Julia Etich

824 total citations
25 papers, 557 citations indexed

About

Julia Etich is a scholar working on Molecular Biology, Cancer Research and Rheumatology. According to data from OpenAlex, Julia Etich has authored 25 papers receiving a total of 557 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 9 papers in Cancer Research and 6 papers in Rheumatology. Recurrent topics in Julia Etich's work include Connective tissue disorders research (5 papers), Cancer-related molecular mechanisms research (5 papers) and MicroRNA in disease regulation (4 papers). Julia Etich is often cited by papers focused on Connective tissue disorders research (5 papers), Cancer-related molecular mechanisms research (5 papers) and MicroRNA in disease regulation (4 papers). Julia Etich collaborates with scholars based in Germany, Australia and United Kingdom. Julia Etich's co-authors include Bent Brachvogel, Frank Zaucke, Christian Frie, Manuel Koch, Ernst Pöschl, Mirko Rehberg, Oliver Semler, John F. Bateman, Raimund Wagener and Daniele Belluoccio and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Cell Biology and PLoS ONE.

In The Last Decade

Julia Etich

22 papers receiving 554 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Julia Etich Germany 16 276 151 100 78 71 25 557
Daniel Kronenberg Germany 12 162 0.6× 126 0.8× 51 0.5× 66 0.8× 23 0.3× 26 600
Koji Takeda Japan 13 189 0.7× 141 0.9× 32 0.3× 70 0.9× 35 0.5× 31 609
Alexander Y. L. Cheah Canada 7 268 1.0× 73 0.5× 102 1.0× 54 0.7× 67 0.9× 7 546
Kei Kuroda Japan 14 246 0.9× 82 0.5× 82 0.8× 50 0.6× 50 0.7× 27 621
Xianding Sun China 9 449 1.6× 83 0.5× 102 1.0× 77 1.0× 18 0.3× 17 731
Lydia Greenlees United States 10 268 1.0× 113 0.7× 44 0.4× 44 0.6× 15 0.2× 16 480
Julian Nüchel Germany 12 304 1.1× 67 0.4× 33 0.3× 34 0.4× 85 1.2× 14 634
Thusanth Thuraisingam Canada 14 202 0.7× 45 0.3× 83 0.8× 38 0.5× 38 0.5× 25 698
Debra J. Warejcka United States 14 165 0.6× 131 0.9× 51 0.5× 19 0.2× 26 0.4× 25 668
Jon M. Carthy Canada 12 296 1.1× 89 0.6× 23 0.2× 55 0.7× 40 0.6× 20 598

Countries citing papers authored by Julia Etich

Since Specialization
Citations

This map shows the geographic impact of Julia Etich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Julia Etich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Julia Etich more than expected).

Fields of papers citing papers by Julia Etich

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Julia Etich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Julia Etich. The network helps show where Julia Etich may publish in the future.

Co-authorship network of co-authors of Julia Etich

This figure shows the co-authorship network connecting the top 25 collaborators of Julia Etich. A scholar is included among the top collaborators of Julia Etich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Julia Etich. Julia Etich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Etich, Julia, Gereon Poschmann, Bent Brachvogel, et al.. (2025). A fluorescent sensor for real-time monitoring of DPP8/9 reveals crucial roles in immunity and cancer. Life Science Alliance. 8(8). e202403076–e202403076.
2.
Etich, Julia, Frederik Dethloff, Marcus Krüger, et al.. (2025). Metabolic rewiring caused by mitochondrial dysfunction promotes mTORC1-dependent skeletal aging. Science Advances. 11(16). eads1842–eads1842. 4 indexed citations
3.
Zaucke, Frank, et al.. (2024). Osteogenesis imperfecta: shifting paradigms in pathophysiology and care in children. Journal of Pediatric Endocrinology and Metabolism. 38(1). 1–15.
4.
Weiland, Daniela, Doris Helbig, Sabine A. Eming, et al.. (2024). Mitochondrial DNA mutations attenuate Bleomycin-induced dermal fibrosis by inhibiting differentiation into myofibroblasts. Matrix Biology. 132. 72–86. 1 indexed citations
5.
Knuever, Jana, David Pla‐Martín, Olivier R. Baris, et al.. (2024). Constitutive HIF-1α Expression in the Epidermis Fuels Proliferation and Is Essential for Effective Barrier Formation. Journal of Investigative Dermatology. 145(7). 1683–1692.e8.
6.
Etich, Julia, Mirko Rehberg, Beate Eckes, et al.. (2020). Signaling pathways affected by mutations causing osteogenesis imperfecta. Cellular Signalling. 76. 109789–109789. 33 indexed citations
7.
Etich, Julia, et al.. (2020). Osteogenesis imperfecta—pathophysiology and therapeutic options. PubMed. 7(1). 9–9. 40 indexed citations
8.
Etich, Julia, Christian Frie, Juliane Heilig, et al.. (2019). Respiratory chain inactivation links cartilage-mediated growth retardation to mitochondrial diseases. The Journal of Cell Biology. 218(6). 1853–1870. 20 indexed citations
9.
Etich, Julia, Manuel Koch, Raimund Wagener, et al.. (2019). Gene Expression Profiling of the Extracellular Matrix Signature in Macrophages of Different Activation Status: Relevance for Skin Wound Healing. International Journal of Molecular Sciences. 20(20). 5086–5086. 52 indexed citations
10.
Brylka, Laura, Anne Babler, Bernd Denecke, et al.. (2017). Post-weaning epiphysiolysis causes distal femur dysplasia and foreshortened hindlimbs in fetuin-A-deficient mice. PLoS ONE. 12(10). e0187030–e0187030. 18 indexed citations
11.
Ehlen, Harald W.A., Juliane Heilig, Julia Etich, et al.. (2017). miR-322 stabilizes MEK1 expression to inhibit RAF/MEK/ERK pathway activation in cartilage. Development. 144(19). 3562–3577. 30 indexed citations
12.
Etich, Julia, Christian Frie, Manuel Koch, et al.. (2017). Identification of a myofibroblast-specific expression signature in skin wounds. Matrix Biology. 65. 59–74. 63 indexed citations
13.
Stoffel, Wilhelm, Erika Binczek, Susanne Brodesser, et al.. (2016). Neutral sphingomyelinase (SMPD3) deficiency disrupts the Golgi secretory pathway and causes growth inhibition. Cell Death and Disease. 7(11). e2488–e2488. 32 indexed citations
14.
Etich, Julia, et al.. (2016). Identification of a reference gene for the quantification of mRNA and miRNA expression during skin wound healing. Connective Tissue Research. 58(2). 196–207. 16 indexed citations
15.
Frie, Christian, Daniele Belluoccio, Jocelyn van den Bergen, et al.. (2016). miR-126-3p Promotes Matrix-Dependent Perivascular Cell Attachment, Migration and Intercellular Interaction. Stem Cells. 34(5). 1297–1309. 26 indexed citations
16.
Etich, Julia, et al.. (2015). MiR-26a modulates extracellular matrix homeostasis in cartilage. Matrix Biology. 43. 27–34. 32 indexed citations
17.
Etich, Julia & Bent Brachvogel. (2015). Bedeutung der extrazellulären Matrix des Knorpels für die Entwicklung und Funktion des Immunsystems. Zeitschrift für Rheumatologie. 74(8). 711–713. 1 indexed citations
18.
Etich, Julia, Christian Frie, Sabine A. Eming, et al.. (2013). PECAM1+/Sca1+/CD38+ Vascular Cells Transform into Myofibroblast-Like Cells in Skin Wound Repair. PLoS ONE. 8(1). e53262–e53262. 19 indexed citations
19.
Etich, Julia, Christian Frie, Benjamin Frey, et al.. (2010). Identification of Novel Binding Partners (Annexins) for the Cell Death Signal Phosphatidylserine and Definition of Their Recognition Motif. Journal of Biological Chemistry. 286(7). 5708–5716. 48 indexed citations
20.
Brachvogel, Bent, Friederike Pausch, Peter G. Farlie, et al.. (2007). Isolated Anxa5+/Sca-1+ perivascular cells from mouse meningeal vasculature retain their perivascular phenotype in vitro and in vivo. Experimental Cell Research. 313(12). 2730–2743. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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