Joo‐ri Kim‐Kaneyama

2.3k total citations
44 papers, 1.8k citations indexed

About

Joo‐ri Kim‐Kaneyama is a scholar working on Molecular Biology, Immunology and Allergy and Cell Biology. According to data from OpenAlex, Joo‐ri Kim‐Kaneyama has authored 44 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 13 papers in Immunology and Allergy and 12 papers in Cell Biology. Recurrent topics in Joo‐ri Kim‐Kaneyama's work include Cell Adhesion Molecules Research (13 papers), Calpain Protease Function and Regulation (6 papers) and Atherosclerosis and Cardiovascular Diseases (6 papers). Joo‐ri Kim‐Kaneyama is often cited by papers focused on Cell Adhesion Molecules Research (13 papers), Calpain Protease Function and Regulation (6 papers) and Atherosclerosis and Cardiovascular Diseases (6 papers). Joo‐ri Kim‐Kaneyama collaborates with scholars based in Japan, United States and China. Joo‐ri Kim‐Kaneyama's co-authors include Motoko Shibanuma, Kiyoshi Nose, Akira Miyazaki, Takuro Miyazaki, Xiao‐Feng Lei, Masataka Sata, Takuya Watanabe, Masako Tomoyasu, Masaharu Nagashima and Tsutomu Hirano and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of Clinical Investigation.

In The Last Decade

Joo‐ri Kim‐Kaneyama

44 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Joo‐ri Kim‐Kaneyama Japan	23	854	330	318	287	240	44	1.8k
Éva Korpos Germany	19	458 0.5×	166 0.5×	341 1.1×	257 0.9×	170 0.7×	35	1.4k
Ghada S. Hassan Canada	24	702 0.8×	440 1.3×	361 1.1×	334 1.2×	136 0.6×	51	1.7k
C. M. Petersen Denmark	17	667 0.8×	269 0.8×	199 0.6×	195 0.7×	223 0.9×	22	1.6k
Behrooz G. Sharifi United States	30	1.1k 1.3×	245 0.7×	523 1.6×	640 2.2×	451 1.9×	67	2.8k
Juan Carlos Rodríguez‐Manzaneque Spain	22	1.2k 1.4×	396 1.2×	319 1.0×	134 0.5×	421 1.8×	40	2.4k
John Doukas United States	20	836 1.0×	125 0.4×	489 1.5×	203 0.7×	243 1.0×	35	1.9k
Ingeborg Klaassen Netherlands	28	1.5k 1.7×	249 0.8×	220 0.7×	125 0.4×	91 0.4×	71	3.0k
Feyza Engin United States	16	904 1.1×	510 1.5×	147 0.5×	680 2.4×	45 0.2×	21	1.9k
Rita Dreier Germany	28	1.1k 1.3×	536 1.6×	203 0.6×	280 1.0×	280 1.2×	51	2.5k
Ivo Cornelissen United States	21	1.8k 2.1×	446 1.4×	498 1.6×	252 0.9×	136 0.6×	28	3.0k

Countries citing papers authored by Joo‐ri Kim‐Kaneyama

Since Specialization

Citations

This map shows the geographic impact of Joo‐ri Kim‐Kaneyama's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joo‐ri Kim‐Kaneyama with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joo‐ri Kim‐Kaneyama more than expected).

Fields of papers citing papers by Joo‐ri Kim‐Kaneyama

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joo‐ri Kim‐Kaneyama. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joo‐ri Kim‐Kaneyama. The network helps show where Joo‐ri Kim‐Kaneyama may publish in the future.

Co-authorship network of co-authors of Joo‐ri Kim‐Kaneyama

This figure shows the co-authorship network connecting the top 25 collaborators of Joo‐ri Kim‐Kaneyama. A scholar is included among the top collaborators of Joo‐ri Kim‐Kaneyama based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joo‐ri Kim‐Kaneyama. Joo‐ri Kim‐Kaneyama is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

MASAKI, Y., Masashi Sakaki, Xiao‐Feng Lei, et al.. (2024). Hic-5 antisense oligonucleotide inhibits advanced hepatic fibrosis and steatosis in vivo. JHEP Reports. 6(11). 101195–101195. 1 indexed citations

Lei, Xiao‐Feng, et al.. (2023). Knockdown of mechanosensitive adaptor Hic-5 ameliorates post-traumatic osteoarthritis in rats through repression of MMP-13. Scientific Reports. 13(1). 7446–7446. 3 indexed citations

Phương, Phạm Trần, Joo‐ri Kim‐Kaneyama, Xiao‐Feng Lei, et al.. (2023). Innate Immune System Regulated by Stimulator of Interferon Genes, a Cytosolic DNA Sensor, Regulates Endothelial Function. Journal of the American Heart Association. 12(22). e030084–e030084. 5 indexed citations

Miyazaki, Takuro, Yoshitaka Taketomi, Hirokazu Ohtaki, et al.. (2022). Hypercholesterolemic Dysregulation of Calpain in Lymphatic Endothelial Cells Interferes With Regulatory T-Cell Stability and Trafficking. Arteriosclerosis Thrombosis and Vascular Biology. 43(2). e66–e82. 13 indexed citations

Gao, Lin, Xiao‐Feng Lei, Shogo Haraguchi, et al.. (2020). Hic-5 is required for activation of pancreatic stellate cells and development of pancreatic fibrosis in chronic pancreatitis. Scientific Reports. 10(1). 19105–19105. 10 indexed citations

Kim‐Kaneyama, Joo‐ri, Xiao‐Feng Lei, Song Ho Chang, et al.. (2019). Alleviation of murine osteoarthritis by deletion of the focal adhesion mechanosensitive adapter, Hic-5. Scientific Reports. 9(1). 15770–15770. 9 indexed citations

Oichi, Takeshi, Yuki Taniguchi, Yasushi Oshima, et al.. (2019). Adamts17 is involved in skeletogenesis through modulation of BMP-Smad1/5/8 pathway. Cellular and Molecular Life Sciences. 76(23). 4795–4809. 27 indexed citations

Chang, Song Ho, Daisuke Mori, Hiroshi Kobayashi, et al.. (2019). Excessive mechanical loading promotes osteoarthritis through the gremlin-1–NF-κB pathway. Nature Communications. 10(1). 1442–1442. 229 indexed citations

Haraguchi, Shogo, Takuro Miyazaki, Joo‐ri Kim‐Kaneyama, et al.. (2018). Expression of steroidogenic enzymes and metabolism of steroids in COS-7 cells known as non-steroidogenic cells. Scientific Reports. 8(1). 2167–2167. 7 indexed citations

10.

Shimabukuro, Michio, Hirotsugu Yamada, Etsuko Uematsu, et al.. (2016). The pathophysiological role of oxidized cholesterols in epicardial fat accumulation and cardiac dysfunction: a study in swine fed a high caloric diet with an inhibitor of intestinal cholesterol absorption, ezetimibe. The Journal of Nutritional Biochemistry. 35. 66–73. 14 indexed citations

11.

Miyazaki, Takuro, Yoshitaka Taketomi, Yuta Saito, et al.. (2015). Calpastatin Counteracts Pathological Angiogenesis by Inhibiting Suppressor of Cytokine Signaling 3 Degradation in Vascular Endothelial Cells. Circulation Research. 116(7). 1170–1181. 43 indexed citations

12.

Kim‐Kaneyama, Joo‐ri, Koji Ohnishi, Motohiro Takeya, et al.. (2015). Role of Hic-5 in the formation of microvilli-like structures and the monocyte–endothelial interaction that accelerates atherosclerosis. Cardiovascular Research. 105(3). 361–371. 16 indexed citations

13.

Kim‐Kaneyama, Joo‐ri, Xiao‐Feng Lei, Susila Arita, et al.. (2012). Identification of Hic‐5 as a novel regulatory factor for integrin αIIbβ3 activation and platelet aggregation in mice. Journal of Thrombosis and Haemostasis. 10(9). 1867–1874. 9 indexed citations

14.

Miyazaki, Takuro, et al.. (2012). Calpain and Atherosclerosis. Journal of Atherosclerosis and Thrombosis. 20(3). 228–237. 35 indexed citations

15.

Kim‐Kaneyama, Joo‐ri, et al.. (2012). Hydrogen Peroxide-Inducible Clone 5 (Hic-5) as a Potential Therapeutic Target for Vascular and Other Disorders. Journal of Atherosclerosis and Thrombosis. 19(7). 601–607. 18 indexed citations

16.

Nagashima, Masaharu, Takuya Watanabe, Michishige Terasaki, et al.. (2011). Native incretins prevent the development of atherosclerotic lesions in apolipoprotein E knockout mice. Diabetologia. 54(10). 2649–2659. 209 indexed citations

17.

Kim‐Kaneyama, Joo‐ri, Naoki Takeda, Akira Miyazaki, et al.. (2010). Hic-5 deficiency enhances mechanosensitive apoptosis and modulates vascular remodeling. Journal of Molecular and Cellular Cardiology. 50(1). 77–86. 36 indexed citations

18.

Kim‐Kaneyama, Joo‐ri, et al.. (2008). Hic-5, an adaptor protein expressed in vascular smooth muscle cells, modulates the arterial response to injury in vivo. Biochemical and Biophysical Research Communications. 376(4). 682–687. 17 indexed citations

19.

Mori, Kazunori, Masayuki Asakawa, Miki Hayashi, et al.. (2006). Oligomerizing Potential of a Focal Adhesion LIM Protein Hic-5 Organizing a Nuclear-Cytoplasmic Shuttling Complex. Journal of Biological Chemistry. 281(31). 22048–22061. 20 indexed citations

20.

Shibanuma, Motoko, Joo‐ri Kim‐Kaneyama, Keiko Ishino, et al.. (2003). Hic-5 Communicates between Focal Adhesions and the Nucleus through Oxidant-Sensitive Nuclear Export Signal. Molecular Biology of the Cell. 14(3). 1158–1171. 76 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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