Judit P. Banáth

11.4k citations

69 papers · 7.3k indexed · 2 hit papers · h-index 38

Cancer Research top 0.5%
- Carcinogens and Genotoxicity Assessment 25
Molecular Biology top 1%
- DNA Repair Mechanisms 34
- Cancer therapeutics and mechanisms 11
- DNA and Nucleic Acid Chemistry 7
- Genomics and Chromatin Dynamics 6
Health, Toxicology and Mutagenesis top 1%
Oncology top 2%
- Cancer-related Molecular Pathways 9
Toxicology top 1%
Biomedical Engineering
- Nanoplatforms for cancer theranostics 6
Pulmonary and Respiratory Medicine
- Photodynamic Therapy Research Studies 6

Co-authors: Peggy L. Olive Ralph E. Durand Susan H. MacPhail D. Włodek R. E. Durand Dmitry Klokov Carmen A. Bañuelos Peter J. Johnston
Cited by: Cancer Research Molecular Biology Health, Toxicology and Mutagenesis
Partner nations: Canada United States New Zealand

In The Last Decade

Judit P. Banáth

68 papers receiving 7.0k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

2006 The comet assay: a method to measure DNA damage in individual cells
1990 Heterogeneity in Radiation-Induced DNA Damage and Repair in Tumor and Normal Cells Measured Using the "Comet" Assay

Peers

Countries citing papers authored by Judit P. Banáth

Since Specialization

Citations

This map shows the geographic impact of Judit P. Banáth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Judit P. Banáth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Judit P. Banáth more than expected).

Fields of papers citing papers by Judit P. Banáth

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Judit P. Banáth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Judit P. Banáth. The network helps show where Judit P. Banáth may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Judit P. Banáth, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Judit P. Banáth Line = papers co-authored together Judit P. Banáth links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	Radiation and Chemo-Sensitizing Effects of DNA-PK Inhibitors Are Proportional in Tumors and Normal Tissues Jennifer H.E. Baker,Alastair H. Kyle,Nannan A. Liu,Taixiang Wang,Xinhe Liu,Sevin Teymori,Judit P. Banáth,Andrew I. Minchinton	2024	4
2	Radiosensitizing oxygenation changes in murine tumors treated with VEGF-ablation therapy are measurable using oxygen enhanced-MRI (OE-MRI) Jennifer H.E. Baker,Firas Moosvi,Alastair H. Kyle,Judit P. Banáth,Katayoun Saatchi,Urs O. Häfeli,Stefan A. Reinsberg,Andrew I. Minchinton	2023	1
3	N-dihydrogalactochitosan-supported tumor control by photothermal therapy and photothermal therapy-generated vaccine Mladen Korbelik,Judit P. Banáth,Wei Zhang,Tomas Hode,Samuel S.K. Lam,Paul Gallagher,Jianhua Zhao,Haishan Zeng,Wei R. Chen	2020	18
4	N-dihydrogalactochitosan as immune and direct antitumor agent amplifying the effects of photodynamic therapy and photodynamic therapy-generated vaccines Mladen Korbelik,Judit P. Banáth,Wei Zhang,Paul Gallagher,Tomas Hode,Samuel S.K. Lam,Wei R. Chen	2019	31
5	Residual γH2AX foci as an indication of lethal DNA lesions Judit P. Banáth,Dmitry Klokov,Susan H. MacPhail,Carmen A. Bañuelos,Peggy L. Olive	2010	158
6	γH2AX Expression in Tumors Exposed to Cisplatin and Fractionated Irradiation Carmen A. Bañuelos,Judit P. Banáth,Joo-Young Kim,Christina Aquino‐Parsons,Peggy L. Olive	2009	42
7	Effect of prolonging radiation delivery time on retention of gammaH2AX Vitali Moiseenko,Judit P. Banáth,Cheryl Duzenli,Peggy L. Olive	2008	22
8	Mouse but not human embryonic stem cells are deficient in rejoining of ionizing radiation-induced DNA double-strand breaks Carmen A. Bañuelos,Judit P. Banáth,Susan H. MacPhail,Jin Zhao,CJ Eaves,Michael D. O’Connor,Peter M. Lansdorp,Peggy L. Olive	2008	67
9	Explanation for excessive DNA single-strand breaks and endogenous repair foci in pluripotent mouse embryonic stem cells Judit P. Banáth,Carmen A. Bañuelos,Dmitry Klokov,Susan Macphail,Peter M. Lansdorp,Peggy L. Olive	2008	73
10	Radiosensitization by the Histone Deacetylase Inhibitor PCI-24781 Carmen A. Bañuelos,Judit P. Banáth,Susan H. MacPhail,Jin Zhao,Tarren Reitsema,Peggy L. Olive	2007	43
11	DNA-PK is responsible for enhanced phosphorylation of histone H2AX under hypertonic conditions Tarren Reitsema,Dmitry Klokov,Judit P. Banáth,Peggy L. Olive	2005	38
12	Growth of V79 Cells as Xenograft Tumors Promotes Multicellular Resistance but does not Increase Spontaneous or Radiation-Induced Mutant Frequency Judit P. Banáth,L. Sinnott,Bruno Larrivée,Susan H. MacPhail,Peggy L. Olive	2005	7
13	Hypertonic Saline Enhances Expression of Phosphorylated Histone H2AX after Irradiation Tarren Reitsema,Judit P. Banáth,Susan H. MacPhail,Peggy L. Olive	2004	21
14	Cell Cycle-Dependent Expression of Phosphorylated Histone H2AX: Reduced Expression in Unirradiated but not X-Irradiated G₁-Phase Cells Susan H. MacPhail,Judit P. Banáth,Ying Yu,En-Ping Chu,Peggy L. Olive	2003	202
15	Local hypoxia is produced at sites of intratumour injection P L Olive,C-M Luo,Judit P. Banáth	2002	7
16	Sizing Highly Fragmented DNA in Individual Apoptotic Cells Using the Comet Assay and a DNA Crosslinking Agent Peggy L. Olive,Judit P. Banáth	1995	131
17	Detection of DNA Double-strand Breaks through the Cell Cycle after Exposure to X-rays, Bleomycin, Etoposide and ¹²⁵ IdUrd Peggy L. Olive,Judit P. Banáth	1993	103
18	Factors influencing DNA migration from individual cells subjected to gel electrophoresis Peggy L. Olive,D. Włodek,R. E. Durand,Judit P. Banáth	1992	313
19	Etoposide Sensitivity and Topoisomerase II Activity in Chinese Hamster V79 Monolayers and Small Spheroids Peggy L. Olive,R. E. Durand,Judit P. Banáth,Helen H. Evans	1991	21
20	Comparison between Pulsed-field and Constant-field Gel Electrophoresis for Measurement of DNA Double-strand Breaks in Irradiated Chinese Hamster Ovary Cells D. Włodek,Judit P. Banáth,Peggy L. Olive	1991	68

About Judit P. Banáth

Judit P. Banáth is a scholar working on Cancer Research, Oncology and Molecular Biology, having authored 69 papers that have together received 7.3k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (34 papers), Carcinogens and Genotoxicity Assessment (25 papers), Cancer therapeutics and mechanisms (11 papers), Cancer-related Molecular Pathways (9 papers), DNA and Nucleic Acid Chemistry (7 papers), Nanoplatforms for cancer theranostics (6 papers), Genomics and Chromatin Dynamics (6 papers) and Photodynamic Therapy Research Studies (6 papers). The work is most often cited by research in Cancer Research (2.7k citations), Molecular Biology (4.4k citations) and Health, Toxicology and Mutagenesis (796 citations). Judit P. Banáth has collaborated with scholars based in Canada, United States and New Zealand. Frequent co-authors include Peggy L. Olive, Ralph E. Durand, Susan H. MacPhail, D. Włodek, R. E. Durand, Dmitry Klokov, Carmen A. Bañuelos, Peter J. Johnston, R. E. Durand and Ying Yu.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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