Joseph Chapo

528 total citations
7 papers, 398 citations indexed

About

Joseph Chapo is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Cell Biology. According to data from OpenAlex, Joseph Chapo has authored 7 papers receiving a total of 398 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 2 papers in Pathology and Forensic Medicine and 2 papers in Cell Biology. Recurrent topics in Joseph Chapo's work include Signaling Pathways in Disease (3 papers), Nitric Oxide and Endothelin Effects (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). Joseph Chapo is often cited by papers focused on Signaling Pathways in Disease (3 papers), Nitric Oxide and Endothelin Effects (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). Joseph Chapo collaborates with scholars based in United States, China and United Kingdom. Joseph Chapo's co-authors include David B. Hood, Timothy A. McKinsey, Philip J. Papst, Wayne Minobe, Eric N. Olson, Erik W. Bush, Michael R. Bristow, Wan-Jin Lu, Ignasi Roig and John Abrams and has published in prestigious journals such as Science, Journal of Biological Chemistry and FEBS Letters.

In The Last Decade

Joseph Chapo

7 papers receiving 396 citations

Peers

Joseph Chapo

MB

SS

CCM

ONCOL

SURGE

David B. Hood China

Alf Beckmann Germany

Mizuka Iwatsubo United States

Mirko Voelkers Germany

Long‐Xin Gui China

Marielle Anger France

Christie‐Ann McCarl United States

Koteswara R. Chava United States

Jan Flesche United States

Wei-Chiao Chen Taiwan

David B. Hood China

Joseph Chapo

258 ×1.0

MB

143 ×1.0

SS

133 ×1.1

CCM

40 ×0.8

ONCOL

24 ×0.8

SURGE

Citations per year, relative to Joseph Chapo Joseph Chapo (= 1×) peers David B. Hood

Countries citing papers authored by Joseph Chapo

Since Specialization

Citations

This map shows the geographic impact of Joseph Chapo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph Chapo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph Chapo more than expected).

Fields of papers citing papers by Joseph Chapo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph Chapo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph Chapo. The network helps show where Joseph Chapo may publish in the future.

Co-authorship network of co-authors of Joseph Chapo

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph Chapo. A scholar is included among the top collaborators of Joseph Chapo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph Chapo. Joseph Chapo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Liles, John T., et al.. (2011). Age exacerbates chronic catecholamine-induced impairments in contractile reserve in the rat. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 301(2). R491–R499. 5 indexed citations

2.

Cavasin, Maria A., et al.. (2010). Acute effects of endothelin receptor antagonists on hepatic hemodynamics of cirrhotic and noncirrhotic ratsThis article is one of a selection of papers published in the two-part special issue entitled 20 Years of Endothelin Research.. Canadian Journal of Physiology and Pharmacology. 88(6). 636–643. 16 indexed citations

3.

Lu, Wan-Jin, Joseph Chapo, Ignasi Roig, & John Abrams. (2010). Meiotic Recombination Provokes Functional Activation of the p53 Regulatory Network. Science. 328(5983). 1278–1281. 73 indexed citations

4.

Monovich, Lauren G., Keith A. Koch, Daniel Wall, et al.. (2009). Suppression of HDAC nuclear export and cardiomyocyte hypertrophy by novel irreversible inhibitors of CRM1. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1789(5). 422–431. 26 indexed citations

5.

Monovich, Lauren G., Erik L. Meredith, Michael Capparelli, et al.. (2009). A novel kinase inhibitor establishes a predominant role for protein kinase D as a cardiac class IIa histone deacetylase kinase. FEBS Letters. 584(3). 631–637. 48 indexed citations

6.

Bush, Erik W., David B. Hood, Philip J. Papst, et al.. (2006). Canonical Transient Receptor Potential Channels Promote Cardiomyocyte Hypertrophy through Activation of Calcineurin Signaling. Journal of Biological Chemistry. 281(44). 33487–33496. 228 indexed citations

7.

Chapo, Joseph, et al.. (2006). A phosphorimager-based filter binding thyroid hormone receptor competition assay for chemical screening. Journal of Pharmacological and Toxicological Methods. 56(1). 28–33. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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