Julia Diegelmann

1.9k total citations
31 papers, 1.5k citations indexed

About

Julia Diegelmann is a scholar working on Genetics, Immunology and Pathology and Forensic Medicine. According to data from OpenAlex, Julia Diegelmann has authored 31 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Genetics, 18 papers in Immunology and 7 papers in Pathology and Forensic Medicine. Recurrent topics in Julia Diegelmann's work include Inflammatory Bowel Disease (21 papers), Immunodeficiency and Autoimmune Disorders (8 papers) and Psoriasis: Treatment and Pathogenesis (7 papers). Julia Diegelmann is often cited by papers focused on Inflammatory Bowel Disease (21 papers), Immunodeficiency and Autoimmune Disorders (8 papers) and Psoriasis: Treatment and Pathogenesis (7 papers). Julia Diegelmann collaborates with scholars based in Germany, United States and Switzerland. Julia Diegelmann's co-authors include Stephan Brand, Burkhard Göke, Jürgen Glas, Florian Beigel, Matthias Friedrich, Peter Lohse, Torsten Olszak, Julia Seiderer, Martin Wetzke and Cornelia Tillack and has published in prestigious journals such as Journal of Biological Chemistry, Gastroenterology and PLoS ONE.

In The Last Decade

Julia Diegelmann

31 papers receiving 1.5k citations

Peers

Julia Diegelmann
Kristina M. Harris United States
Tracy L. Burcin United States
Ş. Targan United States
George P. Christophi United States
Jane Peake Australia
Julia Diegelmann
Citations per year, relative to Julia Diegelmann Julia Diegelmann (= 1×) peers Sanae Fujino

Countries citing papers authored by Julia Diegelmann

Since Specialization
Citations

This map shows the geographic impact of Julia Diegelmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Julia Diegelmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Julia Diegelmann more than expected).

Fields of papers citing papers by Julia Diegelmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Julia Diegelmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Julia Diegelmann. The network helps show where Julia Diegelmann may publish in the future.

Co-authorship network of co-authors of Julia Diegelmann

This figure shows the co-authorship network connecting the top 25 collaborators of Julia Diegelmann. A scholar is included among the top collaborators of Julia Diegelmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Julia Diegelmann. Julia Diegelmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Schnitzler, Fabian, Matthias Friedrich, Christiane Wolf, et al.. (2015). The NOD2 Single Nucleotide Polymorphism rs72796353 (IVS4+10 A>C) Is a Predictor for Perianal Fistulas in Patients with Crohn's Disease in the Absence of Other NOD2 Mutations. PLoS ONE. 10(7). e0116044–e0116044. 24 indexed citations
3.
Beigel, Florian, Matthias Friedrich, Karl Sotlar, et al.. (2014). Oncostatin M Mediates STAT3-Dependent Intestinal Epithelial Restitution via Increased Cell Proliferation, Decreased Apoptosis and Upregulation of SERPIN Family Members. PLoS ONE. 9(4). e93498–e93498. 49 indexed citations
4.
Friedrich, Matthias, Julia Diegelmann, Florian Beigel, & Stephan Brand. (2014). IL-17A Alone Weakly Affects the Transcriptome of Intestinal Epithelial Cells but Strongly Modulates the TNF-α–induced Expression of Inflammatory Mediators and Inflammatory Bowel Disease Susceptibility Genes. Inflammatory Bowel Diseases. 20(9). 1502–1515. 19 indexed citations
5.
6.
Diegelmann, Julia, Darina Czamara, Eva Zimmermann, et al.. (2013). Intestinal DMBT1 Expression Is Modulated by Crohn’s Disease-Associated IL23R Variants and by a DMBT1 Variant Which Influences Binding of the Transcription Factors CREB1 and ATF-2. PLoS ONE. 8(11). e77773–e77773. 22 indexed citations
8.
Glas, Jürgen, Julia Seiderer, Johannes Stallhofer, et al.. (2013). IRGM Variants and Susceptibility to Inflammatory Bowel Disease in the German Population. PLoS ONE. 8(1). e54338–e54338. 52 indexed citations
9.
Glas, Jürgen, Johanna Wagner, Julia Seiderer, et al.. (2012). PTPN2 Gene Variants Are Associated with Susceptibility to Both Crohn's Disease and Ulcerative Colitis Supporting a Common Genetic Disease Background. PLoS ONE. 7(3). e33682–e33682. 55 indexed citations
10.
Glas, Jürgen, Julia Seiderer, Johanna Wagner, et al.. (2012). Analysis of IL12B Gene Variants in Inflammatory Bowel Disease. PLoS ONE. 7(3). e34349–e34349. 50 indexed citations
11.
Glas, Jürgen, Julia Seiderer, Darina Czamara, et al.. (2012). PTGER4 Expression-Modulating Polymorphisms in the 5p13.1 Region Predispose to Crohn's Disease and Affect NF-κB and XBP1 Binding Sites. PLoS ONE. 7(12). e52873–e52873. 31 indexed citations
12.
Glas, Jürgen, Julia Seiderer, Cornelia Tillack, et al.. (2011). CEACAM6 Gene Variants in Inflammatory Bowel Disease. PLoS ONE. 6(4). e19319–e19319. 13 indexed citations
13.
Glas, Jürgen, Julia Seiderer, Martin Wetzke, et al.. (2011). The Role of Osteopontin (OPN/SPP1) Haplotypes in the Susceptibility to Crohn's Disease. PLoS ONE. 6(12). e29309–e29309. 36 indexed citations
14.
Diegelmann, Julia, Torsten Olszak, Burkhard Göke, Richard S. Blumberg, & Stephan Brand. (2011). A Novel Role for Interleukin-27 (IL-27) as Mediator of Intestinal Epithelial Barrier Protection Mediated via Differential Signal Transducer and Activator of Transcription (STAT) Protein Signaling and Induction of Antibacterial and Anti-inflammatory Proteins. Journal of Biological Chemistry. 287(1). 286–298. 110 indexed citations
15.
Glas, Jürgen, Julia Seiderer, Cornelia Tillack, et al.. (2010). The NOD2 Single Nucleotide Polymorphisms rs2066843 and rs2076756 Are Novel and Common Crohn's Disease Susceptibility Gene Variants. PLoS ONE. 5(12). e14466–e14466. 26 indexed citations
16.
Glas, Jürgen, Julia Seiderer, D Fischer, et al.. (2010). Pregnane X receptor (PXR/NR1I2) gene haplotypes modulate susceptibility to inflammatory bowel disease. Inflammatory Bowel Diseases. 17(9). 1917–1924. 31 indexed citations
17.
Storr, Martin, Julia Diegelmann, Simone Pfennig, et al.. (2010). The Cannabinoid 1 Receptor (CNR1) 1359 G/A Polymorphism Modulates Susceptibility to Ulcerative Colitis and the Phenotype in Crohn's Disease. PLoS ONE. 5(2). e9453–e9453. 42 indexed citations
18.
Diegelmann, Julia, Florian Beigel, Kathrin Zitzmann, et al.. (2010). Comparative Analysis of the Lambda-Interferons IL-28A and IL-29 regarding Their Transcriptome and Their Antiviral Properties against Hepatitis C Virus. PLoS ONE. 5(12). e15200–e15200. 71 indexed citations
19.
Storr, Martin, Julia Diegelmann, B Yüce, et al.. (2008). The role of fatty acid hydrolase gene variants in inflammatory bowel disease. Alimentary Pharmacology & Therapeutics. 29(5). 542–551. 24 indexed citations
20.
Schmechel, Silke, Astrid Konrad, Julia Diegelmann, et al.. (2007). Linking genetic susceptibility to Crohnʼs disease with Th17 cell function: IL-22 serum levels are increased in Crohnʼs disease and correlate with disease activity and IL23R genotype status. Inflammatory Bowel Diseases. 14(2). 204–212. 165 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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