Ju‐Ha Kim

648 total citations
20 papers, 495 citations indexed

About

Ju‐Ha Kim is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Ju‐Ha Kim has authored 20 papers receiving a total of 495 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Cancer Research and 4 papers in Oncology. Recurrent topics in Ju‐Ha Kim's work include Cell death mechanisms and regulation (5 papers), RNA modifications and cancer (4 papers) and Cancer-related molecular mechanisms research (3 papers). Ju‐Ha Kim is often cited by papers focused on Cell death mechanisms and regulation (5 papers), RNA modifications and cancer (4 papers) and Cancer-related molecular mechanisms research (3 papers). Ju‐Ha Kim collaborates with scholars based in South Korea and United States. Ju‐Ha Kim's co-authors include Sung‐Hoon Kim, Hyemin Lee, Hyo‐Jung Lee, Ji Hoon Jung, Deok Yong Sim, Eunah Shin, Bum Sang Shim, Sung‐Hoon Kim, Dae‐Young Lee and Bonglee Kim and has published in prestigious journals such as PLoS ONE, Journal of Agricultural and Food Chemistry and International Journal of Molecular Sciences.

In The Last Decade

Ju‐Ha Kim

19 papers receiving 489 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ju‐Ha Kim South Korea 14 309 119 74 50 38 20 495
Victor C. Lin Taiwan 13 352 1.1× 129 1.1× 58 0.8× 32 0.6× 41 1.1× 31 652
Maleppillil Vavachan Vijayakumar India 11 246 0.8× 114 1.0× 134 1.8× 35 0.7× 16 0.4× 14 590
Alyssa Langfald United States 10 290 0.9× 69 0.6× 102 1.4× 25 0.5× 41 1.1× 18 488
Andria Carper United States 9 284 0.9× 53 0.4× 102 1.4× 52 1.0× 24 0.6× 11 438
On‐Yu Hong South Korea 11 219 0.7× 79 0.7× 52 0.7× 23 0.5× 26 0.7× 15 416
Kanamata Reddy United States 15 470 1.5× 65 0.5× 163 2.2× 45 0.9× 31 0.8× 27 706
Pelin Telkoparan‐Akillilar Türkiye 13 440 1.4× 94 0.8× 49 0.7× 67 1.3× 9 0.2× 30 686
Ji‐Hye Song South Korea 16 363 1.2× 63 0.5× 33 0.4× 22 0.4× 21 0.6× 55 658
Yewseok Suh United States 10 377 1.2× 59 0.5× 69 0.9× 43 0.9× 32 0.8× 14 661
Woo-Kwang Jeon South Korea 12 347 1.1× 52 0.4× 85 1.1× 47 0.9× 27 0.7× 15 551

Countries citing papers authored by Ju‐Ha Kim

Since Specialization
Citations

This map shows the geographic impact of Ju‐Ha Kim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ju‐Ha Kim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ju‐Ha Kim more than expected).

Fields of papers citing papers by Ju‐Ha Kim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ju‐Ha Kim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ju‐Ha Kim. The network helps show where Ju‐Ha Kim may publish in the future.

Co-authorship network of co-authors of Ju‐Ha Kim

This figure shows the co-authorship network connecting the top 25 collaborators of Ju‐Ha Kim. A scholar is included among the top collaborators of Ju‐Ha Kim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ju‐Ha Kim. Ju‐Ha Kim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kim, Ju‐Ha, et al.. (2025). Elucidating the Role of CNOT2 in Regulating Cancer Cell Growth via the Modulation of p53 and c-Myc Expression. Current Issues in Molecular Biology. 47(8). 615–615. 1 indexed citations
2.
3.
Jung, Ji Hoon, Hyo‐Jung Lee, Ju‐Ha Kim, et al.. (2020). Colocalization of MID1IP1 and c-Myc is Critically Involved in Liver Cancer Growth via Regulation of Ribosomal Protein L5 and L11 and CNOT2. Cells. 9(4). 985–985. 31 indexed citations
4.
Jung, Ji Hoon, Hyemin Lee, Ju‐Ha Kim, et al.. (2019). p53-Dependent Apoptotic Effect of Puromycin via Binding of Ribosomal Protein L5 and L11 to MDM2 and Its Combination Effect with RITA or Doxorubicin. Cancers. 11(4). 582–582. 27 indexed citations
5.
6.
Lee, Hyo‐Jung, Ji Hoon Jung, Eunji Im, et al.. (2019). The Pivotal Role of Long Noncoding RNA RAB5IF in the Proliferation of Hepatocellular Carcinoma via LGR5 Mediated β-Catenin and c-Myc Signaling. Biomolecules. 9(11). 718–718. 13 indexed citations
7.
8.
Jung, Ji Hoon, Ju‐Ha Kim, Deok Yong Sim, et al.. (2019). Phyotochemical candidates repurposing for cancer therapy and their molecular mechanisms. Seminars in Cancer Biology. 68. 164–174. 12 indexed citations
9.
Kim, Ju‐Ha, Deok Yong Sim, Min‐Ho Nam, et al.. (2019). Inhibition of JAK2/STAT3 and activation of caspase‑9/3 are involved in KYS05090S‑induced apoptosis in ovarian cancer cells. International Journal of Oncology. 55(1). 203–210. 14 indexed citations
10.
Jung, Ji Hoon, Eunah Shin, Ju‐Ha Kim, et al.. (2019). NEDD9 Inhibition by miR-25-5p Activation Is Critically Involved in Co-Treatment of Melatonin- and Pterostilbene-Induced Apoptosis in Colorectal Cancer Cells. Cancers. 11(11). 1684–1684. 27 indexed citations
11.
Park, Eunkyung, et al.. (2018). The Anti-Wrinkle Mechanism of Melatonin in UVB Treated HaCaT Keratinocytes and Hairless Mice via Inhibition of ROS and Sonic Hedgehog Mediated Inflammatory Proteins. International Journal of Molecular Sciences. 19(7). 1995–1995. 37 indexed citations
12.
Kim, Ju‐Ha, et al.. (2018). Reactive Oxygen Species and p53 Mediated Activation of p38 and Caspases is Critically Involved in Kaempferol Induced Apoptosis in Colorectal Cancer Cells. Journal of Agricultural and Food Chemistry. 66(38). 9960–9967. 77 indexed citations
13.
Kim, Ju‐Ha, Hee Young Kwon, Min‐Ho Nam, et al.. (2017). Inhibition of CUG-binding protein 1 and activation of caspases are critically involved in piperazine derivative BK10007S induced apoptosis in hepatocellular carcinoma cells. PLoS ONE. 12(10). e0186490–e0186490. 9 indexed citations
14.
Kwon, Hee Young, Ju‐Ha Kim, Bonglee Kim, Sanjay Srivastava, & Sung‐Hoon Kim. (2017). Regulation of SIRT1/AMPK axis is critically involved in gallotannin-induced senescence and impaired autophagy leading to cell death in hepatocellular carcinoma cells. Archives of Toxicology. 92(1). 241–257. 26 indexed citations
15.
Kim, Ju‐Ha, et al.. (2017). Short Term Effect of Monocular Wearable Device on Accommodation. The Korean Journal of Vision Science. 19(1). 61–68. 2 indexed citations
16.
Lee, Hyemin, Eunah Shin, Jae Hee Lee, et al.. (2017). Caspase inhibitors: a review of recently patented compounds (2013-2015). Expert Opinion on Therapeutic Patents. 28(1). 47–59. 44 indexed citations
17.
Kim, Ju‐Ha, et al.. (2017). Implications of Bcl-2 and its interplay with other molecules and signaling pathways in prostate cancer progression. Expert Opinion on Therapeutic Targets. 21(9). 911–920. 25 indexed citations
18.
19.
Shin, Eunah, Hyemin Lee, Ju‐Ha Kim, et al.. (2017). Apoptotic Effect of Astragalin in Melanoma Skin Cancers via Activation of Caspases and Inhibition of Sry‐related HMg‐Box Gene 10. Phytotherapy Research. 31(10). 1614–1620. 30 indexed citations
20.
Jung, Deok‐Beom, Kyeong‐Hwa Seo, Ju‐Ha Kim, et al.. (2015). Apoptotic Effect of Sanggenol L via Caspase Activation and Inhibition of NF-κB Signaling in Ovarian Cancer Cells. Phytotherapy Research. 30(1). 90–96. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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