Juha Laurén

3.8k total citations · 1 hit paper
16 papers, 2.8k citations indexed

About

Juha Laurén is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Juha Laurén has authored 16 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 5 papers in Cell Biology. Recurrent topics in Juha Laurén's work include Alzheimer's disease research and treatments (5 papers), Prion Diseases and Protein Misfolding (4 papers) and Cellular transport and secretion (3 papers). Juha Laurén is often cited by papers focused on Alzheimer's disease research and treatments (5 papers), Prion Diseases and Protein Misfolding (4 papers) and Cellular transport and secretion (3 papers). Juha Laurén collaborates with scholars based in Finland, United States and Estonia. Juha Laurén's co-authors include Stephen M. Strittmatter, Haakon B. Nygaard, David A. Gimbel, John W. Gilbert, Matti S. Airaksinen, Tõnis Timmusk, Märt Saarma, Erik C. Gunther, Erin E. Coffey and Kari Alitalo and has published in prestigious journals such as Nature, Circulation and Neuron.

In The Last Decade

Juha Laurén

16 papers receiving 2.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Cellular prion protein mediates impairment of synaptic plasticity by amyloid-β oligomers

2009 1.2k citations Juha Laurén, David A. Gimbel et al. Nature profile →

Peers

Juha Laurén

PHYSI

CMN

NEURO

Ji Won Um South Korea

Massimiliano Stagi United States

Edda Thies Germany

Séverine Bégard France

Uwe Konietzko Switzerland

Christopher William United States

Bernadette Allinquant France

Chihiro Hisatsune Japan

Katrien Horré Belgium

Joseph W. Lewcock United States

Ji Won Um South Korea

Juha Laurén

1.8k ×1.1

1.1k ×0.9

PHYSI

1.4k ×1.4

CMN

615 ×1.2

NEURO

569 ×1.6

Citations per year, relative to Juha Laurén Juha Laurén (= 1×) peers Ji Won Um

Countries citing papers authored by Juha Laurén

Since Specialization

Citations

This map shows the geographic impact of Juha Laurén's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Juha Laurén with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Juha Laurén more than expected).

Fields of papers citing papers by Juha Laurén

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Juha Laurén. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Juha Laurén. The network helps show where Juha Laurén may publish in the future.

Co-authorship network of co-authors of Juha Laurén

This figure shows the co-authorship network connecting the top 25 collaborators of Juha Laurén. A scholar is included among the top collaborators of Juha Laurén based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Juha Laurén. Juha Laurén is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Laurén, Juha. (2013). Cellular Prion Protein as a Therapeutic Target in Alzheimer's Disease. Journal of Alzheimer s Disease. 38(2). 227–244. 46 indexed citations

Võikar, Vootele, et al.. (2012). LRRTM1-deficient mice show a rare phenotype of avoiding small enclosures—A tentative mouse model for claustrophobia-like behaviour. Behavioural Brain Research. 238. 69–78. 20 indexed citations

Kysenius, Kai, et al.. (2012). LRRTM3 is Dispensable for Amyloid-β Production in Mice. Journal of Alzheimer s Disease. 31(4). 759–764. 6 indexed citations

Gimbel, David A., Haakon B. Nygaard, Erin E. Coffey, et al.. (2010). Memory Impairment in Transgenic Alzheimer Mice Requires Cellular Prion Protein. Journal of Neuroscience. 30(18). 6367–6374. 342 indexed citations

Laurén, Juha, David A. Gimbel, Haakon B. Nygaard, John W. Gilbert, & Stephen M. Strittmatter. (2010). Laurén et al. reply. Nature. 466(7308). E4–E5. 6 indexed citations

Linhoff, Michael W., Juha Laurén, Robert M. Cassidy, et al.. (2009). An Unbiased Expression Screen for Synaptogenic Proteins Identifies the LRRTM Protein Family as Synaptic Organizers. Neuron. 61(5). 734–749. 286 indexed citations

Laurén, Juha, David A. Gimbel, Haakon B. Nygaard, John W. Gilbert, & Stephen M. Strittmatter. (2009). Cellular prion protein mediates impairment of synaptic plasticity by amyloid-β oligomers. Nature. 457(7233). 1128–1132. 1234 indexed citations breakdown →

Seitsonen, Oula, et al.. (2009). Pastoralists in the northeastern Mara Plains, Kenya: archaeological investigations of the Pastoral Neolithic and the Pastoral Iron Age. Azania Archaeological Research in Africa. 44(2). 163–193. 7 indexed citations

Laurén, Juha, et al.. (2009). Neuralized-2: Expression in human and rodents and interaction with Delta-like ligands. Biochemical and Biophysical Research Communications. 389(3). 420–425. 14 indexed citations

10.

Budel, Stéphane, Thihan Padukkavidana, Betty P. Liu, et al.. (2008). Genetic Variants of Nogo-66 Receptor with Possible Association to Schizophrenia Block Myelin Inhibition of Axon Growth. Journal of Neuroscience. 28(49). 13161–13172. 82 indexed citations

11.

Lindholm, Päivi, Merja H. Voutilainen, Juha Laurén, et al.. (2007). Novel neurotrophic factor CDNF protects and rescues midbrain dopamine neurons in vivo. Nature. 448(7149). 73–77. 349 indexed citations

12.

Laurén, Juha. (2007). Characterization of LRRTM and NGR gene families: Expression and functions. Työväentutkimus Vuosikirja. 3 indexed citations

13.

Laurén, Juha, Matti S. Airaksinen, Märt Saarma, & Tõnis Timmusk. (2003). A novel gene family encoding leucine-rich repeat transmembrane proteins differentially expressed in the nervous system☆☆Sequence data from this article have been deposited with the DDBJ/EMBL/GenBank Data Libraries under Accession Nos. AY182024 (human LRRTM1), AY182026 (human LRRTM2), AY182028 (human LRRTM3), AY182030 (human LRRTM4), AY182025 (mouse LRRTM1), AY182027 (mouse LRRTM2), AY182029 (mouse LRRTM3), and AY182031 (mouse LRRTM4).. Genomics. 81(4). 411–421. 115 indexed citations

14.

Laurén, Juha, Matti S. Airaksinen, Märt Saarma, & Tõnis Timmusk. (2003). Two novel mammalian nogo receptor homologs differentially expressed in the central and peripheral nervous systems☆. Molecular and Cellular Neuroscience. 24(3). 581–594. 63 indexed citations

15.

Uutela, Marko, Juha Laurén, Xuri Li, et al.. (2001). Chromosomal Location, Exon Structure, and Vascular Expression Patterns of the Human PDGFC and PDGFD Genes. Circulation. 103(18). 2242–2247. 104 indexed citations

16.

Laurén, Juha, Yuji Gunji, & Kari Alitalo. (1998). Is Angiopoietin-2 Necessary for the Initiation of Tumor Angiogenesis?. American Journal Of Pathology. 153(5). 1333–1339. 81 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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