzold, et al.. (2019). Adipocyte-specific deletion of IL-6 does not attenuate obesity-induced weight gain or glucose intolerance in mice. American Journal of Physiology-Endocrinology and Metabolism. 317(4). E597–E604. 24 indexed citations
14.
Saferding, Victoria, Christopher Schliehe, Robert A. Benson, et al.. (2019). MicroRNA-155 Controls T Helper Cell Activation During Viral Infection. Frontiers in Immunology. 10. 1367–1367. 26 indexed citations
15.
Toplak, Marina, Julia S. Brunner, Julia Schmidt, & Peter Macheroux. (2019). Biochemical characterization of human D-2-hydroxyglutarate dehydrogenase and two disease related variants reveals the molecular cause of D-2-hydroxyglutaric aciduria. Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics. 1867(11). 140255–140255. 19 indexed citations
16.
Kral, Julia Barbara, Mario Kuttke, Waltraud C. Schrottmaier, et al.. (2016). Sustained PI3K Activation exacerbates BLM-induced Lung Fibrosis via activation of pro-inflammatory and pro-fibrotic pathways. Scientific Reports. 6(1). 23034–23034. 74 indexed citations
17.
Blüml, Stephan, Emine Sahin, Victoria Saferding, et al.. (2015). Phosphatase and tensin homolog (PTEN) in antigen-presenting cells controls Th17-mediated autoimmune arthritis. Arthritis Research & Therapy. 17(1). 230–230. 26 indexed citations
18.
Sharif, Omar, Riem Gawish, Joanna Warszawska, et al.. (2014). The Triggering Receptor Expressed on Myeloid Cells 2 Inhibits Complement Component 1q Effector Mechanisms and Exerts Detrimental Effects during Pneumococcal Pneumonia. PLoS Pathogens. 10(6). e1004167–e1004167. 45 indexed citations
19.
Guenzl, Philipp, et al.. (2013). Insulin Hypersensitivity Induced by Hepatic PTEN Gene Ablation Protects from Murine Endotoxemia. PLoS ONE. 8(6). e67013–e67013. 7 indexed citations
20.
Bramberger, Michael, Julia S. Brunner, Bernhard Rinner, & Helmut Schwabach. (2004). Real-time video analysis on an embedded smart camera for traffic surveillance. 174–181. 107 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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